Conditional deletion of TrkC does not modify limbic epileptogenesis

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Conditional deletion of TrkC does not modify limbic epileptogenesis

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Conditional deletion of TrkC does not modify limbic epileptogenesis

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Conditional deletion of TrkC does not modify limbic epileptogenesis

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Conditional deletion of TrkC does not modify limbic epileptogenesis

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Conditional deletion of TrkC does not modify limbic epileptogenesis

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Conditional deletion of TrkC does not modify limbic epileptogenesis

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Conditional deletion of TrkC does not modify limbic epileptogenesis

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P1476

Conditional deletion of TrkC does not modify limbic epileptogenesis

@en

P2093

A Soren Leonard

http://www.w3.org/2001/XMLSchema#string

Gumei Liu

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James O McNamara

http://www.w3.org/2001/XMLSchema#string

Jeffrey Helgager

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P2860

Neurotrophin signal transduction in the nervous system.

Modification of seizure activity by electrical stimulation. II. Motor seizure

Disruption of the neurotrophin-3 receptor gene trkC eliminates la muscle afferents and results in abnormal movements

trkC, a new member of the trk family of tyrosine protein kinases, is a receptor for neurotrophin-3

Ablation of NF1 function in neurons induces abnormal development of cerebral cortex and reactive gliosis in the brain

Localized delivery of fibroblast growth factor-2 and brain-derived neurotrophic factor reduces spontaneous seizures in an epilepsy model.

Disruption of TrkB-mediated phospholipase Cgamma signaling inhibits limbic epileptogenesis

Molecular signaling mechanisms underlying epileptogenesis.

Reduction of TrkB expression de novo in the adult mouse impairs epileptogenesis in the kindling model.

Brain-derived neurotrophic factor transgenic mice exhibit passive avoidance deficits, increased seizure severity and in vitro hyperexcitability in the hippocampus and entorhinal cortex.

P304

126-130

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scholarly article

P356

10.1016/J.EPLEPSYRES.2012.07.019

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P433

1-2

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P478

102

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P50

Ram S Puranam

P577

2012-09-12T00:00:00Z

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P698

22980123

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P932

3489989

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