Clinical implications of the basic defects in Cockayne syndrome and xeroderma pigmentosum and the DNA lesions responsible for cancer, neurodegeneration and aging
about
Rad26p regulates the occupancy of histone H2A-H2B dimer at the active genes in vivoDiagnosis of Xeroderma Pigmentosum and Related DNA Repair-Deficient Cutaneous DiseasesE2F1 and p53 transcription factors as accessory factors for nucleotide excision repair.Sirt1 suppresses RNA synthesis after UV irradiation in combined xeroderma pigmentosum group D/Cockayne syndrome (XP-D/CS) cells.Haploinsufficiency in mouse models of DNA repair deficiency: modifiers of penetrance.Lack of XPC leads to a shift between respiratory complexes I and II but sensitizes cells to mitochondrial stress.Bone marrow transplantation improves the outcome of Atm-deficient mice through the migration of ATM-competent cells.Single-stranded DNA Binding by the Helix-Hairpin-Helix Domain of XPF Protein Contributes to the Substrate Specificity of the ERCC1-XPF Protein Complex.
P2860
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P2860
Clinical implications of the basic defects in Cockayne syndrome and xeroderma pigmentosum and the DNA lesions responsible for cancer, neurodegeneration and aging
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Clinical implications of the b ...... r, neurodegeneration and aging
@en
type
label
Clinical implications of the b ...... r, neurodegeneration and aging
@en
prefLabel
Clinical implications of the b ...... r, neurodegeneration and aging
@en
P2860
P1476
Clinical implications of the b ...... r, neurodegeneration and aging
@en
P2093
P2860
P304
P356
10.1016/J.MAD.2008.01.005
P577
2008-02-03T00:00:00Z