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FoxO Transcription Factors and Regenerative Pathways in Diabetes MellitusMesangial cell-specific antibodies are central to the pathogenesis of lupus nephritisMicroRNAs as potential therapeutic targets in kidney diseaseNew Insights for Oxidative Stress and Diabetes MellitusProteasome Activators, PA28α and PA28β, Govern Development of Microvascular Injury in Diabetic Nephropathy and RetinopathySodium potassium adenosine triphosphatase (Na/K-ATPase) as a therapeutic target for uremic cardiomyopathy.Anti-microRNA-21 oligonucleotides prevent Alport nephropathy progression by stimulating metabolic pathwaysRole of Nuclear Factor Erythroid 2-Related Factor 2 in Diabetic Nephropathy.Olmesartan medoxomil reverses glomerulosclerosis in renal tissue induced by myocardial infarction without changes in renal function.Therapeutic Applications of Rose Hips from Different Rosa Species.MicroRNAs are potential therapeutic targets in fibrosing kidney disease: lessons from animal models.Fenugreek Prevents the Development of STZ-Induced Diabetic Nephropathy in a Rat Model of Diabetes.Sequence-based polymorphisms in the mitochondrial D-loop and potential SNP predictors for chronic dialysisPRAS40 is an integral regulatory component of erythropoietin mTOR signaling and cytoprotectionSuppression of XBP1S mediates high glucose-induced oxidative stress and extracellular matrix synthesis in renal mesangial cell and kidney of diabetic rats.Genetic deficiency of aldose reductase counteracts the development of diabetic nephropathy in C57BL/6 mice.Hyperglycemia, p53, and mitochondrial pathway of apoptosis are involved in the susceptibility of diabetic models to ischemic acute kidney injury.EPO relies upon novel signaling of Wnt1 that requires Akt1, FoxO3a, GSK-3β, and β-catenin to foster vascular integrity during experimental diabetes.Liraglutide inhibits autophagy and apoptosis induced by high glucose through GLP-1R in renal tubular epithelial cellsInhibitory role of the KEAP1-NRF2 pathway in TGFβ1-stimulated renal epithelial transition to fibroblastic cells: a modulatory effect on SMAD signaling.Mammalian target of rapamycin: hitting the bull's-eye for neurological disorders.Epac1-mediated, high glucose-induced renal proximal tubular cells hypertrophy via the Akt/p21 pathwayTherapeutic potential of Nrf2 activators in streptozotocin-induced diabetic nephropathy.Upstream regulators and downstream effectors of NADPH oxidases as novel therapeutic targets for diabetic kidney disease.EGF receptor deletion in podocytes attenuates diabetic nephropathy.Induction of hemeoxygenase-1 reduces renal oxidative stress and inflammation in diabetic spontaneously hypertensive rats.An Atherogenic Paigen-Diet Aggravates Nephropathy in Type 2 Diabetic OLETF RatsTherapeutic potential of digitoflavone on diabetic nephropathy: nuclear factor erythroid 2-related factor 2-dependent anti-oxidant and anti-inflammatory effect.The endocannabinoid system and plant-derived cannabinoids in diabetes and diabetic complicationsAngiotensin-(1-7) administration benefits cardiac, renal and progenitor cell function in db/db mice.Ebselen and congeners inhibit NADPH oxidase 2-dependent superoxide generation by interrupting the binding of regulatory subunits.Chronic Running Exercise Alleviates Early Progression of Nephropathy with Upregulation of Nitric Oxide Synthases and Suppression of Glycation in Zucker Diabetic Rats.Circulating TGF-β1, glycation, and oxidation in children with diabetes mellitus type 1.Thioredoxin-Interacting Protein Deficiency Protects against Diabetic Nephropathy.Naringin Alleviates Diabetic Kidney Disease through Inhibiting Oxidative Stress and Inflammatory ReactionRenal protection of in vivo administration of tempol in streptozotocin-induced diabetic rats.Synergistic antioxidant activity of resveratrol with genistein in high-glucose treated Madin-Darby canine kidney epithelial cells.myo-Inositol Oxygenase Overexpression Accentuates Generation of Reactive Oxygen Species and Exacerbates Cellular Injury following High Glucose Ambience: A NEW MECHANISM RELEVANT TO THE PATHOGENESIS OF DIABETIC NEPHROPATHYKCa3.1 mediates dysfunction of tubular autophagy in diabetic kidneys via PI3k/Akt/mTOR signaling pathwaysHigh glucose induces autophagy in podocytes.
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P2860
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on January 2010
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Oxidative stress in diabetic nephropathy.
@en
Oxidative stress in diabetic nephropathy.
@nl
type
label
Oxidative stress in diabetic nephropathy.
@en
Oxidative stress in diabetic nephropathy.
@nl
prefLabel
Oxidative stress in diabetic nephropathy.
@en
Oxidative stress in diabetic nephropathy.
@nl
P2093
P2860
P1476
Oxidative stress in diabetic nephropathy.
@en
P2093
N Kashihara
V K Kondeti
Y S Kanwar
P2860
P304
P356
10.2174/092986710793348581
P577
2010-01-01T00:00:00Z