AP39, A Mitochondrially Targeted Hydrogen Sulfide Donor, Exerts Protective Effects in Renal Epithelial Cells Subjected to Oxidative Stress in Vitro and in Acute Renal Injury in Vivo.
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Hydrogen Sulfide, Oxidative Stress and Periodontal Diseases: A Concise ReviewThe Role of Hydrogen Sulfide in Renal SystemPre- and posttreatment with hydrogen sulfide prevents ventilator-induced lung injury by limiting inflammation and oxidation.The novel mitochondria-targeted hydrogen sulfide (H2S) donors AP123 and AP39 protect against hyperglycemic injury in microvascular endothelial cells in vitro.Cardioprotection by H2S Donors: Nitric Oxide-Dependent and ‑Independent Mechanisms.An integrative view of cisplatin-induced renal and cardiac toxicities: Molecular mechanisms, current treatment challenges and potential protective measures.Mitochondria-Targeted Triphenylphosphonium-Based Compounds: Syntheses, Mechanisms of Action, and Therapeutic and Diagnostic Applications.Delayed Treatment with Sodium Hydrosulfide Improves Regional Blood Flow and Alleviates Cecal Ligation and Puncture (CLP)-Induced Septic ShockEsterase-Sensitive Prodrugs with Tunable Release Rates and Direct Generation of Hydrogen Sulfide.Cooperative Oxygen Sensing by the Kidney and Carotid Body in Blood Pressure Control.Chemical Biology of H2S Signaling through Persulfidation.Hydrogen Sulfide Protects Renal Grafts Against Prolonged Cold Ischemia-Reperfusion Injury via Specific Mitochondrial Actions.Drug resistance induces the upregulation of H2S-producing enzymes in HCT116 colon cancer cells.International Union of Basic and Clinical Pharmacology. CII: Pharmacological Modulation of H2S Levels: H2S Donors and H2S Biosynthesis Inhibitors.AP39, a mitochondria-targeting hydrogen sulfide (H2 S) donor, protects against myocardial reperfusion injury independently of salvage kinase signalling.Hydrogen Sulfide Attenuates LPS-Induced Acute Kidney Injury by Inhibiting Inflammation and Oxidative Stress.
P2860
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P2860
AP39, A Mitochondrially Targeted Hydrogen Sulfide Donor, Exerts Protective Effects in Renal Epithelial Cells Subjected to Oxidative Stress in Vitro and in Acute Renal Injury in Vivo.
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2015 nî lūn-bûn
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2015年の論文
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
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name
AP39, A Mitochondrially Target ...... in Acute Renal Injury in Vivo.
@ast
AP39, A Mitochondrially Target ...... in Acute Renal Injury in Vivo.
@en
type
label
AP39, A Mitochondrially Target ...... in Acute Renal Injury in Vivo.
@ast
AP39, A Mitochondrially Target ...... in Acute Renal Injury in Vivo.
@en
prefLabel
AP39, A Mitochondrially Target ...... in Acute Renal Injury in Vivo.
@ast
AP39, A Mitochondrially Target ...... in Acute Renal Injury in Vivo.
@en
P2093
P2860
P1433
P1476
AP39, A Mitochondrially Target ...... in Acute Renal Injury in Vivo.
@en
P2093
Akbar Ahmad
Bartosz Szczesny
Csaba Szabo
Gabor Olah
Mark E Wood
P2860
P356
10.1097/SHK.0000000000000478
P577
2015-10-28T00:00:00Z