Escherichia coli lacking RpoS are rare in natural populations of non-pathogens
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dsdA Does Not Affect Colonization of the Murine Urinary Tract by Escherichia coli CFT073The General Stress Response Is Conserved in Long-Term Soil-Persistent Strains of Escherichia coliThe rpoS gene is predominantly inactivated during laboratory storage and undergoes source-sink evolution in Escherichia coli species.Mutations in global regulators lead to metabolic selection during adaptation to complex environments.RpoS contributes to phagocyte oxidase-mediated stress resistance during urinary tract infection by Escherichia coli CFT073.Fitness, stress resistance, and extraintestinal virulence in Escherichia coli.Benefit of transferred mutations is better predicted by the fitness of recipients than by their ecological or genetic relatednessElucidating the function of the RpoS regulon.Bacterial virulence phenotypes of Escherichia coli and host susceptibility determine risk for urinary tract infections.RpoS role in virulence and fitness in enteropathogenic Escherichia coli.Genome-Wide Transcriptional Response to Varying RpoS Levels in Escherichia coli K-12A network of regulators promotes dehydration tolerance in Escherichia coli.
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Escherichia coli lacking RpoS are rare in natural populations of non-pathogens
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Escherichia coli lacking RpoS are rare in natural populations of non-pathogens
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Escherichia coli lacking RpoS are rare in natural populations of non-pathogens
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Escherichia coli lacking RpoS are rare in natural populations of non-pathogens
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Escherichia coli lacking RpoS are rare in natural populations of non-pathogens
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Escherichia coli lacking RpoS are rare in natural populations of non-pathogens
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Escherichia coli lacking RpoS are rare in natural populations of non-pathogens
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Escherichia coli lacking RpoS are rare in natural populations of non-pathogens
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David M Gordon
Emily Snyder
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P304
P356
10.1534/G3.112.003855
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2012-11-01T00:00:00Z