The TNFα-Transgenic Rat: Hippocampal Synaptic Integrity, Cognition, Function, and Post-Ischemic Cell Loss.

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Infectious immunity in the central nervous system and brain function.Tumor Necrosis Factor and Interleukin-1β Modulate Synaptic Plasticity during Neuroinflammation.

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P2860

The TNFα-Transgenic Rat: Hippocampal Synaptic Integrity, Cognition, Function, and Post-Ischemic Cell Loss.

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2016 nî lūn-bûn

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年论文

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The TNFα-Transgenic Rat: Hippo ...... , and Post-Ischemic Cell Loss.

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The TNFα-Transgenic Rat: Hippo ...... , and Post-Ischemic Cell Loss.

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The TNFα-Transgenic Rat: Hippo ...... , and Post-Ischemic Cell Loss.

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The TNFα-Transgenic Rat: Hippo ...... , and Post-Ischemic Cell Loss.

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Christopher M Norris

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L Creed Pettigrew

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Richard J Kryscio

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P2860

TNF signaling inhibition in the CNS: implications for normal brain function and neurodegenerative disease

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Human T-cell lymphotropic virus type 1-infected T lymphocytes impair catabolism and uptake of glutamate by astrocytes via Tax-1 and tumor necrosis factor alpha

Effects of the cyclooxygenase-2 inhibitor nimesulide on cerebral infarction and neurological deficits induced by permanent middle cerebral artery occlusion in the rat

Evidence for the involvement of TNF and NF-kappaB in hippocampal synaptic plasticity

Inflammation and infection in clinical stroke

Differential regulation of AMPA receptor and GABA receptor trafficking by tumor necrosis factor-alpha

Reversible middle cerebral artery occlusion without craniectomy in rats

Tumor necrosis factor (TNF)-mediated neuroprotection against glutamate-induced excitotoxicity is enhanced by N-methyl-D-aspartate receptor activation. Essential role of a TNF receptor 2-mediated phosphatidylinositol 3-kinase-dependent NF-kappa B pat

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scholarly article

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10.1371/JOURNAL.PONE.0154721

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English

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The TNFα-Transgenic Rat: Hippocampal Synaptic Integrity, Cognition, Function, and Post-Ischemic Cell Loss.

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P2860

P2860

The TNFα-Transgenic Rat: Hippocampal Synaptic Integrity, Cognition, Function, and Post-Ischemic Cell Loss.

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