IF1 limits the apoptotic-signalling cascade by preventing mitochondrial remodelling.
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The Permeability Transition in Plant Mitochondria: The Missing LinkCell death disguised: The mitochondrial permeability transition pore as the c-subunit of the F(1)F(O) ATP synthasePathway of binding of the intrinsically disordered mitochondrial inhibitor protein to F1-ATPaseChanging perspective on oncometabolites: from metabolic signature of cancer to tumorigenic and immunosuppressive agentsRegulation of mitochondrial ATP synthase in cardiac pathophysiologyMitochondrial fusion and fission proteins as novel therapeutic targets for treating cardiovascular diseaseMitochondrial and postmitochondrial survival signaling in cancer.Role for the ATPase inhibitory factor 1 in the environmental carcinogen-induced Warburg phenotype.Regulation of the H+-ATP synthase by IF1: a role in mitohormesis.ATPase inhibitory factor 1 is a potential prognostic marker for the migration and invasion of glioma.The Mitochondrial Permeability Transition Pore: Channel Formation by F-ATP Synthase, Integration in Signal Transduction, and Role in Pathophysiology.ATPase inhibitory factor 1 expression is an independent prognostic factor in non-small cell lung cancerFunctional and morphological impact of ER stress on mitochondria.Molecular mechanisms of cell death: central implication of ATP synthase in mitochondrial permeability transition.Hitting the Bull's-Eye in Metastatic Cancers-NSAIDs Elevate ROS in Mitochondria, Inducing Malignant Cell Death.The c-Ring of the F1FO-ATP Synthase: Facts and Perspectives.In vivo inhibition of the mitochondrial H+-ATP synthase in neurons promotes metabolic preconditioning.Reciprocal activation between ATPase inhibitory factor 1 and NF-κB drives hepatocellular carcinoma angiogenesis and metastasis.The compound BTB06584 is an IF1 -dependent selective inhibitor of the mitochondrial F1 Fo-ATPase.Down-regulation of oxidative phosphorylation in the liver by expression of the ATPase inhibitory factor 1 induces a tumor-promoter metabolic stateCaenorhabditis elegans ATPase inhibitor factor 1 (IF1) MAI-2 preserves the mitochondrial membrane potential (Δψm) and is important to induce germ cell apoptosis.Mitochondrial IF₁ preserves cristae structure to limit apoptotic cell death signaling.Biological Implications of Differential Expression of Mitochondrial-Shaping Proteins in Parkinson's Disease.Reduction of the ATPase inhibitory factor 1 (IF1) leads to visual impairment in vertebrates.A Review of the Inhibition of the Mitochondrial ATP Synthase by IF1 : Reprogramming Energy Metabolism and Inducing MitohormesisThe cristae modulator Optic atrophy 1 requires mitochondrial ATP synthase oligomers to safeguard mitochondrial function
P2860
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P2860
IF1 limits the apoptotic-signalling cascade by preventing mitochondrial remodelling.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
IF1 limits the apoptotic-signalling cascade by preventing mitochondrial remodelling.
@en
type
label
IF1 limits the apoptotic-signalling cascade by preventing mitochondrial remodelling.
@en
prefLabel
IF1 limits the apoptotic-signalling cascade by preventing mitochondrial remodelling.
@en
P2093
P2860
P356
P1476
IF1 limits the apoptotic-signalling cascade by preventing mitochondrial remodelling.
@en
P2093
A Seraphim
D Faccenda
M Campanella
M R Duchen
P2860
P2888
P304
P356
10.1038/CDD.2012.163
P577
2013-01-25T00:00:00Z
P5875
P6179
1012616346