Endothelin-converting enzymes degrade intracellular β-amyloid produced within the endosomal/lysosomal pathway and autophagosomes.
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Mechanisms of Aβ Clearance and Degradation by Glial CellsEndothelinCerebrovascular disease in ageing and Alzheimer's diseaseAAV2/1 CD74 Gene Transfer Reduces β-amyloidosis and Improves Learning and Memory in a Mouse Model of Alzheimer's Disease.Autologous transplantation of intestine-isolated glia cells improves neuropathology and restores cognitive deficits in β amyloid-induced neurodegenerationEndothelin@25 - new agonists, antagonists, inhibitors and emerging research frontiers: IUPHAR Review 12Opioid receptor function is regulated by post-endocytic peptide processing.Evidence of a novel mechanism for partial γ-secretase inhibition induced paradoxical increase in secreted amyloid β proteinComputational re-engineering of Amylin sequence with reduced amyloidogenic potential.Common mechanisms of Alzheimer's disease and ischemic stroke: the role of protein kinase C in the progression of age-related neurodegeneration.The endocytic pathway in microglia during health, aging and Alzheimer's disease.Oleocanthal enhances amyloid-β clearance from the brains of TgSwDI mice and in vitro across a human blood-brain barrier modelPathophysiology of Hypoperfusion of the Precuneus in Early Alzheimer's Disease.Amyloid-clearing proteins and their epigenetic regulation as a therapeutic target in Alzheimer's disease.Recent advances in autophagy-based neuroprotection.Endothelin-converting enzymes degrade α-synuclein and are reduced in dementia with Lewy bodies.Sex Hormone Decline and Amyloid β Synthesis, Transport and Clearance in the Brain.Plaque formation and the intraneuronal accumulation of β-amyloid in Alzheimer's disease.Hypoxia Affects Neprilysin Expression Through Caspase Activation and an APP Intracellular Domain-dependent Mechanism.Cathepsin B Improves ß-Amyloidosis and Learning and Memory in Models of Alzheimer's Disease.Endothelin-converting enzyme 2 differentially regulates opioid receptor activity.Calmodulin Binding Proteins and Alzheimer's Disease.Major amyloid-β-degrading enzymes, endothelin-converting enzyme-2 and neprilysin, are expressed by distinct populations of GABAergic interneurons in hippocampus and neocortex.Alzheimer disease: modeling an Aβ-centered biological network.Androgen alleviates neurotoxicity of β-amyloid peptide (Aβ) by promoting microglial clearance of Aβ and inhibiting microglial inflammatory response to Aβ.
P2860
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P2860
Endothelin-converting enzymes degrade intracellular β-amyloid produced within the endosomal/lysosomal pathway and autophagosomes.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Endothelin-converting enzymes ...... al pathway and autophagosomes.
@ast
Endothelin-converting enzymes ...... al pathway and autophagosomes.
@en
type
label
Endothelin-converting enzymes ...... al pathway and autophagosomes.
@ast
Endothelin-converting enzymes ...... al pathway and autophagosomes.
@en
prefLabel
Endothelin-converting enzymes ...... al pathway and autophagosomes.
@ast
Endothelin-converting enzymes ...... al pathway and autophagosomes.
@en
P2860
P356
P1476
Endothelin-converting enzymes ...... mal pathway and autophagosomes
@en
P2093
Javier Pacheco-Quinto
P2860
P304
P356
10.1074/JBC.M112.422964
P407
P577
2013-01-02T00:00:00Z