Overexpression of the cellular retinoic acid binding protein-I (CRABP-I) results in a reduction in differentiation-specific gene expression in F9 teratocarcinoma cells.
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Identification and characterization of a retinoid-induced class II tumor suppressor/growth regulatory geneLoss of CAK phosphorylation of RAR{alpha} mediates transcriptional control of retinoid-induced cancer cell differentiationMembers of the fatty acid binding protein family are differentiation factors for the mammary glandRetinoid-binding proteins: mediators of retinoid actionDirect channeling of retinoic acid between cellular retinoic acid-binding protein II and retinoic acid receptor sensitizes mammary carcinoma cells to retinoic acid-induced growth arrestRetinoic acid receptors and retinoid X receptors: interactions with endogenous retinoic acidsIntracellular transport of fat-soluble vitamins A and EStructural analysis of site-directed mutants of cellular retinoic acid-binding protein II addresses the relationship between structural integrity and ligand bindingDistinct patterns of all-trans retinoic acid dependent expression of HOXB and HOXC homeogenes in human embryonal and small-cell lung carcinoma cell linesInvolvement of retinoic acid receptor alpha in the stimulation of tissue-type plasminogen-activator gene expression in human endothelial cellsRetinoic acid is a potential dorsalising signal in the late embryonic chick hindbrain.Characterization of a fusion cDNA (RARA/myl) transcribed from the t(15;17) translocation breakpoint in acute promyelocytic leukemia.Genomic cloning of hGSTP1*C, an allelic human Pi class glutathione S-transferase gene variant and functional characterization of its retinoic acid response elements.All trans retinoic acid with low dose cytosine arabinoside in the treatment of myelodysplastic syndrome.CRABP1 provides high malignancy of transformed mesenchymal cells and contributes to the pathogenesis of mesenchymal and neuroendocrine tumors.A mutation in segment I-S6 alters slow inactivation of sodium channels.CRABP I expression and the mediation of the sensitivity of human tumour cells to retinoic acid and irradiation.Cellular retinoic acid binding protein I: expression and functional influence in renal cell carcinoma.SIRT1-mediated deacetylation of CRABPII regulates cellular retinoic acid signaling and modulates embryonic stem cell differentiation.A mid-life vitamin A supplementation prevents age-related spatial memory deficits and hippocampal neurogenesis alterations through CRABP-I.The present status in all-trans retinoic acid (ATRA) treatment for acute promyelocytic leukemia patients: further understanding and comprehensive strategy are required in the future.Physiological insights into all-trans-retinoic acid biosynthesis.CRABP1 is associated with a poor prognosis in breast cancer: adding to the complexity of breast cancer cell response to retinoic acid.Expression of cellular retinol- and cellular retinoic acid-binding proteins in the rat cervical epithelium is regulated by endocrine stimuli during normal squamous metaplasiaCellular retinoic acid-binding proteins are essential for hindbrain patterning and signal robustness in zebrafish.New drugs in acute myelogenous leukemia: a review.RARalpha-PLZF overcomes PLZF-mediated repression of CRABPI, contributing to retinoid resistance in t(11;17) acute promyelocytic leukemia.Regulation of Ras-MAPK pathway mitogenic activity by restricting nuclear entry of activated MAPK in endoderm differentiation of embryonic carcinoma and stem cellsLIF removal increases CRABPI and CRABPII transcripts in embryonic stem cells cultured in retinol or 4-oxoretinol.Retinoic acid induces neurogenesis by activating both retinoic acid receptors (RARs) and peroxisome proliferator-activated receptor β/δ (PPARβ/δ)All-trans-retinol is a ligand for the retinoic acid receptors.Targeted disruption of retinoic acid receptor alpha (RAR alpha) and RAR gamma results in receptor-specific alterations in retinoic acid-mediated differentiation and retinoic acid metabolism.Loss of retinoic acid receptor gamma function in F9 cells by gene disruption results in aberrant Hoxa-1 expression and differentiation upon retinoic acid treatment.Enzymology of retinoic acid biosynthesis and degradation.Disruption of the interaction between transcriptional intermediary factor 1{beta} and heterochromatin protein 1 leads to a switch from DNA hyper- to hypomethylation and H3K9 to H3K27 trimethylation on the MEST promoter correlating with gene reactivaHow degrading: Cyp26s in hindbrain developmentAssociation of the transcriptional corepressor TIF1beta with heterochromatin protein 1 (HP1): an essential role for progression through differentiation4-Oxoretinol, a new natural ligand and transactivator of the retinoic acid receptors.Dissection of the critical binding determinants of cellular retinoic acid binding protein II by mutagenesis and fluorescence binding assay.Arginine 132 of cellular retinoic acid-binding protein (type II) is important for binding of retinoic acid.
P2860
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P2860
Overexpression of the cellular retinoic acid binding protein-I (CRABP-I) results in a reduction in differentiation-specific gene expression in F9 teratocarcinoma cells.
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
1991年论文
@zh
1991年论文
@zh-cn
name
Overexpression of the cellular ...... n in F9 teratocarcinoma cells.
@ast
Overexpression of the cellular ...... n in F9 teratocarcinoma cells.
@en
type
label
Overexpression of the cellular ...... n in F9 teratocarcinoma cells.
@ast
Overexpression of the cellular ...... n in F9 teratocarcinoma cells.
@en
prefLabel
Overexpression of the cellular ...... n in F9 teratocarcinoma cells.
@ast
Overexpression of the cellular ...... n in F9 teratocarcinoma cells.
@en
P2860
P356
P1476
Overexpression of the cellular ...... n in F9 teratocarcinoma cells.
@en
P2093
P2860
P304
P356
10.1083/JCB.112.5.965
P407
P577
1991-03-01T00:00:00Z