Monocytes control second-phase neutrophil emigration in established lipopolysaccharide-induced murine lung injury. - Test2 - elhamkhani - TriplyDB

Monocytes control second-phase neutrophil emigration in established lipopolysaccharide-induced murine lung injury.

Monocytes control second-phase neutrophil emigration in established lipopolysaccharide-induced murine lung injury.

http://www.wikidata.org/entity/Q36345279

about

Low-dose AgNPs reduce lung mechanical function and innate immune defense in the absence of cellular toxicity Flow cytometric analysis of macrophages and dendritic cell subsets in the mouse lung SAA drives proinflammatory heterotypic macrophage differentiation in the lung via CSF-1R-dependent signaling.FXYD5 Is an Essential Mediator of the Inflammatory Response during Lung Injury.Macrophage phenotype is associated with disease severity in preterm infants with chronic lung disease.Role of alveolar macrophages in chronic obstructive pulmonary disease.Spironolactone attenuates bleomycin-induced pulmonary injury partially via modulating mononuclear phagocyte phenotype switching in circulating and alveolar compartments.In vivo compartmental analysis of leukocytes in mouse lungs.VEGF Production by Ly6C+high Monocytes Contributes to Ventilator-Induced Lung Injury.Macrophages are critical to the maintenance of IL-13-dependent lung inflammation and fibrosis Leukocyte-specific protein 1 regulates neutrophil recruitment in acute lung inflammation.CXCL10/IP-10 Neutralization Can Ameliorate Lipopolysaccharide-Induced Acute Respiratory Distress Syndrome in Rats Effects of basic drugs on prognosis of acute lung injury in mice.A Single 9-Colour Flow Cytometric Method to Characterise Major Leukocyte Populations in the Rat: Validation in a Model of LPS-Induced Pulmonary Inflammation.Activated prostaglandin D2 receptors on macrophages enhance neutrophil recruitment into the lung.Activation of human mesenchymal stem cells impacts their therapeutic abilities in lung injury by increasing interleukin (IL)-10 and IL-1RN levels.CXCR4 identifies transitional bone marrow premonocytes that replenish the mature monocyte pool for peripheral responses.Extracellular ATP mediates the late phase of neutrophil recruitment to the lung in murine models of acute lung injury.Decidual neutrophil infiltration is not required for preterm birth in a mouse model of infection-induced preterm labor.Diverse macrophage populations mediate acute lung inflammation and resolution.Update in acute respiratory distress syndrome and mechanical ventilation 2012.Novel concepts of acute lung injury and alveolar-capillary barrier dysfunction.Dynamics of pulmonary endothelial barrier function in acute inflammation: mechanisms and therapeutic perspectives.Immunosenescence in monocytes, macrophages, and dendritic cells: lessons learned from the lung and heart.Macrophage-epithelial interactions in pulmonary alveoli.Inhibition of TNF Receptor p55 By a Domain Antibody Attenuates the Initial Phase of Acid-Induced Lung Injury in Mice.Wogonin prevents lipopolysaccharide-induced acute lung injury and inflammation in mice via peroxisome proliferator-activated receptor gamma-mediated attenuation of the nuclear factor-kappaB pathway.Increased levels of Gab1 and Gab2 adaptor proteins skew interleukin-4 (IL-4) signaling toward M2 macrophage-driven pulmonary fibrosis in mice.Signals of vagal circuits engaging with AKT1 in α7 nAChR+CD11b+ cells lessen E. coli and LPS-induced acute inflammatory injury.Donor pulmonary intravascular nonclassical monocytes recruit recipient neutrophils and mediate primary lung allograft dysfunction.Activated endothelial cells limit inflammatory response, but increase chemoattractant potential and bacterial clearance by human monocytes.Monocytes regulate systemic coagulation and inflammation in abdominal sepsis.Akt2 deficiency as a therapeutic strategy protects against acute lung injury.Paired immunoglobulin-like receptor-B inhibits pulmonary fibrosis by suppressing profibrogenic properties of alveolar macrophages.The monocyte and acute respiratory distress syndrome: implicated, innocent bystander, or awash in research translation?Pathophysiology and classification of primary graft dysfunction after lung transplantation.Novel role for endogenous mitochondrial formylated peptide-driven formyl peptide receptor 1 signalling in acute respiratory distress syndrome.Depletion of circulating monocytes suppresses IL-17 and HMGB1 expression in mice with LPS-induced acute lung injury.A randomized controlled trial of peripheral blood mononuclear cell depletion in experimental human lung inflammation.CD11b+ interstitial macrophages are required for ischemia-induced lung angiogenesis.

P2860

Q28389887-1BD1EB65-6828-4BF7-ABA9-B806004D658D Q28390828-A0C3574A-5E72-421D-B8AD-CEA35B5A9ABD Q33576194-EFFA8B9F-9274-48F6-AC18-D3DE5BFC8B58 Q33747385-3AA683B2-A3FA-4BB3-BBFF-B3EC2C827858 Q34034954-3287690C-2078-4535-B800-DAE46A44841C Q34159624-667D33ED-D378-450E-AECE-3F1A7D30DB2F Q35049443-BF0BF7E3-248B-4C92-A8A3-01C11C1C4992 Q36122872-7E9993ED-D8D5-47A6-999E-CD7A3FE3DF84 Q36174967-E0D4B19D-F9DB-4115-8EAB-7BBFC28651CC Q36225950-0C8FC5E6-84CF-42D8-BA98-41570E9A5565 Q36237194-64880A2C-C1EE-4077-B94C-8D53D38604F8 Q36238175-14A668F0-F701-451D-AA37-41153A698A8E Q36412118-5682CF1C-9A3C-4F33-BFE7-4B532B6CE079 Q36463374-06D5A1DF-BD7B-4C6B-8781-4D912DF74875 Q37112098-13A98CB5-3148-4DF1-91A4-84A0369B1B44 Q37255056-B84C7FE6-E2A5-4F35-9ED9-712E893D319D Q37346711-4EF552F3-3723-4857-B74D-9CFD141343BA Q37576085-F707A3AA-4CF6-469F-8A2C-F796DDB3EDF5 Q37599663-37D5EF64-B2FB-43F7-A3E1-32D4F2ACAED9 Q37707609-F659FA5E-1609-42E5-B31A-036B9E1218CF Q38125713-24EBE014-8D30-4C28-BE99-783D84FE50E5 Q38138015-56F89A71-4241-418B-A9EF-492AE1314045 Q38193705-E1841E0C-0361-4979-A0FB-501691CB801F Q38253911-604BFD30-1357-4CB4-832A-3C3B6B46B7E2 Q38832516-7E0253B9-C678-44D6-BD37-D959E0C7D2A7 Q38938464-E4AF1ED2-D6DB-4F9F-8E07-648E867CEAE3 Q39001240-49711ABA-DF09-42C9-ADC8-59A38F96DFA6 Q40143861-18F5886D-1BD9-4AA0-8984-3617F3101328 Q40191513-BA783651-8DC7-4010-82B4-4603C1695F33 Q41502574-F0D93322-BF66-459E-A096-1344A9EA6F6F Q41566183-72105D41-F9BD-4DE1-B18C-A7D22CB675A8 Q41602019-B6996E81-4242-44FD-86AB-EA20DB78E24E Q43213895-B08C9ECD-7EEE-41A0-9DC3-0065E063CBC5 Q43631328-6EEDC61C-9369-4773-9A5B-25A2E32BDE26 Q46125002-919F8390-0BD7-459D-B7AD-E232C56DC5D8 Q47120100-3D549373-FEEC-4CE1-9921-48CD7C375DA6 Q47134075-8ED5D627-7D4D-4B07-BCB4-5EA7274EFCC2 Q48027811-94342DB8-1DB2-4F10-B95C-DF6D72CEDB2D Q51032764-5386D453-F62A-44E6-89D3-04854C31ADB8 Q55257731-E541CCF1-CBE8-4FEC-B0F9-2A1AE88CA9CF

P2860

Monocytes control second-phase neutrophil emigration in established lipopolysaccharide-induced murine lung injury.

http://www.wikidata.org/entity/Q36345279

description

2012 nî lūn-bûn

@nan

2012年の論文

@ja

2012年学术文章

@wuu

2012年学术文章

@zh-cn

2012年学术文章

@zh-hans

2012年学术文章

@zh-my

2012年学术文章

@zh-sg

2012年學術文章

@yue

2012年學術文章

@zh

2012年學術文章

@zh-hant

name

Monocytes control second-phase ...... de-induced murine lung injury.

@ast

Monocytes control second-phase ...... de-induced murine lung injury.

@en

type

label

Monocytes control second-phase ...... de-induced murine lung injury.

@ast

Monocytes control second-phase ...... de-induced murine lung injury.

@en

prefLabel

Monocytes control second-phase ...... de-induced murine lung injury.

@ast

Monocytes control second-phase ...... de-induced murine lung injury.

@en

P1433

Q36345279-177CA3F3-B38F-4A44-A42F-A29A2DA52974

P1476

Q36345279-4FD8721C-ABE5-428C-941B-160827D898D8

P2093

Q36345279-0FBCE14A-3AA4-4979-96E6-4F8E0BCFEC59

Q36345279-1174C0CC-F5CF-4A59-818B-E2A79FF1924D

Q36345279-19A5F2C9-60E5-48F8-84AD-4DCC5E8C987B

Q36345279-1C0426E2-4384-4CFD-BE61-2719CD1DEB55

Q36345279-24CD2AB8-C5D6-4A37-99D9-B2510532A821

Q36345279-4E752918-FD58-4C94-94DF-44A6CF576C8C

Q36345279-632D9FC0-34B8-44DB-91B0-EF560BC4B864

Q36345279-6D0F1C99-BA12-4A3E-8C52-5984EAEAECC4

Q36345279-85116C2A-1926-48C0-9B3B-722FA3FFC517

Q36345279-8BDAFA87-EDF3-4C9A-9DA2-F1B334C0300F

P2860

Q36345279-057F725C-4F14-4D0B-B51F-09D3C976D0EA

Q36345279-0E27971F-69D9-4B49-93DE-C0C1DD4BDC3B

Q36345279-0E754025-441A-4949-8EAC-F12C10CBAB34

Q36345279-0F974D84-DC81-4AEF-985C-84F0399C9504

Q36345279-18E5851B-B791-4AAE-BD2C-991380176529

Q36345279-1F9B39F4-5C39-48DE-8117-2D98AF88AE4E

Q36345279-227F6408-BB18-437F-941D-236364151484

Q36345279-279BC576-415C-41D1-A6D1-99DE32CD4358

Q36345279-3E4C5EA3-795C-46DE-A0EB-A9F35B55B139

Q36345279-3EAF837B-36EA-457A-BBCA-26AEC9C80612

P304

Q36345279-C672C6E4-AB9B-41DC-828C-71E9F3641FD0

P31

Q36345279-833BE33E-A8C7-4490-8BA8-A0F993CC086E

P356

Q36345279-D50F68FE-6885-4001-ABF0-140A9B557359

P407

Q36345279-87964C76-9AE6-453F-A4AD-407DAACB8EF4

P433

Q36345279-B6539589-2E6E-4B2D-986A-790FD9B33D37

P478

Q36345279-5C3E851B-D275-4CAD-837C-F108EE2F047A

P50

Q36345279-25C57F04-C7EB-4B1E-AA6F-F0AC3E8878D7

Q36345279-5D0358B5-79D4-4A87-89D3-55981D764AAF

Q36345279-71AA17B6-02A1-4A13-8ACB-A607ADBF0568

Q36345279-85C6DCA5-549D-4055-85F3-8F4B39891C9B

Q36345279-B6CFE98D-C6F3-481C-B4EE-98986953D7D3

Q36345279-FEFA1C70-619A-402B-9B04-48FA79A53865

P577

Q36345279-7EE2CDAA-CD81-43F6-B514-B9DD32FF3F36

P698

Q36345279-8BE63634-C25A-45FB-B426-43A1EEC6FCC1

P921

Q36345279-BB769A25-6E6F-472E-A140-E65C0EC4045E

P932

Q36345279-619C5A91-DF44-4FF9-8894-A31D89175FCE

P356

RCCM.201112-2132OC

P1433

American Journal of Respiratory and Critical Care Medicine

P1476

Monocytes control second-phase ...... ide-induced murine lung injury

@en

P2093

Adriano G Rossi

http://www.w3.org/2001/XMLSchema#string

Alison MacKinnon

http://www.w3.org/2001/XMLSchema#string

Andrew Conway Morris

http://www.w3.org/2001/XMLSchema#string

Chris Haslett

http://www.w3.org/2001/XMLSchema#string

David A Dorward

http://www.w3.org/2001/XMLSchema#string

Duncan Humphries

http://www.w3.org/2001/XMLSchema#string

Emma Scholefield

http://www.w3.org/2001/XMLSchema#string

Jeremy Hughes

http://www.w3.org/2001/XMLSchema#string

Matthias Mack

http://www.w3.org/2001/XMLSchema#string

Michael Gibbons

http://www.w3.org/2001/XMLSchema#string

P2860

TRPM2-mediated Ca2+influx induces chemokine production in monocytes that aggravates inflammatory neutrophil infiltration

Alveolar macrophages regulate neutrophil recruitment in endotoxin-induced lung injury.

Incidence and outcomes of acute lung injury

The acute respiratory distress syndrome

Monitoring of blood vessels and tissues by a population of monocytes with patrolling behavior

The American-European Consensus Conference on ARDS. Definitions, mechanisms, relevant outcomes, and clinical trial coordination

Critical role of endothelial CXCR2 in LPS-induced neutrophil migration into the lung.

Diverse and potent chemokine production by lung CD11bhigh dendritic cells in homeostasis and in allergic lung inflammation.

In vivo two-photon imaging reveals monocyte-dependent neutrophil extravasation during pulmonary inflammation.

Nomenclature of monocytes and dendritic cells in blood.

P304

514-524

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1164/RCCM.201112-2132OC

http://www.w3.org/2001/XMLSchema#string

P407

P433

6

http://www.w3.org/2001/XMLSchema#string

P478

186

http://www.w3.org/2001/XMLSchema#string

P50

Thomas S. Wilkinson

David A Ferenbach

Donald J. Davidson

P577

2012-07-19T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

22822022

http://www.w3.org/2001/XMLSchema#string

P921

lipopolysaccharide

P932

3480527

http://www.w3.org/2001/XMLSchema#string