Short-term single treatment of chemotherapy results in the enrichment of ovarian cancer stem cell-like cells leading to an increased tumor burden - Test2 - elhamkhani - TriplyDB

Short-term single treatment of chemotherapy results in the enrichment of ovarian cancer stem cell-like cells leading to an increased tumor burden

Short-term single treatment of chemotherapy results in the enrichment of ovarian cancer stem cell-like cells leading to an increased tumor burden

http://www.wikidata.org/entity/Q36891437

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Role of mesenchymal cells in the natural history of ovarian cancer: a review Exploiting nanotechnology to overcome tumor drug resistance: Challenges and opportunities Bidirectional interconversion of stem and non-stem cancer cell populations: A reassessment of theoretical models for tumor heterogeneity Chemotherapy targeting cancer stem cells Withaferin a alone and in combination with cisplatin suppresses growth and metastasis of ovarian cancer by targeting putative cancer stem cells Knockdown of stem cell regulator Oct4A in ovarian cancer reveals cellular reprogramming associated with key regulators of cytoskeleton-extracellular matrix remodelling Inhibition of the JAK2/STAT3 pathway in ovarian cancer results in the loss of cancer stem cell-like characteristics and a reduced tumor burden Epithelial-to-Mesenchymal Transition in the Female Reproductive Tract: From Normal Functioning to Disease Pathology.Metformin against cancer stem cells through the modulation of energy metabolism: special considerations on ovarian cancer.Enhanced breast cancer therapy with nsPEFs and low concentrations of gemcitabine.Ovarian cancer stem cells express ROR1, which can be targeted for anti-cancer-stem-cell therapy.Ovarian tumor-initiating cells display a flexible metabolism Melanoma-initiating cells exploit M2 macrophage TGFβ and arginase pathway for survival and proliferation.Eradication of Human Ovarian Cancer Cells by Transgenic Expression of Recombinant DNASE1, DNASE1L3, DNASE2, and DFFB Controlled by EGFR Promoter: Novel Strategy for Targeted Therapy of Cancer.Enhanced expression of DNA polymerase eta contributes to cisplatin resistance of ovarian cancer stem cells.Cancer stem cells as a target population for drug discovery.Targeting tumor-initiating cells: eliminating anabolic cancer stem cells with inhibitors of protein synthesis or by mimicking caloric restriction.CD133+ ovarian cancer stem-like cells promote non-stem cancer cell metastasis via CCL5 induced epithelial-mesenchymal transition.Blockage of TGFβ-SMAD2 by demethylation-activated miR-148a is involved in caffeic acid-induced inhibition of cancer stem cell-like properties in vitro and in vivo.Breast cancer stem cell selectivity of synthetic nanomolar-active salinomycin analogs A critical role of Oct4A in mediating metastasis and disease-free survival in a mouse model of ovarian cancer.Staurosporine Induced Apoptosis May Activate Cancer Stem-Like Cells (CD44(+)/CD24(-)) in MCF-7 by Upregulating Mucin1 and EpCAM.Coalition of Oct4A and β1 integrins in facilitating metastasis in ovarian cancer.Accumulation of ALDH1-positive cells after neoadjuvant chemotherapy predicts treatment resistance and prognosticates poor outcome in ovarian cancer.Predictive significance of DNA damage and repair biomarkers in triple-negative breast cancer patients treated with neoadjuvant chemotherapy: An exploratory analysis.Role of stress-activated OCT4A in the cell fate decisions of embryonal carcinoma cells treated with etoposide.Enrichment for chemoresistant ovarian cancer stem cells from human cell lines.Low-dose cisplatin-induced CXCR4 expression promotes proliferation of ovarian cancer stem-like cells.Getting to know ovarian cancer ascites: opportunities for targeted therapy-based translational research.DOXIL when combined with Withaferin A (WFA) targets ALDH1 positive cancer stem cells in ovarian cancer Hepatocellular carcinoma stem cell-like cells are enriched following low-dose 5-fluorouracil chemotherapy.Cisplatin induces stemness in ovarian cancer.The bromodomain inhibitor OTX015 (MK-8628) exerts anti-tumor activity in triple-negative breast cancer models as single agent and in combination with everolimus.Targeted Disruption of the JAK2/STAT3 Pathway in Combination with Systemic Administration of Paclitaxel Inhibits the Priming of Ovarian Cancer Stem Cells Leading to a Reduced Tumor Burden.Induced cancer stem cells generated by radiochemotherapy and their therapeutic implications.Activating adaptive cellular mechanisms of resistance following sublethal cytotoxic chemotherapy: implications for diagnostic microdosing.Delivery of therapeutics using nanocarriers for targeting cancer cells and cancer stem cells.miRNAs as prognostic and therapeutic tools in epithelial ovarian cancer.Non-genetic cancer cell plasticity and therapy-induced stemness in tumour relapse: 'What does not kill me strengthens me'.miRNA-34c-5p inhibits amphiregulin-induced ovarian cancer stemness and drug resistance via downregulation of the AREG-EGFR-ERK pathway.

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Short-term single treatment of chemotherapy results in the enrichment of ovarian cancer stem cell-like cells leading to an increased tumor burden

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Identification and characterization of ovarian cancer-initiating cells from primary human tumors

E-cadherin's dark side: possible role in tumor progression

Class III β-tubulin and the cytoskeletal gateway for drug resistance in ovarian cancer

Cancer stem cell niche: the place to be

Molecular phenotyping of human ovarian cancer stem cells unravels the mechanisms for repair and chemoresistance

Overexpression of alpha(v)beta6 integrin in serous epithelial ovarian cancer regulates extracellular matrix degradation via the plasminogen activation cascade

Ovarian cancer side population defines cells with stem cell-like characteristics and Mullerian Inhibiting Substance responsiveness

Ovarian cancer stem-like side-population cells are tumourigenic and chemoresistant.

Hepatitis C virus induces the cannabinoid receptor 1.

Misregulated E-cadherin expression associated with an aggressive brain tumor phenotype.

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