Muscle expression of a local Igf-1 isoform protects motor neurons in an ALS mouse model. - Test2 - elhamkhani - TriplyDB

Muscle expression of a local Igf-1 isoform protects motor neurons in an ALS mouse model.

Muscle expression of a local Igf-1 isoform protects motor neurons in an ALS mouse model.

http://www.wikidata.org/entity/Q36321086

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Intramuscular viral delivery of paraplegin rescues peripheral axonopathy in a model of hereditary spastic paraplegia Insulin-like growth factor-I for the treatment of amyotrophic lateral sclerosis Delivery of recombinant follistatin lessens disease severity in a mouse model of spinal muscular atrophy.Subcutaneous IGF-1 is not beneficial in 2-year ALS trial Androgen-dependent pathology demonstrates myopathic contribution to the Kennedy disease phenotype in a mouse knock-in model Regulating Axonal Responses to Injury: The Intersection between Signaling Pathways Involved in Axon Myelination and The Inhibition of Axon Regeneration Genetic variation and exercise-induced muscle damage: implications for athletic performance, injury and ageing Mechanisms of Muscle Denervation in Aging: Insights from a Mouse Model of Amyotrophic Lateral Sclerosis Optimizing IGF-I for skeletal muscle therapeutics Increased androgenic sensitivity in the hind limb muscular system marks the evolution of a derived gestural display Postnatal muscle modification by myogenic factors modulates neuropathology and survival in an ALS mouse model The Emerging Roles of the Calcineurin-Nuclear Factor of Activated T-Lymphocytes Pathway in Nervous System Functions and Diseases Systems biology of neurodegenerative diseases ALS as a distal axonopathy: molecular mechanisms affecting neuromuscular junction stability in the presymptomatic stages of the disease Ablation of proliferating cells in the CNS exacerbates motor neuron disease caused by mutant superoxide dismutase The Evolutionarily Conserved Tre2/Bub2/Cdc16 (TBC), Lysin Motif (LysM), Domain Catalytic (TLDc) Domain Is Neuroprotective against Oxidative Stress Hormones and the neuromuscular control of courtship in the golden-collared manakin (Manacus vitellinus).A randomized, double blind, placebo-controlled trial of pioglitazone in combination with riluzole in amyotrophic lateral sclerosis Dissecting muscle and neuronal disorders in a Drosophila model of muscular dystrophy.Peripheral androgen receptors sustain the acrobatics and fine motor skill of elaborate male courtship.Research Resource: Hormones, Genes, and Athleticism: Effect of Androgens on the Avian Muscular Transcriptome Evolutionary patterns of adaptive acrobatics and physical performance predict expression profiles of androgen receptor - but not oestrogen receptor - in the forelimb musculature Fast skeletal muscle troponin activator tirasemtiv increases muscle function and performance in the B6SJL-SOD1G93A ALS mouse model Hyperactive intracellular calcium signaling associated with localized mitochondrial defects in skeletal muscle of an animal model of amyotrophic lateral sclerosis Growth Hormone Deteriorates the Functional Outcome in an Experimental Model of Huntington's Disease Induced by 3-Nitropionic Acid Satellite cell dysfunction and impaired IGF-1 signaling cause CKD-induced muscle atrophy Functional muscle regeneration with combined delivery of angiogenesis and myogenesis factors.Skeletal muscle-restricted expression of human SOD1 causes motor neuron degeneration in transgenic mice Insulin-like growth factor 1 physiology: lessons from mouse models.Role of exercise in maintaining the integrity of the neuromuscular junction.Cross-talk between IGF-1 and estrogen receptors attenuates intracellular changes in ventral spinal cord 4.1 motoneuron cells because of interferon-gamma exposure.Muscle expression of mutant androgen receptor accounts for systemic and motor neuron disease phenotypes in spinal and bulbar muscular atrophy Moderate exercise delays the motor performance decline in a transgenic model of ALS.Neurotrophic growth factors for the treatment of amyotrophic lateral sclerosis: where do we stand?Neuroprotection for amyotrophic lateral sclerosis: role of stem cells, growth factors, and gene therapy.Dihydrotestosterone ameliorates degeneration in muscle, axons and motoneurons and improves motor function in amyotrophic lateral sclerosis model mice.Virus-delivered small RNA silencing sustains strength in amyotrophic lateral sclerosis.Delayed recovery of skeletal muscle mass following hindlimb immobilization in mTOR heterozygous mice.AAV4-mediated expression of IGF-1 and VEGF within cellular components of the ventricular system improves survival outcome in familial ALS mice Loss of synaptic vesicles from neuromuscular junctions in aged MRF4-null mice.

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P2860

Muscle expression of a local Igf-1 isoform protects motor neurons in an ALS mouse model.

http://www.wikidata.org/entity/Q36321086

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name

Muscle expression of a local Igf-1 isoform protects motor neurons in an ALS mouse model.

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Muscle expression of a local Igf-1 isoform protects motor neurons in an ALS mouse model.

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type

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Muscle expression of a local Igf-1 isoform protects motor neurons in an ALS mouse model.

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Muscle expression of a local Igf-1 isoform protects motor neurons in an ALS mouse model.

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prefLabel

Muscle expression of a local Igf-1 isoform protects motor neurons in an ALS mouse model.

@ast

Muscle expression of a local Igf-1 isoform protects motor neurons in an ALS mouse model.

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P1476

Muscle expression of a local Igf-1 isoform protects motor neurons in an ALS mouse model.

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P2093

Antonio Musarò

http://www.w3.org/2001/XMLSchema#string

Carmine Nicoletti

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Cristina Giacinti

http://www.w3.org/2001/XMLSchema#string

Gabriella Dobrowolny

http://www.w3.org/2001/XMLSchema#string

Laura Barberi

http://www.w3.org/2001/XMLSchema#string

Laura Pelosi

http://www.w3.org/2001/XMLSchema#string

Mario Molinaro

http://www.w3.org/2001/XMLSchema#string

Nadia Rosenthal

http://www.w3.org/2001/XMLSchema#string

Nadine Winn

http://www.w3.org/2001/XMLSchema#string

P2860

Viral mediated expression of insulin-like growth factor I blocks the aging-related loss of skeletal muscle function

Calcineurin is a potent regulator for skeletal muscle regeneration by association with NFATc1 and GATA-2

Motor neuron degeneration in mice that express a human Cu,Zn superoxide dismutase mutation

Knockout of glutamate transporters reveals a major role for astroglial transport in excitotoxicity and clearance of glutamate

Localized Igf-1 transgene expression sustains hypertrophy and regeneration in senescent skeletal muscle.

Maturation of the myogenic program is induced by postmitotic expression of insulin-like growth factor I.

Diseases of the neuromuscular junction.

A calcineurin-dependent transcriptional pathway controls skeletal muscle fiber type

Stem cell-mediated muscle regeneration is enhanced by local isoform of insulin-like growth factor 1.

Muscle-specific expression of insulin-like growth factor I counters muscle decline in mdx mice.

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193-199

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scholarly article

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10.1083/JCB.200407021

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P433

2

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168

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2005-01-01T00:00:00Z

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8074808

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15657392

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P921

amyotrophic lateral sclerosis

P932

2171577

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