Temporal neutrophil polarization following myocardial infarction
about
Immune-Mediated Repair: A Matter of Plasticity.Maturing Glycoproteomics Technologies Provide Unique Structural Insights into the N-glycoproteome and Its Regulation in Health and Disease.Heart regeneration and repair after myocardial infarction: translational opportunities for novel therapeutics.Leukocyte diversity in resolving and nonresolving mechanisms of cardiac remodeling.The life cycle of phagosomes: formation, maturation, and resolution.Recent advances in understanding neutrophils.The barrier hypothesis and Oncostatin M: Restoration of epithelial barrier function as a novel therapeutic strategy for the treatment of type 2 inflammatory disease.Fractional laser exposure induces neutrophil infiltration (N1 phenotype) into the tumor and stimulates systemic anti-tumor immune response.Defining the sham environment for post-myocardial infarction studies in miceExpression and Role of the Calcium-Sensing Receptor in Rat Peripheral Blood Polymorphonuclear Neutrophils.Early matrix metalloproteinase-9 inhibition post-myocardial infarction worsens cardiac dysfunction by delaying inflammation resolution.Altered Expression of TXNIP in the peripheral leukocytes of patients with coronary atherosclerotic heart disease.Macrophage overexpression of matrix metalloproteinase-9 in aged mice improves diastolic physiology and cardiac wound healing after myocardial infarction.Cardiac macrophage biology in the steady-state heart, the aging heart, and following myocardial infarction.Inflammation following acute myocardial infarction: Multiple players, dynamic roles, and novel therapeutic opportunities.Neutrophils are a major source of the epithelial barrier disrupting cytokine oncostatin M in patients with mucosal airways disease.Partners in crime: neutrophils and monocytes/macrophages in inflammation and disease.Neutrophils in tissue injury and repair.Guidelines for Experimental Models of Myocardial Ischemia and Infarction.Activation of the sympathetic nervous system modulates neutrophil function.The neutrophil: one cell on many missions or many cells with different agendas?Adenosine Production by Biomaterial-Supported Mesenchymal Stromal Cells Reduces the Innate Inflammatory Response in Myocardial Ischemia/Reperfusion Injury.Immune mechanisms in heart failure.Pathogenesis of Recalcitrant Chronic Rhinosinusitis: The Emerging Role of Innate Immune Cells.Parallels between vertebrate cardiac and cutaneous wound healing and regenerationLXR/RXR signaling and neutrophil phenotype following myocardial infarction classify sex differences in remodeling
P2860
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P2860
Temporal neutrophil polarization following myocardial infarction
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Temporal neutrophil polarization following myocardial infarction
@ast
Temporal neutrophil polarization following myocardial infarction
@en
type
label
Temporal neutrophil polarization following myocardial infarction
@ast
Temporal neutrophil polarization following myocardial infarction
@en
prefLabel
Temporal neutrophil polarization following myocardial infarction
@ast
Temporal neutrophil polarization following myocardial infarction
@en
P2093
P2860
P50
P356
P1476
Temporal neutrophil polarization following myocardial infarction
@en
P2093
Courtney A Cates
Elizabeth R Flynn
Jeffrey Henry
Presley L Cannon
Rugmani Padmanabhan Iyer
Yonggang Ma
P2860
P356
10.1093/CVR/CVW024
P577
2016-01-29T00:00:00Z