Hyperactive ERK and persistent mTOR signaling characterize vemurafenib resistance in papillary thyroid cancer cells.
about
Genome-wide functional analysis on the molecular mechanism of specifically biosynthesized fluorescence Eu complex.Vemurafenib-resistance via de novo RBM genes mutations and chromosome 5 aberrations is overcome by combined therapy with palbociclib in thyroid carcinoma with BRAFV600E.Gene master regulators of papillary and anaplastic thyroid cancers.ERK inhibition represses gefitinib resistance in non-small cell lung cancer cells.
P2860
Hyperactive ERK and persistent mTOR signaling characterize vemurafenib resistance in papillary thyroid cancer cells.
description
2015 nî lūn-bûn
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2015年の論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年论文
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2015年论文
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2015年论文
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name
Hyperactive ERK and persistent ...... apillary thyroid cancer cells.
@en
type
label
Hyperactive ERK and persistent ...... apillary thyroid cancer cells.
@en
prefLabel
Hyperactive ERK and persistent ...... apillary thyroid cancer cells.
@en
P2093
P2860
P356
P1433
P1476
Hyperactive ERK and persistent ...... apillary thyroid cancer cells.
@en
P2093
Augustine L Moscatello
Elyse K Hanly
Jan Geliebter
Neha Y Tuli
Raj K Tiwari
Robert B Bednarczyk
Robert Suriano
P2860
P304
P356
10.18632/ONCOTARGET.6779
P407
P577
2015-12-28T00:00:00Z