Control of alternative pre-mRNA splicing by Ca(++) signals.
about
Dynamic changes in neurexins' alternative splicing: role of Rho-associated protein kinases and relevance to memory formationDigoxin suppresses HIV-1 replication by altering viral RNA processingControl of alternative splicing by forskolin through hnRNP K during neuronal differentiationA conserved serine of heterogeneous nuclear ribonucleoprotein L (hnRNP L) mediates depolarization-regulated alternative splicing of potassium channelsNFAT1 and NFAT3 cooperate with HDAC4 during regulation of alternative splicing of PMCA isoforms in PC12 cellsMorphine and MK-801 administration leads to alternative N-methyl-D-aspartate receptor 1 splicing and associated changes in reward seeking behavior and nociception on an operant orofacial assay.A G-tract element in apoptotic agents-induced alternative splicing.In vivo selection of kinase-responsive RNA elements controlling alternative splicing.Ca2+-signaling, alternative splicing and endoplasmic reticulum stress responses.Emerging mechanisms and consequences of calcium regulation of alternative splicing in neurons and endocrine cells.Aberrant splicing in neurological diseases.Mechano-regulation of alternative splicing.Differential evolution of signal-responsive RNA elements and upstream factors that control alternative splicing.Exonal elements and factors involved in the depolarization-induced alternative splicing of neurexin 2.Differential effects of PKA-controlled CaMKK2 variants on neuronal differentiationDepolarization-mediated regulation of alternative splicing.Activity-mediated AMPA receptor remodeling, driven by alternative splicing in the ligand-binding domainThe heterogeneous nuclear ribonucleoprotein L is an essential component in the Ca2+/calmodulin-dependent protein kinase IV-regulated alternative splicing through cytidine-adenosine repeats.Nuclear ALG-2 protein interacts with Ca2+ homeostasis endoplasmic reticulum protein (CHERP) Ca2+-dependently and participates in regulation of alternative splicing of inositol trisphosphate receptor type 1 (IP3R1) pre-mRNA.Cardiac glycoside/aglycones inhibit HIV-1 gene expression by a mechanism requiring MEK1/2-ERK1/2 signaling.
P2860
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P2860
Control of alternative pre-mRNA splicing by Ca(++) signals.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh
2008年學術文章
@zh-hant
name
Control of alternative pre-mRNA splicing by Ca(++) signals.
@ast
Control of alternative pre-mRNA splicing by Ca(++) signals.
@en
type
label
Control of alternative pre-mRNA splicing by Ca(++) signals.
@ast
Control of alternative pre-mRNA splicing by Ca(++) signals.
@en
prefLabel
Control of alternative pre-mRNA splicing by Ca(++) signals.
@ast
Control of alternative pre-mRNA splicing by Ca(++) signals.
@en
P2860
P1476
Control of alternative pre-mRNA splicing by Ca(++) signals
@en
P2093
Jiuyong Xie
P2860
P304
P356
10.1016/J.BBAGRM.2008.01.003
P407
P50
P577
2008-01-17T00:00:00Z