miR-150 promotes renal fibrosis in lupus nephritis by downregulating SOCS1 - Test2 - elhamkhani - TriplyDB

miR-150 promotes renal fibrosis in lupus nephritis by downregulating SOCS1

miR-150 promotes renal fibrosis in lupus nephritis by downregulating SOCS1

http://www.wikidata.org/entity/Q36977367

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miRNAs in the Pathogenesis of Systemic Lupus Erythematosus miR-150 influences B-cell receptor signaling in chronic lymphocytic leukemia by regulating expression of GAB1 and FOXP1 Anti-Double-Stranded DNA IgG Participates in Renal Fibrosis through Suppressing the Suppressor of Cytokine Signaling 1 Signals.Recent advances in the understanding of renal inflammation and fibrosis in lupus nephritis.Up-regulation of Serum MiR-130b-3p Level is Associated with Renal Damage in Early Lupus Nephritis.MicroRNA-150 deletion in mice protects kidney from myocardial infarction-induced acute kidney injury.Selection of urinary sediment miRNAs as specific biomarkers of IgA nephropathy.MicroRNA-27a promotes renal tubulointerstitial fibrosis via suppressing PPARγ pathway in diabetic nephropathy.Leukotriene B4 enhances the generation of proinflammatory microRNAs to promote MyD88-dependent macrophage activation SOCS signaling in autoimmune diseases: molecular mechanisms and therapeutic implications.Role of microRNA machinery in kidney fibrosis.microRNAs in glomerular diseases from pathophysiology to potential treatment target.MicroRNA: master controllers of intracellular signaling pathways.Molecular and Immunological Basis of Tubulo-Interstitial Injury in Lupus Nephritis: a Comprehensive Review.MicroRNAs and drug-induced kidney injury.Interaction Between Gastric Carcinoma Cells and Neural Cells Promotes Perineural Invasion by a Pathway Involving VCAM1.MicroRNAs as Master Regulators of Glomerular Function in Health and Disease.MicroRNA-150: A potential regulator in pathogens infection and autoimmune diseases.Translating epigenetics into clinic: focus on lupus.MicroRNA-196a/b Mitigate Renal Fibrosis by Targeting TGF-β Receptor 2 Excessive activation of the TLR9/TGF-β1/PDGF-B pathway in the peripheral blood of patients with systemic lupus erythematosus.Urinary Exosomal MicroRNA Profiling in Incipient Type 2 Diabetic Kidney Disease.Micro-RNA analysis of renal biopsies in human lupus nephritis demonstrates up-regulated miR-422a driving reduction of kallikrein-related peptidase 4.Urinary miR-21 as a potential biomarker of hypertensive kidney injury and fibrosis.Defective Suppressor of Cytokine Signaling 1 Signaling Contributes to the Pathogenesis of Systemic Lupus Erythematosus.Schisandrin B down-regulated lncRNA BCYRN1 expression of airway smooth muscle cells by improving miR-150 expression to inhibit the proliferation and migration of ASMC in asthmatic rats.Silencing of SOCS-1 and SOCS-3 suppresses renal interstitial fibrosis by alleviating renal tubular damage in a rat model of hydronephrosis.circHLA-C Plays an Important Role in Lupus Nephritis by Sponging miR-150.MicroRNAs associated with the development of kidney diseases in humans and animals.MicroRNA-130b Ameliorates Murine Lupus Nephritis Through Targeting the Type I Interferon Pathway on Renal Mesangial Cells.miR-29c in urinary exosomes as predictor of early renal fibrosis in lupus nephritis.Investigation of mechanisms of mesenchymal stem cells for treatment of diabetic nephropathy via construction of a miRNA-TF-mRNA network.Effects of Major Epigenetic Factors on Systemic Lupus Erythematosus

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P2860

miR-150 promotes renal fibrosis in lupus nephritis by downregulating SOCS1

http://www.wikidata.org/entity/Q36977367

description

2013 nî lūn-bûn

@nan

2013年の論文

@ja

2013年論文

@yue

2013年論文

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2013年論文

@zh-hk

2013年論文

@zh-mo

2013年論文

@zh-tw

2013年论文

@wuu

2013年论文

@zh

2013年论文

@zh-cn

name

miR-150 promotes renal fibrosis in lupus nephritis by downregulating SOCS1

@en

type

label

miR-150 promotes renal fibrosis in lupus nephritis by downregulating SOCS1

@en

prefLabel

miR-150 promotes renal fibrosis in lupus nephritis by downregulating SOCS1

@en

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Journal of the American Society of Nephrology

P1476

miR-150 promotes renal fibrosis in lupus nephritis by downregulating SOCS1

@en

P2093

Changyu Zheng

http://www.w3.org/2001/XMLSchema#string

Gabor G Illei

http://www.w3.org/2001/XMLSchema#string

Howard Austin

http://www.w3.org/2001/XMLSchema#string

Hua Zhou

http://www.w3.org/2001/XMLSchema#string

Ilias Alevizos

http://www.w3.org/2001/XMLSchema#string

James E Balow

http://www.w3.org/2001/XMLSchema#string

Mayank Tandon

http://www.w3.org/2001/XMLSchema#string

Paola Perez

http://www.w3.org/2001/XMLSchema#string

Sarfaraz A Hasni

http://www.w3.org/2001/XMLSchema#string

Shyh-Ing Jang

http://www.w3.org/2001/XMLSchema#string

P2860

A mammalian microRNA expression atlas based on small RNA library sequencing

Mechanisms of tissue injury in lupus nephritis

Dysregulation of microRNAs after myocardial infarction reveals a role of miR-29 in cardiac fibrosis

Loss of MicroRNA-192 promotes fibrogenesis in diabetic nephropathy

Modulation of microRNA expression in human T-cell development: targeting of NOTCH3 by miR-150.

Polycystic kidneys and chronic inflammatory lesions are the delayed consequences of loss of the suppressor of cytokine signaling-1 (SOCS-1).

HSP72 attenuates renal tubular cell apoptosis and interstitial fibrosis in obstructive nephropathy.

miR-192 mediates TGF-beta/Smad3-driven renal fibrosis.

Glomerular and tubulointerstitial miR-638, miR-198 and miR-146a expression in lupus nephritis.

TGF-beta1 reduces Wilms' tumor suppressor gene expression in podocytes.

P304

1073-1087

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scholarly article

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10.1681/ASN.2012080849

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P433

7

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P478

24

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Jeffrey B. Kopp

P577

2013-05-30T00:00:00Z

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23723424

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P932

3699828

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