Two factors that bind to highly conserved sequences in mammalian type C retroviral enhancers.
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Cellular transcription factor ZASC1 regulates murine leukemia virus transcriptionThe E47 transcription factor binds to the enhancer sequences of recombinant murine leukemia viruses and influences enhancer functionSequence-specific and/or stereospecific constraints of the U3 enhancer elements of MCF 247-W are important for pathogenicity.Members of the GATA family of transcription factors bind to the U3 region of Cas-Br-E and graffi retroviruses and transactivate their expression.In vivo footprinting of the enhancer sequences in the upstream long terminal repeat of Moloney murine leukemia virus: differential binding of nuclear factors in different cell typesMobilization and mechanism of transcription of integrated self-inactivating lentiviral vectors.The feline leukemia virus long terminal repeat contains a potent genetic determinant of T-cell lymphomagenicityIdentification of ETS domain proteins in murine T lymphocytes that interact with the Moloney murine leukemia virus enhancer.Characterization of a protein that binds multiple sequences in mammalian type C retrovirus enhancers.The t(8;21) fusion protein interferes with AML-1B-dependent transcriptional activation.Mutation of all Runx (AML1/core) sites in the enhancer of T-lymphomagenic SL3-3 murine leukemia virus unmasks a significant potential for myeloid leukemia induction and favors enhancer evolution toward induction of other disease patterns.Transactivation of the Moloney murine leukemia virus and T-cell receptor beta-chain enhancers by cbf and ets requires intact binding sites for both proteins.Transcriptional activity of core binding factor-alpha (AML1) and beta subunits on murine leukemia virus enhancer cores.Suppressor mutations within the core binding factor (CBF/AML1) binding site of a T-cell lymphomagenic retrovirus.Importance of a c-Myb binding site for lymphomagenesis by the retrovirus SL3-3.The potent enhancer activity of the polycythemic strain of spleen focus-forming virus in hematopoietic cells is governed by a binding site for Sp1 in the upstream control region and by a unique enhancer core motif, creating an exclusive target for PZASC1 knockout mice exhibit an early bone marrow-specific defect in murine leukemia virus replication.Novel insights into the pathogenesis of the Graffi murine leukemia retrovirus.Basic helix-loop-helix proteins in murine type C retrovirus transcriptional regulation.
P2860
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P2860
Two factors that bind to highly conserved sequences in mammalian type C retroviral enhancers.
description
1993 nî lūn-bûn
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1993年の論文
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name
Two factors that bind to highl ...... n type C retroviral enhancers.
@ast
Two factors that bind to highl ...... n type C retroviral enhancers.
@en
type
label
Two factors that bind to highl ...... n type C retroviral enhancers.
@ast
Two factors that bind to highl ...... n type C retroviral enhancers.
@en
prefLabel
Two factors that bind to highl ...... n type C retroviral enhancers.
@ast
Two factors that bind to highl ...... n type C retroviral enhancers.
@en
P2093
P2860
P1433
P1476
Two factors that bind to highl ...... n type C retroviral enhancers.
@en
P2093
P2860
P304
P407
P577
1993-04-01T00:00:00Z