Hippocampal neurogenesis protects against cocaine-primed relapse.
about
Adult hippocampal neurogenesis in the pathogenesis of addiction and dual diagnosis disordersRole of NG2 expressing cells in addiction: a new approach for an old problemTransforming growth factor beta receptor 1 is increased following abstinence from cocaine self-administration, but not cocaine sensitizationThe Plasticity of Extinction: Contribution of the Prefrontal Cortex in Treating Addiction through Inhibitory LearningThe Interplay between the Hippocampus and Amygdala in Regulating Aberrant Hippocampal Neurogenesis during Protracted Abstinence from Alcohol Dependence.Effect of dehydroepiandrosterone add-on therapy on mood, decision making and subsequent relapse of polydrug users.Methamphetamine affects cell proliferation in the medial prefrontal cortex: a new niche for toxicity.Methamphetamine differentially affects BDNF and cell death factors in anatomically defined regions of the hippocampus.Activity-regulated gene expression in immature neurons in the dentate gyrus following re-exposure to a cocaine-paired environment.Pharmacological blockade of either cannabinoid CB1 or CB2 receptors prevents both cocaine-induced conditioned locomotion and cocaine-induced reduction of cell proliferation in the hippocampus of adult male rat.Long-lasting memory deficits in mice withdrawn from cocaine are concomitant with neuroadaptations in hippocampal basal activity, GABAergic interneurons and adult neurogenesisDentate gyrus neurogenesis ablation via cranial irradiation enhances morphine self-administration and locomotor sensitization.Adult Neurogenesis and Psychiatric Disorders.Evaluation of plasma cytokines in patients with cocaine use disorders in abstinence identifies transforming growth factor alpha (TGFα) as a potential biomarker of consumption and dual diagnosis.Neurogenesis during abstinence is necessary for context-driven methamphetamine-related memory.Pharmacological reduction of adult hippocampal neurogenesis modifies functional brain circuits in mice exposed to a cocaine conditioned place preference paradigm.A single exposure to cocaine during development elicits regionally-selective changes in basal basic Fibroblast Growth Factor (FGF-2) gene expression and alters the trophic response to a second injection.Rat-strain dependent changes of dendritic and spine morphology in the hippocampus after cocaine self-administration.Prefrontal gray matter volume recovery in treatment-seeking cocaine-addicted individuals: a longitudinal study.
P2860
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P2860
Hippocampal neurogenesis protects against cocaine-primed relapse.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Hippocampal neurogenesis protects against cocaine-primed relapse.
@en
type
label
Hippocampal neurogenesis protects against cocaine-primed relapse.
@en
prefLabel
Hippocampal neurogenesis protects against cocaine-primed relapse.
@en
P2093
P2860
P356
P1433
P1476
Hippocampal neurogenesis protects against cocaine-primed relapse.
@en
P2093
Chitra D Mandyam
Clara J Yuan
George F Koob
Jeffery C Sobieraj
Joel E Schlosburg
Leandro F Vendruscolo
Luis Diaz-Aguilar
Olivier Deschaux
Olivier George
P2860
P304
P356
10.1111/ADB.12019
P577
2012-12-24T00:00:00Z