Lipid traffic between high density lipoproteins and Plasmodium falciparum-infected red blood cells.
about
Cultivation of plasmodium spp.Uptake of proteins and degradation of human serum albumin by Plasmodium falciparum-infected human erythrocytesRole of cholesterol in parasitic infectionsCopper Homeostasis for the Developmental Progression of Intraerythrocytic Malarial ParasiteProbucol-Induced α-Tocopherol Deficiency Protects Mice against Malaria InfectionBee venom phospholipase A2 induces stage-specific growth arrest of the intraerythrocytic Plasmodium falciparum via modifications of human serum components.Inward cholesterol gradient of the membrane system in P. falciparum-infected erythrocytes involves a dilution effect from parasite-produced lipids.Sphingolipid synthesis as a target for chemotherapy against malaria parasites.Cerebral malaria: insights from host-parasite protein-protein interactionsRecent insights into fatty acid acquisition and metabolism in malarial parasites.Membrane transport in the malaria-infected erythrocyte.Metabolomics and malaria biologyProtein and lipid trafficking induced in erythrocytes infected by malaria parasites.Inhibition of the Growth of Plasmodium falciparum in Culture by Stearylamine-Phosphatidylcholine Liposomes.Interactions between docetaxel (Taxotere) and Plasmodium falciparum-infected erythrocytes.3-Hydroxy-3-methylglutaryl coenzyme A reductase inhibitors lovastatin and simvastatin inhibit in vitro development of Plasmodium falciparum and Babesia divergens in human erythrocytes.Maternal malaria status and metabolic profiles in pregnancy and in cord blood: relationships with birth size in Nigerian infants.Serum lipoproteins promote efficient presentation of the malaria virulence protein PfEMP1 at the erythrocyte surface.Plasmodium falciparum exports the Golgi marker sphingomyelin synthase into a tubovesicular network in the cytoplasm of mature erythrocytes.Early prediction of cerebral malaria by (1)H NMR based metabolomicsCryptosporidium parvum scavenges LDL-derived cholesterol and micellar cholesterol internalized into enterocytes.Lipid parameters in a hyperendemic area for malariaIsoprenoid metabolism in apicomplexan parasites.Protein sorting in Plasmodium falciparum-infected red blood cells permeabilized with the pore-forming protein streptolysin O.Parasite-regulated membrane transport processes and metabolic control in malaria-infected erythrocytes.A high-density-lipoprotein receptor appears to mediate the transfer of essential fatty acids from high-density lipoprotein to lymphocytes.Modulation of Lipoprotein Cholesterol Levels in Plasmodium berghei Malarial Infection by Crude Aqueous Extract of Ganoderma lucidumHost plasma low density lipoprotein particles as an essential source of lipids for the bloodstream forms of Trypanosoma brucei.Trafficking of malarial proteins to the host cell cytoplasm and erythrocyte surface membrane involves multiple pathwaysEthoxyquin-induced resistance to aflatoxin B1 in the rat is associated with the expression of a novel alpha-class glutathione S-transferase subunit, Yc2, which possesses high catalytic activity for aflatoxin B1-8,9-epoxide.Active isoprenoid pathway in the intra-erythrocytic stages of Plasmodium falciparum: presence of dolichols of 11 and 12 isoprene units.Neutral sphingomyelinase activity dependent on Mg2+ and anionic phospholipids in the intraerythrocytic malaria parasite Plasmodium falciparum.Continuous cultivation of intraerythrocytic Plasmodium falciparum in a serum-free medium with the use of a growth-promoting factor.Hypoxanthine: a low molecular weight factor essential for growth of erythrocytic Plasmodium falciparum in a serum-free medium.Selective cytotoxicity of dermaseptin S3 toward intraerythrocytic Plasmodium falciparum and the underlying molecular basis.Direct interaction of dermaseptin S4 aminoheptanoyl derivative with intraerythrocytic malaria parasite leading to increased specific antiparasitic activity in culture.Preliminary Evaluation of Artemisinin-Cholesterol Conjugates as Potential Drugs for the Treatment of Intractable Forms of Malaria and Tuberculosis.Testosterone-induced changes in phosphatidylcholine molecular species composition of Plasmodium chabaudi-infected erythrocytes.Intraerythrocytic Plasmodium falciparum utilize a broad range of serum-derived fatty acids with limited modification for their growth.Characterization of membrane proteins exported from Plasmodium falciparum into the host erythrocyte.
P2860
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P2860
Lipid traffic between high density lipoproteins and Plasmodium falciparum-infected red blood cells.
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
1991年论文
@zh
1991年论文
@zh-cn
name
Lipid traffic between high den ...... arum-infected red blood cells.
@ast
Lipid traffic between high den ...... arum-infected red blood cells.
@en
type
label
Lipid traffic between high den ...... arum-infected red blood cells.
@ast
Lipid traffic between high den ...... arum-infected red blood cells.
@en
prefLabel
Lipid traffic between high den ...... arum-infected red blood cells.
@ast
Lipid traffic between high den ...... arum-infected red blood cells.
@en
P2093
P2860
P356
P1476
Lipid traffic between high den ...... arum-infected red blood cells.
@en
P2093
Fruchart JC
Grellier P
Rigomier D
Schrevel J
P2860
P304
P356
10.1083/JCB.112.2.267
P407
P577
1991-01-01T00:00:00Z