Brucella evades macrophage killing via VirB-dependent sustained interactions with the endoplasmic reticulum
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Invasion of the central nervous system by intracellular bacteriaEstablishment of Chronic Infection: Brucella's Stealth StrategyType IV secretion system of Brucella spp. and its effectorsProteomic analysis of detergent resistant membrane domains during early interaction of macrophages with rough and smooth Brucella melitensisAn atypical riboflavin pathway is essential for Brucella abortus virulenceContrasting Lifestyles Within the Host CellYip1A, a novel host factor for the activation of the IRE1 pathway of the unfolded protein response during Brucella infectionEarly trafficking and intracellular replication of Legionella longbeachaea within an ER-derived late endosome-like phagosomeFunctional characterization of the incomplete phosphotransferase system (PTS) of the intracellular pathogen Brucella melitensisSalmonella acquires lysosome-associated membrane protein 1 (LAMP1) on phagosomes from Golgi via SipC protein-mediated recruitment of host Syntaxin6.Brucella induces an unfolded protein response via TcpB that supports intracellular replication in macrophagesLOV Histidine Kinase Modulates the General Stress Response System and Affects the virB Operon Expression in Brucella abortusG1-arrested newborn cells are the predominant infectious form of the pathogen Brucella abortus.Maturation of Rhodococcus equi-containing vacuoles is arrested after completion of the early endosome stage.Generation of the Brucella melitensis ORFeome version 1.1.The phagosome: compartment with a license to kill.Brucella abortus uses a stealthy strategy to avoid activation of the innate immune system during the onset of infection.Brucella control of dendritic cell maturation is dependent on the TIR-containing protein Btp1.RNAi screen of endoplasmic reticulum-associated host factors reveals a role for IRE1alpha in supporting Brucella replication.A Legionella pneumophila effector protein encoded in a region of genomic plasticity binds to Dot/Icm-modified vacuolesComparative proteomics analyses reveal the virB of B. melitensis affects expression of intracellular survival related proteins.The glyceraldehyde-3-phosphate dehydrogenase and the small GTPase Rab 2 are crucial for Brucella replication.Brucella melitensis VjbR and C12-HSL regulons: contributions of the N-dodecanoyl homoserine lactone signaling molecule and LuxR homologue VjbR to gene expression.Innate immune recognition of flagellin limits systemic persistence of BrucellaGetting "Inside" Type I IFNs: Type I IFNs in Intracellular Bacterial InfectionsAnaplasma marginale type IV secretion system proteins VirB2, VirB7, VirB11, and VirD4 are immunogenic components of a protective bacterial membrane vaccine.Intracellular bacteria interfere with dendritic cell functions: role of the type I interferon pathway.ChIP-seq analysis of the LuxR-type regulator VjbR reveals novel insights into the Brucella virulence gene expression network.Opsonized virulent Brucella abortus replicates within nonacidic, endoplasmic reticulum-negative, LAMP-1-positive phagosomes in human monocytesBrucella coopts the small GTPase Sar1 for intracellular replicationThe Brucella abortus phosphoglycerate kinase mutant is highly attenuated and induces protection superior to that of vaccine strain 19 in immunocompromised and immunocompetent miceThe Burkholderia pseudomallei type III secretion system and BopA are required for evasion of LC3-associated phagocytosisEndocytosis of viruses and bacteria.Brucella abortus virB12 is expressed during infection but is not an essential component of the type IV secretion systemDeep-sequencing analysis of the mouse transcriptome response to infection with Brucella melitensis strains of differing virulence.Expression of VjbR under nutrient limitation conditions is regulated at the post-transcriptional level by specific acidic pH values and urocanic acid.Early transcriptional responses of bovine chorioallantoic membrane explants to wild type, ΔvirB2 or ΔbtpB Brucella abortus infection.Identification of mechanisms for attenuation of the FSC043 mutant of Francisella tularensis SCHU S4.Mechanisms of microbial escape from phagocyte killing.Evidence of the presence of a functional Dot/Icm type IV-B secretion system in the fish bacterial pathogen Piscirickettsia salmonis
P2860
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P2860
Brucella evades macrophage killing via VirB-dependent sustained interactions with the endoplasmic reticulum
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
@zh
2003年學術文章
@zh-hant
name
Brucella evades macrophage kil ...... with the endoplasmic reticulum
@ast
Brucella evades macrophage kil ...... with the endoplasmic reticulum
@en
type
label
Brucella evades macrophage kil ...... with the endoplasmic reticulum
@ast
Brucella evades macrophage kil ...... with the endoplasmic reticulum
@en
prefLabel
Brucella evades macrophage kil ...... with the endoplasmic reticulum
@ast
Brucella evades macrophage kil ...... with the endoplasmic reticulum
@en
P2093
P2860
P356
P1476
Brucella evades macrophage kil ...... with the endoplasmic reticulum
@en
P2093
Chantal de Chastellier
Don-Marc Franchini
Edgardo Moreno
Javier Pizarro-Cerda
Jean Celli
Jean-Pierre Gorvel
P2860
P304
P356
10.1084/JEM.20030088
P407
P577
2003-08-01T00:00:00Z