Cbl-b is a critical regulator of macrophage activation associated with obesity-induced insulin resistance in mice.
about
TAF7L modulates brown adipose tissue formation.Nedd4 haploinsufficient mice display moderate insulin resistance, enhanced lipolysis, and protection against high-fat diet-induced obesity.A novel CBL-Bflox/flox mouse model allows tissue-selective fully conditional CBL/CBL-B double-knockout: CD4-Cre mediated CBL/CBL-B deletion occurs in both T-cells and hematopoietic stem cells.Modulation of Immune Cell Functions by the E3 Ligase Cbl-b.Role of E3 ubiquitin ligases in insulin resistance.Involvement of Cbl-b-mediated macrophage inactivation in insulin resistance.CD14 gene silencing alters the microRNA expression profile of RAW264.7 cells stimulated by Brucella melitensis infection.Ubiquitylation of MFHAS1 by the ubiquitin ligase praja2 promotes M1 macrophage polarization by activating JNK and p38 pathways.Toll-like receptor 4 (TLR4) deficient mice are protected from adipose tissue inflammation in agingDDB1-Mediated CRY1 Degradation Promotes FOXO1-Driven Gluconeogenesis in Liver.
P2860
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P2860
Cbl-b is a critical regulator of macrophage activation associated with obesity-induced insulin resistance in mice.
description
2013 nî lūn-bûn
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2013年の論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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2013年论文
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2013年论文
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2013年论文
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name
Cbl-b is a critical regulator ...... ed insulin resistance in mice.
@en
type
label
Cbl-b is a critical regulator ...... ed insulin resistance in mice.
@en
prefLabel
Cbl-b is a critical regulator ...... ed insulin resistance in mice.
@en
P2093
P2860
P356
P1433
P1476
Cbl-b is a critical regulator ...... ced insulin resistance in mice
@en
P2093
Arisa Ochi
Atsuko Sakai
Ayako Ohno
Edward M Mills
Junji Terao
Kenro Utsunomiya
Motoko Oarada
Sachiko Kagawa
Shigetada Teshima-Kondo
Shohei Kohno
P2860
P304
P356
10.2337/DB12-0677
P407
P577
2013-01-24T00:00:00Z