Progesterone treatment reduces neuroinflammation, oxidative stress and brain damage and improves long-term outcomes in a rat model of repeated mild traumatic brain injury.
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The far-reaching scope of neuroinflammation after traumatic brain injury.Progesterone for acute traumatic brain injury.The neuroprotective steroid progesterone promotes mitochondrial uncoupling, reduces cytosolic calcium and augments stress resistance in yeast cellsProgesterone exerts neuroprotective effects and improves long-term neurologic outcome after intracerebral hemorrhage in middle-aged miceAltered Blood Biomarker Profiles in Athletes with a History of Repetitive Head Impacts.The synthetic progestin norgestrel modulates Nrf2 signaling and acts as an antioxidant in a model of retinal degeneration.Hormone replacement therapy reduces lipid oxidation directly at the arterial wall: A possible link to estrogens' cardioprotective effect through atherosclerosis prevention.Characterizing the "POAGome": A bioinformatics-driven approach to primary open-angle glaucoma.Sex matters: repetitive mild traumatic brain injury in adolescent rats.The Serum Changes of Neuron-Specific Enolase and Intercellular Adhesion Molecule-1 in Patients With Diffuse Axonal Injury Following Progesterone Administration: A Randomized Clinical Trial.The effects of 7-nitroindazole on serum neuron-specific enolase and astroglia-derived protein (S100β) levels after traumatic brain injury.Role of Sex Hormones on Brain Mitochondrial Function, with Special Reference to Aging and Neurodegenerative Diseases.Effects of Female Sex Steroids Administration on Pathophysiologic Mechanisms in Traumatic Brain Injury.Repeated mild traumatic brain injury produces neuroinflammation, anxiety-like behaviour and impaired spatial memory in mice.Acute Alcohol Binge Deteriorates Metabolic and Respiratory Compensation Capability After Blunt Chest Trauma Followed by Hemorrhagic Shock-A New Research Model.Lipid nanoparticles for administration of poorly water soluble neuroactive drugs.Neuroprotective effects of pifithrin-α against traumatic brain injury in the striatum through suppression of neuroinflammation, oxidative stress, autophagy, and apoptosis.Mesenchymal stem cells maintain the microenvironment of central nervous system by regulating the polarization of macrophages/microglia after traumatic brain injury.Repeated Mild Traumatic Brain Injury: Potential Mechanisms of Damage.Repeated mild traumatic brain injury can cause acute neurologic impairment without overt structural damage in juvenile rats.Telomere length and advanced diffusion MRI as biomarkers for repetitive mild traumatic brain injury in adolescent rats.Mild Traumatic Brain Injury in Adolescent Mice Alters Skull Bone Properties to Influence a Subsequent Brain Impact at Adulthood: A Pilot Study.Emerging Therapeutic Role of PPAR-α in Cognition and Emotions
P2860
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P2860
Progesterone treatment reduces neuroinflammation, oxidative stress and brain damage and improves long-term outcomes in a rat model of repeated mild traumatic brain injury.
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2015 nî lūn-bûn
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2015年の論文
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2015年学术文章
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2015年学术文章
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2015年学术文章
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name
Progesterone treatment reduces ...... d mild traumatic brain injury.
@ast
Progesterone treatment reduces ...... d mild traumatic brain injury.
@en
type
label
Progesterone treatment reduces ...... d mild traumatic brain injury.
@ast
Progesterone treatment reduces ...... d mild traumatic brain injury.
@en
prefLabel
Progesterone treatment reduces ...... d mild traumatic brain injury.
@ast
Progesterone treatment reduces ...... d mild traumatic brain injury.
@en
P2093
P2860
P50
P921
P1476
Progesterone treatment reduces ...... ed mild traumatic brain injury
@en
P2093
Bridgette D Semple
Donald G Stein
Ezgi Ozturk
Kyria M Webster
Sandy R Shultz
P2860
P2888
P356
10.1186/S12974-015-0457-7
P577
2015-12-18T00:00:00Z
P5875
P6179
1019138085