Kidney tubules: intertubular, vascular, and glomerular cross-talk.
about
Restored nitric oxide bioavailability reduces the severity of acute-to-chronic transition in a mouse model of aristolochic acid nephropathy.Proximal tubule apical endocytosis is modulated by fluid shear stress via an mTOR-dependent pathway.Hyperactivation of the NLRP3 Inflammasome in Myeloid Cells Leads to Severe Organ Damage in Experimental Lupus.CD39-adenosinergic axis in renal pathophysiology and therapeutics.Inducible podocyte-specific deletion of CTCF drives progressive kidney disease and bone abnormalities.Tubulointerstitial fibrosis can sensitize the kidney to subsequent glomerular injury.Jagged1/Notch2 controls kidney fibrosis via Tfam-mediated metabolic reprogramming
P2860
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P2860
Kidney tubules: intertubular, vascular, and glomerular cross-talk.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Kidney tubules: intertubular, vascular, and glomerular cross-talk.
@en
type
label
Kidney tubules: intertubular, vascular, and glomerular cross-talk.
@en
prefLabel
Kidney tubules: intertubular, vascular, and glomerular cross-talk.
@en
P2860
P1476
Kidney tubules: intertubular, vascular, and glomerular cross-talk.
@en
P2093
Joseph V Bonventre
P2860
P304
P356
10.1097/MNH.0000000000000218
P577
2016-03-26T00:00:00Z