Septic impairment of capillary blood flow requires nicotinamide adenine dinucleotide phosphate oxidase but not nitric oxide synthase and is rapidly reversed by ascorbate through an endothelial nitric oxide synthase-dependent mechanism
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Pathophysiology of microcirculatory dysfunction and the pathogenesis of septic shockEvaluation of vitamin C for adjuvant sepsis therapyIntravenous ascorbic acid as an adjuvant to interleukin-2 immunotherapyAscorbate protects endothelial barrier function during septic insult: Role of protein phosphatase type 2A.The role of renal hypoperfusion in development of renal microcirculatory dysfunction in endotoxemic rats.Impaired microvascular perfusion in sepsis requires activated coagulation and P-selectin-mediated platelet adhesion in capillariesIntravenous ascorbic acid to prevent and treat cancer-associated sepsis?The effect of vitamin C on plasma volume in the early stage of sepsis in the rat.Inhibition of MicroRNA 195 Prevents Apoptosis and Multiple-Organ Injury in Mouse Models of Sepsis.Pathophysiology, management and treatment of smoke inhalation injury.Mechanism of action of vitamin C in sepsis: ascorbate modulates redox signaling in endothelium.The response of the host microcirculation to bacterial sepsis: does the pathogen matter?Role of connexins in microvascular dysfunction during inflammation.Critical role for oxidative stress, platelets, and coagulation in capillary blood flow impairment in sepsis.Microcirculatory alterations: potential mechanisms and implications for therapy.Vitamin C revisitedNovel relationships between oxidative stress and angiogenesis-related factors in sepsis: New biomarkers and therapies.Microcirculatory dysfunction in sepsis: pathophysiology, clinical monitoring, and potential therapies.Ascorbate stimulates endothelial nitric oxide synthase enzyme activity by rapid modulation of its phosphorylation status.Mechanisms of attenuation of abdominal sepsis induced acute lung injury by ascorbic acid.Vitamin C and Microvascular Dysfunction in Systemic Inflammation.Voluntary running exercise protects against sepsis-induced early inflammatory and pro-coagulant responses in aged mice.The Effect of Sepsis on the Erythrocyte.NADPH oxidase-dependent reactive oxygen species mediate amplified TLR4 signaling and sepsis-induced mortality in Nrf2-deficient mice.Ascorbate reduces mouse platelet aggregation and surface P-selectin expression in an ex vivo model of sepsis.L-NIL prevents renal microvascular hypoxia and increase of renal oxygen consumption after ischemia-reperfusion in rats.Seven unconfirmed ideas to improve future ICU practice.C for colored urine: acute hemolysis induced by high-dose ascorbic acid.Vitamin C supplementation in the critically ill: A systematic review and meta-analysis
P2860
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P2860
Septic impairment of capillary blood flow requires nicotinamide adenine dinucleotide phosphate oxidase but not nitric oxide synthase and is rapidly reversed by ascorbate through an endothelial nitric oxide synthase-dependent mechanism
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Septic impairment of capillary ...... e synthase-dependent mechanism
@en
type
label
Septic impairment of capillary ...... e synthase-dependent mechanism
@en
prefLabel
Septic impairment of capillary ...... e synthase-dependent mechanism
@en
P2093
P2860
P1476
Septic impairment of capillary ...... e synthase-dependent mechanism
@en
P2093
John X Wilson
Karel Tyml
P2860
P304
P356
10.1097/CCM.0B013E31818024F6
P407
P577
2008-08-01T00:00:00Z