Spontaneous development of intestinal and colonic atrophy and inflammation in the carnitine-deficient jvs (OCTN2(-/-)) mice - Test2 - elhamkhani - TriplyDB

Spontaneous development of intestinal and colonic atrophy and inflammation in the carnitine-deficient jvs (OCTN2(-/-)) mice

Spontaneous development of intestinal and colonic atrophy and inflammation in the carnitine-deficient jvs (OCTN2(-/-)) mice

http://www.wikidata.org/entity/Q36319365

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Mitochondrial dysfunction in inflammatory bowel disease Inflammatory bowel disease Xenobiotic, bile acid, and cholesterol transporters: function and regulation.Potential Use of Chemoprotectants against the Toxic Effects of Cyanotoxins: A Review.Mechanisms of intestinal inflammation and development of associated cancers: lessons learned from mouse models.Colon OCTN2 gene expression is up-regulated by peroxisome proliferator-activated receptor gamma in humans and mice and contributes to local and systemic carnitine homeostasis Genes and environment: how will our concepts on the pathophysiology of IBD develop in the future?Carnitine deficiency in OCTN2-/- newborn mice leads to a severe gut and immune phenotype with widespread atrophy, apoptosis and a pro-inflammatory response Remote communication through solute carriers and ATP binding cassette drug transporter pathways: an update on the remote sensing and signaling hypothesis.The contribution of natural selection to present-day susceptibility to chronic inflammatory and autoimmune disease.Induction of dsRNA-activated protein kinase links mitochondrial unfolded protein response to the pathogenesis of intestinal inflammation.Propionyl-L-carnitine hydrochloride for treatment of mild to moderate colonic inflammatory bowel diseases What do drug transporters really do?Transport of butyryl-L-carnitine, a potential prodrug, via the carnitine transporter OCTN2 and the amino acid transporter ATB(0,+)Influence of l-carnitine on metabolism and performance of sows.Environmental Enteric Dysfunction is Associated with Carnitine Deficiency and Altered Fatty Acid Oxidation.Mitochondria at the interface between danger signaling and metabolism: role of unfolded protein responses in chronic inflammation.Renal transport of organic anions and cations.Mouse models of intestinal inflammation and cancer.Downregulation of duodenal SLC transporters and activation of proinflammatory signaling constitute the early response to high altitude in humans.Ulcerative Colitis Is Under Dual (Mitochondrial and Nuclear) Genetic Control.Enzymes involved in L-carnitine biosynthesis are expressed by small intestinal enterocytes in mice: implications for gut health.A Potential Biomarker for Acute Kidney Injury in Preterm Infants from Metabolic Profiling Inflammatory bowel disease: recent advances on genetics and innate immunity Reduced mitochondrial activity in colonocytes facilitates AMPKα2-dependent inflammation.Necrotizing enterocolitis and respiratory distress syndrome as first clinical presentation of mitochondrial trifunctional protein deficiency.L-carnitine, a diet component and organic cation transporter OCTN ligand, displays immunosuppressive properties and abrogates intestinal inflammation.Examining the protective effects of acetyl l-carnitine on cisplatin-induced uterine tube toxicity.Studies on the chemopreventive effect of carnitine on tumorigenesis in vivo, using two experimental murine models of colon cancer.Gene expression of carnitine organic cation transporters 1 and 2 (OCTN) is downregulated in patients with ulcerative colitis.Tissue-specific changes in mRNA expression of Abc and Slc transporters in murine pulmonary tuberculosis.Emerging Mechanisms of Innate Immunity and Their Translational Potential in Inflammatory Bowel Disease.Dietaryl-carnitine prevents histopathological changes in tilapia (Oreochromis Niloticus) exposed to cylindrospermopsin

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Spontaneous development of intestinal and colonic atrophy and inflammation in the carnitine-deficient jvs (OCTN2(-/-)) mice

http://www.wikidata.org/entity/Q36319365

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2007 nî lūn-bûn

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J.YMGME.2007.08.002

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Molecular Genetics and Metabolism

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Spontaneous development of int ...... eficient jvs (OCTN2(-/-)) mice

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Dietrich Matern

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Hongyan Xu

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Michael J Bennett

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Penny Roon

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Prem S Shekhawat

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Puttur D Prasad

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Richard Boriack

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Sonne R Srinivas

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Varghese George

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Primary systemic carnitine deficiency is caused by mutations in a gene encoding sodium ion-dependent carnitine transporter

The Orphan G protein-coupled receptors GPR41 and GPR43 are activated by propionate and other short chain carboxylic acids

Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls

Disorders of carnitine transport and the carnitine cycle

Functional domains in the carnitine transporter OCTN2, defective in primary carnitine deficiency

Functional variants of OCTN cation transporter genes are associated with Crohn disease

Single nucleotide polymorphisms of OCTN1, OCTN2, and DLG5 genes in Greek patients with Crohn's disease

A risk haplotype in the Solute Carrier Family 22A4/22A5 gene cluster influences phenotypic expression of Crohn's disease

Na+- and Cl--coupled active transport of carnitine by the amino acid transporter ATB(0,+) from mouse colon expressed in HRPE cells and Xenopus oocytes

Short-chain fatty acid receptor, GPR43, is expressed by enteroendocrine cells and mucosal mast cells in rat intestine

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4

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92

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Vadivel Ganapathy

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