eNOS overexpression exacerbates vascular closure in the obliterative phase of OIR and increases angiogenic drive in the subsequent proliferative stage
about
Arginase 2 deficiency prevents oxidative stress and limits hyperoxia-induced retinal vascular degeneration.Nitric oxide in the pathophysiology of retinopathy: evidences from preclinical and clinical researches.eNOS plays an important role in the regulation of colonic inflammation: a novel therapeutic target and a predictive marker for the prognosis of ulcerative colitis.IGFBP-3 reduces eNOS and PKCzeta phosphorylation, leading to lowered VEGF levels.Tetrahydrobiopterin (BH4) deficiency is associated with augmented inflammation and microvascular degeneration in the retina.BH4-Mediated Enhancement of Endothelial Nitric Oxide Synthase Activity Reduces Hyperoxia-Induced Endothelial Damage and Preserves Vascular Integrity in the Neonate.
P2860
eNOS overexpression exacerbates vascular closure in the obliterative phase of OIR and increases angiogenic drive in the subsequent proliferative stage
description
2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
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eNOS overexpression exacerbate ...... subsequent proliferative stage
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eNOS overexpression exacerbate ...... subsequent proliferative stage
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type
label
eNOS overexpression exacerbate ...... subsequent proliferative stage
@ast
eNOS overexpression exacerbate ...... subsequent proliferative stage
@en
prefLabel
eNOS overexpression exacerbate ...... subsequent proliferative stage
@ast
eNOS overexpression exacerbate ...... subsequent proliferative stage
@en
P2093
P2860
P356
P1476
eNOS overexpression exacerbate ...... subsequent proliferative stage
@en
P2093
Denise M McDonald
Rien van Haperen
Rini de Crom
Thomas A Gardiner
P2860
P304
P356
10.1167/IOVS.12-9797
P407
P577
2012-10-03T00:00:00Z