Succination is Increased on Select Proteins in the Brainstem of the NADH dehydrogenase (ubiquinone) Fe-S protein 4 (Ndufs4) Knockout Mouse, a Model of Leigh Syndrome.
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Abnormal Hypermethylation of the VDAC2 Promoter is a Potential Cause of Idiopathic Asthenospermia in Men.VDAC-2: Mitochondrial outer membrane regulator masquerading as a channel?Succination of Protein Disulfide Isomerase Links Mitochondrial Stress and Endoplasmic Reticulum Stress in the Adipocyte During Diabetes.Therapeutic effects of the mitochondrial ROS-redox modulator KH176 in a mammalian model of Leigh Disease.Neuroproteomics: How Many Angels can be Identified in an Extract from the Head of a Pin?Mitochondrial VDAC2 and cell homeostasis: highlighting hidden structural features and unique functionalities.An Essential Postdevelopmental Role for Lis1 in Mice.Identification of a metabolic disposal route for the oncometabolite S-(2-succino)cysteine in Bacillus subtilis.
P2860
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P2860
Succination is Increased on Select Proteins in the Brainstem of the NADH dehydrogenase (ubiquinone) Fe-S protein 4 (Ndufs4) Knockout Mouse, a Model of Leigh Syndrome.
description
2015 nî lūn-bûn
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2015年の論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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name
Succination is Increased on Se ...... se, a Model of Leigh Syndrome.
@ast
Succination is Increased on Se ...... se, a Model of Leigh Syndrome.
@en
type
label
Succination is Increased on Se ...... se, a Model of Leigh Syndrome.
@ast
Succination is Increased on Se ...... se, a Model of Leigh Syndrome.
@en
prefLabel
Succination is Increased on Se ...... se, a Model of Leigh Syndrome.
@ast
Succination is Increased on Se ...... se, a Model of Leigh Syndrome.
@en
P2093
P2860
P356
P1476
Succination is Increased on Se ...... se, a Model of Leigh Syndrome.
@en
P2093
Allison M Manuel
Anna C Clapper
Gerardo G Piroli
John E Baatz
Michael D Walla
Norma Frizzell
Richard D Palmiter
P2860
P304
P356
10.1074/MCP.M115.051516
P577
2015-10-08T00:00:00Z