Array painting reveals a high frequency of balanced translocations in breast cancer cell lines that break in cancer-relevant genes.
about
Rif1 provides a new DNA-binding interface for the Bloom syndrome complex to maintain normal replicationIdentification of somatically acquired rearrangements in cancer using genome-wide massively parallel paired-end sequencingTargeted Cancer Therapy: Vital Oncogenes and a New Molecular Genetic Paradigm for Cancer Initiation Progression and TreatmentRVD2: an ultra-sensitive variant detection model for low-depth heterogeneous next-generation sequencing data.High-resolution array CGH clarifies events occurring on 8p in carcinogenesisPICNIC: an algorithm to predict absolute allelic copy number variation with microarray cancer data.Array painting: a protocol for the rapid analysis of aberrant chromosomes using DNA microarrays.Laser excitation power and the flow cytometric resolution of complex karyotypes.Characterization of genetic rearrangements in esophageal squamous carcinoma cell lines by a combination of M-FISH and array-CGH: further confirmation of some split genomic regions in primary tumors.Structural analysis of the genome of breast cancer cell line ZR-75-30 identifies twelve expressed fusion genes.Gene rearrangements in hormone receptor negative breast cancers revealed by mate pair sequencing.The relative timing of mutations in a breast cancer genome.Cancer gene discovery in mouse and man.Mouse Rif1 is a key regulator of the replication-timing programme in mammalian cells.Are breast cancers driven by fusion genes?Back to the origin: reconsidering replication, transcription, epigenetics, and cell cycle control.Life stage differences in mammary gland gene expression profile in non-human primates.Mammalian Rif1 contributes to replication stress survival and homology-directed repair.Epigenetic mechanisms in senescence, immortalisation and cancer.Characterising chromosome rearrangements: recent technical advances in molecular cytogeneticsForkhead Box P family members at the crossroad between tolerance and immunity: a balancing act.Large duplications at reciprocal translocation breakpoints that might be the counterpart of large deletions and could arise from stalled replication bubbles.Single-molecule analysis of genome rearrangements in cancer.Estimation of rearrangement phylogeny for cancer genomes.High-throughput analysis of chromosome translocations and other genome rearrangements in epithelial cancersStrong expression of the neuronal transcription factor FOXP2 is linked to an increased risk of early PSA recurrence in ERG fusion-negative cancers.Translocation Breakpoints Preferentially Occur in Euchromatin and Acrocentric Chromosomes.TFEB-VEGFA (6p21.1) co-amplified renal cell carcinoma: a distinct entity with potential implications for clinical management.Downregulation of FOXP2 promoter human hepatocellular carcinoma cell invasion.The untold stories of the speech gene, the FOXP2 cancer gene.
P2860
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P2860
Array painting reveals a high frequency of balanced translocations in breast cancer cell lines that break in cancer-relevant genes.
description
2007 nî lūn-bûn
@nan
2007年の論文
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2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Array painting reveals a high ...... reak in cancer-relevant genes.
@ast
Array painting reveals a high ...... reak in cancer-relevant genes.
@en
type
label
Array painting reveals a high ...... reak in cancer-relevant genes.
@ast
Array painting reveals a high ...... reak in cancer-relevant genes.
@en
prefLabel
Array painting reveals a high ...... reak in cancer-relevant genes.
@ast
Array painting reveals a high ...... reak in cancer-relevant genes.
@en
P2093
P2860
P356
P1433
P1476
Array painting reveals a high ...... reak in cancer-relevant genes.
@en
P2093
J C Beavis
K D Howarth
K Ichimura
N P Carter
P A W Edwards
V P Collins
P2860
P2888
P304
P356
10.1038/SJ.ONC.1210993
P407
P577
2007-12-17T00:00:00Z