HilD-mediated transcriptional cross-talk between SPI-1 and SPI-2.
about
Fructose-asparagine is a primary nutrient during growth of Salmonella in the inflamed intestineRegulatory principles governing Salmonella and Yersinia virulenceSalmonella effector proteins and host-cell responsesRegulation of bacterial virulence by Csr (Rsm) systemsReplication of Salmonella enterica Serovar Typhimurium in Human Monocyte-Derived MacrophagesTranscriptional priming of Salmonella Pathogenicity Island-2 precedes cellular invasionPoxA, YjeK, and Elongation Factor P Coordinately Modulate Virulence and Drug Resistance in Salmonella entericaThe expression of Longus type 4 pilus of enterotoxigenic Escherichia coli is regulated by LngR and LngS and by H-NS, CpxR and CRP global regulators.In silico clustering of Salmonella global gene expression data reveals novel genes co-regulated with the SPI-1 virulence genes through HilD.The Salmonella Pathogenicity Island (SPI) 1 contributes more than SPI2 to the colonization of the chicken by Salmonella enterica serovar Typhimurium.Control of Salmonella pathogenicity island-2 gene expression.Gre factors-mediated control of hilD transcription is essential for the invasion of epithelial cells by Salmonella enterica serovar Typhimurium.QseC mediates Salmonella enterica serovar typhimurium virulence in vitro and in vivo.Salmonella enterica replication in hemophagocytic macrophages requires two type three secretion systems.The architecture and ppGpp-dependent expression of the primary transcriptome of Salmonella Typhimurium during invasion gene expression.Crosstalk between virulence loci: regulation of Salmonella enterica pathogenicity island 1 (SPI-1) by products of the std fimbrial operon.Role of cross talk in regulating the dynamic expression of the flagellar Salmonella pathogenicity island 1 and type 1 fimbrial genesSalmonella enterica serovar typhimurium colonizing the lumen of the chicken intestine grows slowly and upregulates a unique set of virulence and metabolism genes.A fundamental regulatory mechanism operating through OmpR and DNA topology controls expression of Salmonella pathogenicity islands SPI-1 and SPI-2Transcriptional regulator PerA influences biofilm-associated, platelet binding, and metabolic gene expression in Enterococcus faecalisSalmonella enterica serovar Typhimurium skills to succeed in the host: virulence and regulation.Salmonella phage ST64B encodes a member of the SseK/NleB effector familyIntegration of a complex regulatory cascade involving the SirA/BarA and Csr global regulatory systems that controls expression of the Salmonella SPI-1 and SPI-2 virulence regulons through HilDThe primary transcriptome of Salmonella enterica Serovar Typhimurium and its dependence on ppGpp during late stationary phaseSelected lactic acid-producing bacterial isolates with the capacity to reduce Salmonella translocation and virulence gene expression in chickens.Elongation factor P mediates a novel post-transcriptional regulatory pathway critical for bacterial virulenceRNA-seq Brings New Insights to the Intra-Macrophage Transcriptome of Salmonella TyphimuriumA multi-drug resistant Salmonella Typhimurium ST213 human-invasive strain (33676) containing the bla CMY-2 gene on an IncF plasmid is attenuated for virulence in BALB/c mice.The two-component system CpxR/A represses the expression of Salmonella virulence genes by affecting the stability of the transcriptional regulator HilDThe Impact of 18 Ancestral and Horizontally-Acquired Regulatory Proteins upon the Transcriptome and sRNA Landscape of Salmonella enterica serovar Typhimurium.Obacunone represses Salmonella pathogenicity islands 1 and 2 in an envZ-dependent fashionInterplay between the QseC and QseE bacterial adrenergic sensor kinases in Salmonella enterica serovar Typhimurium pathogenesisMapping the Regulatory Network for Salmonella enterica Serovar Typhimurium Invasion.Upward mobility and alternative lifestyles: a report from the 10th biennial meeting on Bacterial Locomotion and Signal TransductionIn silico identification and experimental characterization of regulatory elements controlling the expression of the Salmonella csrB and csrC genesIdentification of HilD-regulated genes in Salmonella enterica serovar Typhimurium.The Salmonella Spi1 virulence regulatory protein HilD directly activates transcription of the flagellar master operon flhDC.The Csr/Rsm system of Yersinia and related pathogens: a post-transcriptional strategy for managing virulence.Mechanisms used by virulent Salmonella to impair dendritic cell function and evade adaptive immunity.An overview of the domestication and impact of the Salmonella mobilome.
P2860
Q21131360-B247ECD2-ACE1-40DB-BF50-D57D562D72B3Q26781384-A5FBAC52-D843-47B0-A60E-CC8CF44C99E0Q26825603-5C50CF72-6889-486C-87D0-EEDC1FEA8E76Q26852008-E3284E79-9E87-45EB-8836-F9E29E4776A8Q27322789-8DDCD051-69EC-4812-9ACB-1014C1499B58Q27334132-62C3496F-E22F-4DAC-9438-0A0B54BD50F0Q27663668-F83EA951-B6C7-479D-B5B2-397BD9121A66Q30275249-D4445CDF-EB1F-4F44-8956-FD288FE33668Q31144688-7FAD95DD-FE25-4F86-87F9-CFE880BB383EQ33397858-EC826608-50F3-44B6-B556-1F93BB6B7B61Q33584937-65DFED8C-3BFC-4AC6-844D-F80E5EE98843Q33585337-F8C693A6-A1D1-459B-9845-A474C126D29EQ33675951-DF799D7C-70D0-45FA-911E-6DEB85E33F4DQ34045086-FD2E27C5-8E33-485B-977D-B0BA2D196A4AQ34129364-5678C5FC-48C5-49AC-A1AD-45E9EB97C649Q34146148-85B4D957-A25B-48FA-AE6F-23FAC5F2F90DQ34192742-989C9E60-35CB-4CE1-85B6-A7D8AD0D4C80Q34201510-7BDF8FBD-3EA8-48E3-BD9A-21C91F13CDC8Q34211607-1B08DCB6-12E7-4BB8-A507-039A72CE4D2FQ34230076-0C40B857-4C38-488B-923F-A8C1A6A5414EQ34336985-A3D96FAA-F55C-4BAC-A4EA-61CD43163492Q34698809-D2A8DDC7-F0C9-4D3A-94BF-CEBDB03ED66EQ35049823-10A7ADA9-ACCA-4121-BAF1-773BAD0C393AQ35130307-7C37687F-4890-4E8E-82C1-5D6C172719C8Q35146711-51DBA770-94EB-4991-86ED-A4CC1AC7F206Q35693689-2566DDAF-936F-4EDE-8B3B-877BF5D2D2E6Q35839664-D281AF79-C02C-47AD-90C8-12F9CDFAB242Q35919194-49D5B93C-32F0-4708-B300-CB1520804C93Q35921961-C50D76C6-B8E6-4F88-9BF9-ADE68E44B0F8Q36114219-74ABF10F-14EF-490F-B1B6-A3C36BD4C49FQ36277143-BF64627C-7962-422F-ADB6-B3350FDAB18AQ36396912-79A9BBBC-6CEB-4033-8AC2-9A526424FC04Q37236308-844B8CF6-A2F3-4581-A970-FAADF2DE5B4BQ37299789-62473718-E259-46D5-B8F0-D76C94611843Q37545958-28F301A8-ECC7-430D-99DE-970686715DB2Q37643366-21CC2CF1-09A5-4FF6-B02A-C24EB4BCA7B2Q37713164-E5043EFC-78E7-4CE9-AF40-2B478E9ED177Q37984185-7CB00455-0CF7-469C-BAD1-55DB0178ACF8Q38019049-4AE0677F-7C32-4695-8F4F-76B68495701CQ38077273-B97A171D-9DC2-4EB8-8E6B-6F539119143D
P2860
HilD-mediated transcriptional cross-talk between SPI-1 and SPI-2.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
HilD-mediated transcriptional cross-talk between SPI-1 and SPI-2.
@en
type
label
HilD-mediated transcriptional cross-talk between SPI-1 and SPI-2.
@en
prefLabel
HilD-mediated transcriptional cross-talk between SPI-1 and SPI-2.
@en
P2093
P2860
P356
P1476
HilD-mediated transcriptional cross-talk between SPI-1 and SPI-2
@en
P2093
Francisco J Santana
José L Puente
Luary C Martínez
Olivia Steele-Mortimer
Víctor H Bustamante
P2860
P304
14591-14596
P356
10.1073/PNAS.0801205105
P407
P577
2008-09-17T00:00:00Z