Robust inducible Cre recombinase activity in the human malaria parasite Plasmodium falciparum enables efficient gene deletion within a single asexual erythrocytic growth cycle
about
Paths to a malaria vaccine illuminated by parasite genomicsConditional expression of apical membrane antigen 1 in Plasmodium falciparum shows it is required for erythrocyte invasion by merozoitesProcessing of Plasmodium falciparum Merozoite Surface Protein MSP1 Activates a Spectrin-Binding Function Enabling Parasite Egress from RBCsUnique apicomplexan IMC sub-compartment proteins are early markers for apical polarity in the malaria parasiteType II fatty acid biosynthesis is essential for Plasmodium falciparum sporozoite development in the midgut of Anopheles mosquitoesNovel thioredoxin-like proteins are components of a protein complex coating the cortical microtubules of Toxoplasma gondiiEvidence that the Malaria Parasite Plasmodium falciparum Putative Rhoptry Protein 2 Localizes to the Golgi Apparatus throughout the Erythrocytic CycleHost cell remodeling by pathogens: the exomembrane system in Plasmodium-infected erythrocytesInducible knockdown of Plasmodium gene expression using the glmS ribozymeThe malarial serine protease SUB1 plays an essential role in parasite liver stage developmentRegulation and Essentiality of the StAR-related Lipid Transfer (START) Domain-containing Phospholipid Transfer Protein PFA0210c in Malaria ParasitesA genetic system to study Plasmodium falciparum protein functionThe Plasmodium falciparum rhoptry protein RhopH3 plays essential roles in host cell invasion and nutrient uptakePlasmodium falciparum SERA5 plays a non-enzymatic role in the malarial asexual blood-stage lifecycleParasitophorous vacuole poration precedes its rupture and rapid host erythrocyte cytoskeleton collapse in Plasmodium falciparum egress.The Plasmodium falciparum pseudoprotease SERA5 regulates the kinetics and efficiency of malaria parasite egress from host erythrocytes.Generation of rodent malaria parasites with a high mutation rate by destructing proofreading activity of DNA polymerase δ.Versatile control of Plasmodium falciparum gene expression with an inducible protein-RNA interactionA genome-scale vector resource enables high-throughput reverse genetic screening in a malaria parasiteAntiparasitic chemotherapy: from genomes to mechanisms.Conditional U1 Gene Silencing in Toxoplasma gondii.Conditional Degradation of Plasmodium Calcineurin Reveals Functions in Parasite Colonization of both Host and Vector.Development and Application of a Simple Plaque Assay for the Human Malaria Parasite Plasmodium falciparum.Localisation-based imaging of malarial antigens during erythrocyte entry reaffirms a role for AMA1 but not MTRAP in invasionConditional gene deletion with DiCre demonstrates an essential role for CRK3 in Leishmania mexicana cell cycle regulation.Human red blood cell-adapted Plasmodium knowlesi parasites: a new model system for malaria research.From the genome to the phenome: tools to understand the basic biology of Plasmodium falciparum.Role and Regulation of Glutathione Metabolism in Plasmodium falciparum.A versatile strategy for rapid conditional genome engineering using loxP sites in a small synthetic intron in Plasmodium falciparum.Everybody needs sphingolipids, right! Mining for new drug targets in protozoan sphingolipid biosynthesis.Calcium signalling in malaria parasites.Proteases as antimalarial targets: strategies for genetic, chemical, and therapeutic validation.A Telomeric Cluster of Antimony Resistance Genes on Chromosome 34 of Leishmania infantum.Multiple essential functions of Plasmodium falciparum actin-1 during malaria blood-stage development.Generating conditional gene knockouts in Plasmodium - a toolkit to produce stable DiCre recombinase-expressing parasite lines using CRISPR/Cas9.The HIV-1 transmission bottleneck.Characterization of a putative Plasmodium falciparum SAC1 phosphoinositide-phosphatase homologue potentially required for survival during the asexual erythrocytic stages.CRISPR-Cas9-mediated functional dissection of 3'-UTRs.From technology to biology: a malaria genetic toolbox for the functional dissection of essential genes.Compositional and expression analyses of the glideosome during the Plasmodium life cycle reveal an additional myosin light chain required for maximum motility.
P2860
Q26853267-D3A3930B-7D18-4A5C-B102-42589DB8D2C3Q27230092-273B3807-D1B9-4DA6-81E8-60F13625A483Q27972787-7917C293-25B5-4202-BE38-F1897C648E0FQ27973410-193291CC-0924-498D-B15F-65CA4165B582Q27973456-89988D9B-433F-40AA-906B-D4235DDBE457Q27973671-AEA8FF58-0CAE-4CA7-A191-9A0A4FD201F4Q27974462-649D00BF-5A12-452C-98CA-BFAE8B4945EBQ28072465-57C0B840-7688-4656-85DB-A184FF67E8C4Q28536278-02CC8141-2BE3-4757-87F2-A52E675E596FQ28537649-9C960B4B-708E-469E-9BF6-F5CE0C03EB16Q30039045-E24BCF63-ADC4-4AF5-A8F8-239160A2B986Q30039542-C5702737-4C84-40DC-85BF-77D2E9C3E419Q30039603-80B93AB5-981C-49DC-BC8A-4A8939AF079EQ30046956-12F5FC92-9463-4913-9642-50064E1AD0D3Q30844001-CB16048F-06D2-4BE3-B6A7-BA901423A1FCQ33880801-84DA6482-FF78-4ABE-A7FD-56006FFA082CQ34040908-E5E367F9-C52A-4958-A2C4-0CD8C895FF64Q34470286-2BDC14F6-641E-442E-9DBE-97064E400AC6Q35185939-B7EF6250-414B-4AA6-9556-D368BC27355FQ35238319-3E16CBF7-7CBA-4893-A67A-F80F0EA8E0B0Q35668397-0569AC4C-E62E-4881-8315-F6BFC19BB678Q35871673-25AB6CC0-6AD1-46B3-A154-30E5C0E46122Q36058850-18D3DF4D-FB5A-428A-92F2-41820F18EF3FQ36518609-E002A139-BC4A-49B3-8C3E-75D4FA946A83Q37020701-DD17A74F-544B-4627-82E4-69C80B27EEA3Q37731191-8C91DB81-0A89-4C0C-9810-9C36A51E7159Q38250222-05B88926-00F3-49E4-98B2-661D6935F987Q38523502-7A0E31E6-3D93-41A4-8B4B-C659C71DEBF4Q38556666-EE4B5DDF-39A6-42A9-AE7A-4BC5517CB77EQ38668340-6E9240B9-45B2-4E62-84F0-85773E492830Q38691315-8CDF2D6A-CD74-45BD-A40F-2AD6B732A427Q39362514-2B504B1D-35E5-428A-AB11-881DD6D70FC1Q39515983-A7FA6676-2D54-4B86-BD3C-3B01101B591DQ40092367-BD077D70-817F-4050-8B6B-F7FA913F8903Q41081800-EA3BD21E-8E68-4EE1-AD44-7FA122B6908AQ42314454-0A22B6B1-9F0B-4FE8-8AB1-119CCC93CC67Q42371004-7FA1C059-9647-4F34-A89B-78A24A8B450BQ46067907-CE1C5A5E-6023-4720-8DD6-029E682D1312Q46462397-287FA29F-18A6-4C69-80B9-6EBEBE6032ACQ47119134-26434D77-E2F1-4A1A-A449-0FD88D3A2581
P2860
Robust inducible Cre recombinase activity in the human malaria parasite Plasmodium falciparum enables efficient gene deletion within a single asexual erythrocytic growth cycle
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 26 March 2013
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Robust inducible Cre recombina ...... xual erythrocytic growth cycle
@en
Robust inducible Cre recombina ...... ual erythrocytic growth cycle.
@nl
type
label
Robust inducible Cre recombina ...... xual erythrocytic growth cycle
@en
Robust inducible Cre recombina ...... ual erythrocytic growth cycle.
@nl
prefLabel
Robust inducible Cre recombina ...... xual erythrocytic growth cycle
@en
Robust inducible Cre recombina ...... ual erythrocytic growth cycle.
@nl
P2093
P2860
P356
P1476
Robust inducible Cre recombina ...... xual erythrocytic growth cycle
@en
P2093
Christine R Collins
Eleanor H Wong
Fiona Hackett
Jean-Paul Herman
Markus Meissner
Michael J Blackman
Nicole Andenmatten
Robert Stallmach
Sujaan Das
Sylke Müller
P2860
P304
P356
10.1111/MMI.12206
P407
P577
2013-03-26T00:00:00Z