Neurorestoration after traumatic brain injury through angiotensin II receptor blockage - Test2 - elhamkhani - TriplyDB

Neurorestoration after traumatic brain injury through angiotensin II receptor blockage

Neurorestoration after traumatic brain injury through angiotensin II receptor blockage

http://www.wikidata.org/entity/Q36515931

about

Type-1 angiotensin receptor signaling in central nervous system myeloid cells is pathogenic during fatal alphavirus encephalitis in mice Chronic cerebrovascular dysfunction after traumatic brain injury Hepatic expression of serum amyloid A1 is induced by traumatic brain injury and modulated by telmisartan NOX2 deficiency alters macrophage phenotype through an IL-10/STAT3 dependent mechanism: implications for traumatic brain injury Evidence to Consider Angiotensin II Receptor Blockers for the Treatment of Early Alzheimer's Disease.Temporal Changes in Cortical and Hippocampal Expression of Genes Important for Brain Glucose Metabolism Following Controlled Cortical Impact Injury in Mice.Angiotensin II Causes Neuronal Damage in Stretch-Injured Neurons: Protective Effects of Losartan, an Angiotensin T1 Receptor Blocker.Sexual dimorphism in the inflammatory response to traumatic brain injury.Neurotoxicity of Zinc.Roles of Peroxisome Proliferator-Activated Receptor Gamma on Brain and Peripheral Inflammation.Previous physical exercise alters the hepatic profile of oxidative-inflammatory status and limits the secondary brain damage induced by severe traumatic brain injury in rats.Cell-based and pharmacological neurorestorative therapies for ischemic stroke.Telmisartan reduced cerebral edema by inhibiting NLRP3 inflammasome in mice with cold brain injury.Within the Brain: The Renin Angiotensin System.Does inhibition of angiotensin function cause neuroprotection in diffuse traumatic brain injury?decoction improves neurological dysfunction by increasing synapsin expression after traumatic brain injury Brain injury induces HIF-1α-dependent transcriptional activation of LRRK2 that exacerbates brain damage

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P2860

Neurorestoration after traumatic brain injury through angiotensin II receptor blockage

http://www.wikidata.org/entity/Q36515931

description

2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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2015年论文

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2015年论文

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name

Neurorestoration after traumatic brain injury through angiotensin II receptor blockage

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Neurorestoration after traumatic brain injury through angiotensin II receptor blockage

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type

label

Neurorestoration after traumatic brain injury through angiotensin II receptor blockage

@ast

Neurorestoration after traumatic brain injury through angiotensin II receptor blockage

@en

prefLabel

Neurorestoration after traumatic brain injury through angiotensin II receptor blockage

@ast

Neurorestoration after traumatic brain injury through angiotensin II receptor blockage

@en

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P1476

Neurorestoration after traumatic brain injury through angiotensin II receptor blockage

@en

P2093

Juan M Saavedra

http://www.w3.org/2001/XMLSchema#string

Kwame Affram

http://www.w3.org/2001/XMLSchema#string

María G Balarezo

http://www.w3.org/2001/XMLSchema#string

Sonia Villapol

http://www.w3.org/2001/XMLSchema#string

P2860

Differential clinical profile of candesartan compared to other angiotensin receptor blockers

Use of angiotensin receptor blockers and risk of dementia in a predominantly male population: prospective cohort analysis

Clinical characteristics and pathophysiological mechanisms of focal and diffuse traumatic brain injury

Vascular protection by angiotensin receptor antagonism involves differential VEGF expression in both hemispheres after experimental stroke

Candesartan, an angiotensin II AT₁-receptor blocker and PPAR-γ agonist, reduces lesion volume and improves motor and memory function after traumatic brain injury in mice.

Angiotensin receptor type 2 activation induces neuroprotection and neurogenesis after traumatic brain injury.

Cloning, characterization, and expression of two angiotensin receptor (AT-1) isoforms from the mouse genome.

Postischemic administration of angiotensin II type 1 receptor blocker reduces cerebral infarction size in hypertensive rats.

Cognitive deficit in amyloid-beta-injected mice was improved by pretreatment with a low dose of telmisartan partly because of peroxisome proliferator-activated receptor-gamma activation.

Unilateral blood flow decrease induces bilateral and symmetric responses in the immature brain.

P304

3299-3315

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scholarly article

P356

10.1093/BRAIN/AWV172

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P433

Pt 11

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138

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P577

2015-06-26T00:00:00Z

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279300904

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26115674

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P921

traumatic brain injury

P932

4731413

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