Proteasome activation by hepatitis C core protein is reversed by ethanol-induced oxidative stress
about
Ethanol-induced oxidant stress modulates hepatic autophagy and proteasome activityHepatitis C virus and ethanol alter antigen presentation in liver cellsChronic ethanol feeding affects proteasome-interacting proteinsTobacco smoking effect on HIV-1 pathogenesis: role of cytochrome P450 isozymes.Impaired methylation as a novel mechanism for proteasome suppression in liver cellsEthanol inhibits antigen presentation by dendritic cellsNetwork-based study reveals potential infection pathways of hepatitis-C leading to various diseasesAlcohol and hepatitis C virus--interactions in immune dysfunctions and liver damage.Early growth response-1 contributes to steatosis development after acute ethanol administration.Ethanol and hepatitis C virus suppress peptide-MHC class I presentation in hepatocytes by altering proteasome function.Preventing p38 MAPK-mediated MafA degradation ameliorates β-cell dysfunction under oxidative stress.Ethanol metabolism alters major histocompatibility complex class I-restricted antigen presentation in liver cellsChronic ethanol diet increases regulatory T-cell activity and inhibits hepatitis C virus core-specific cellular immune responses in mice.Effects of ethanol on the proteasome interacting proteins.Roles of the two distinct proteasome pathways in hepatitis C virus infection.Are human polyomaviruses co-factors for cancers induced by other oncoviruses?Proteasome- and ethanol-dependent regulation of HCV-infection pathogenesisHepatitis C, innate immunity and alcohol: friends or foes?Increased oxidative stress and toxicity in ADH and CYP2E1 overexpressing human hepatoma VL-17A cells exposed to high glucose.Methamphetamine oxidatively damages parkin and decreases the activity of 26S proteasome in vivo.In vitro evidence for chronic alcohol and high glucose mediated increased oxidative stress and hepatotoxicity.Prolonged feeding with guanidinoacetate, a methyl group consumer, exacerbates ethanol-induced liver injury.Alcoholic Liver Disease: Pathogenesis and Current Management.Hepatitis C and alcohol exacerbate liver injury by suppression of FOXO3.Amino acid substitution in the core protein has no impact on relapse in hepatitis C genotype 1 patients treated with peginterferon and ribavirin.Creatine Supplementation Does Not Prevent the Development of Alcoholic Steatosis.Amyloid-β Increases Activity of Proteasomes Capped with 19S and 11S Regulators.
P2860
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P2860
Proteasome activation by hepatitis C core protein is reversed by ethanol-induced oxidative stress
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Proteasome activation by hepat ...... hanol-induced oxidative stress
@en
type
label
Proteasome activation by hepat ...... hanol-induced oxidative stress
@en
prefLabel
Proteasome activation by hepat ...... hanol-induced oxidative stress
@en
P2093
P2860
P1433
P1476
Proteasome activation by hepat ...... hanol-induced oxidative stress
@en
P2093
Natalia A Osna
Ronda L White
Steven A Weinman
Terrence M Donohue
Viatcheslav M Krutik
P2860
P304
P356
10.1053/J.GASTRO.2008.02.063
P407
P577
2008-03-04T00:00:00Z