Bortezomib enhances expression of effector molecules in anti-tumor CD8+ T lymphocytes by promoting Notch-nuclear factor-κB crosstalk.
about
Notching tumor: Signaling through Notch receptors improves antitumor T cell immunity.Bortezomib augments lymphocyte stimulatory cytokine signaling in the tumor microenvironment to sustain CD8+T cell antitumor function.Anti-Jagged Immunotherapy Inhibits MDSCs and Overcomes Tumor-Induced Tolerance.Regulation of CD8+ T Cells and Antitumor Immunity by Notch Signaling.Trial watch: Immunogenic cell death induction by anticancer chemotherapeutics
P2860
Bortezomib enhances expression of effector molecules in anti-tumor CD8+ T lymphocytes by promoting Notch-nuclear factor-κB crosstalk.
description
2015 nî lūn-bûn
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2015年の論文
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name
Bortezomib enhances expression ...... h-nuclear factor-κB crosstalk.
@ast
Bortezomib enhances expression ...... h-nuclear factor-κB crosstalk.
@en
type
label
Bortezomib enhances expression ...... h-nuclear factor-κB crosstalk.
@ast
Bortezomib enhances expression ...... h-nuclear factor-κB crosstalk.
@en
prefLabel
Bortezomib enhances expression ...... h-nuclear factor-κB crosstalk.
@ast
Bortezomib enhances expression ...... h-nuclear factor-κB crosstalk.
@en
P2093
P2860
P356
P1433
P1476
Bortezomib enhances expression ...... h-nuclear factor-κB crosstalk.
@en
P2093
Anil Shanker
David P Carbone
Duafalia F Dudimah
Menaka C Thounaojam
Mikhail M Dikov
Roman V Uzhachenko
Samuel T Pellom
P2860
P304
32439-32455
P356
10.18632/ONCOTARGET.5857
P407
P577
2015-09-09T00:00:00Z