Toll-like receptors in defense and damage of the central nervous system. - Test2 - elhamkhani - TriplyDB

Toll-like receptors in defense and damage of the central nervous system.

Toll-like receptors in defense and damage of the central nervous system.

http://www.wikidata.org/entity/Q37015536

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Stress-induced neuroinflammatory priming: A liability factor in the etiology of psychiatric disorders Role of the innate immune system in the pathogenesis of multiple sclerosis.Herpes simplex virus induces neural oxidative damage via microglial cell Toll-like receptor-2.Microglial phenotype and adaptation.Progesterone administration modulates cortical TLR4/NF-κB signaling pathway after subarachnoid hemorrhage in male rats Toll-like receptor signaling in neural plasticity and disease.Activation of BV2 microglia by lipopolysaccharide triggers an inflammatory reaction in PC12 cell apoptosis through a toll-like receptor 4-dependent pathway Interferon regulatory factor-7 modulates experimental autoimmune encephalomyelitis in mice.Toll-like receptors in chronic pain.Acetate supplementation modulates brain histone acetylation and decreases interleukin-1β expression in a rat model of neuroinflammation.Guilty molecules, guilty minds? The conflicting roles of the innate immune response to traumatic brain injury Cannabinoid modulation of neuroinflammatory disorders.Adjunctive daptomycin attenuates brain damage and hearing loss more efficiently than rifampin in infant rat pneumococcal meningitis.GM-CSF increases LPS-induced production of proinflammatory mediators via upregulation of TLR4 and CD14 in murine microglia.Toll-like receptor 4 signaling in intracerebral hemorrhage-induced inflammation and injury.Effect of chronic delivery of the Toll-like receptor 4 antagonist (+)-naltrexone on incubation of heroin craving.Microglia during development and aging.Activation of toll-like receptor-3 induces interferon-lambda expression in human neuronal cells.Toll-like receptors in neurodegeneration.Glia in pathological pain: a role for fractalkine The contributing role of CD14 in toll-like receptor 4 dependent neuropathic pain Iron is essential for oligodendrocyte genesis following intraspinal macrophage activation.Efficient subtractive cloning of genes activated by lipopolysaccharide and interferon γ in primary-cultured cortical cells of newborn mice HMGB1 Activates Proinflammatory Signaling via TLR5 Leading to Allodynia.Defense against HSV-1 in a murine model is mediated by iNOS and orchestrated by the activation of TLR2 and TLR9 in trigeminal ganglia.Aging and amyloid β oligomers enhance TLR4 expression, LPS-induced Ca2+ responses, and neuron cell death in cultured rat hippocampal neurons.Recognition of herpes simplex viruses: toll-like receptors and beyond Inflammation in Alzheimer's disease: relevance to pathogenesis and therapy Microglia: key innate immune cells of the brain.Stress- and glucocorticoid-induced priming of neuroinflammatory responses: potential mechanisms of stress-induced vulnerability to drugs of abuse.Inflammation in Alzheimer disease-a brief review of the basic science and clinical literature.Stress-induced glucocorticoids as a neuroendocrine alarm signal of danger.Neuroimmune and neuroendocrine abnormalities in depression: two sides of the same coin.Mesenchymal Stem Cell-Derived Microvesicles Modulate Lipopolysaccharides-Induced Inflammatory Responses to Microglia Cells.Microglia Function in the Normal Brain.Danger Signals and Inflammasomes: Stress-Evoked Sterile Inflammation in Mood Disorders.The role of microglia in multiple sclerosis.Vitamin d deficiency reduces the immune response, phagocytosis rate, and intracellular killing rate of microglial cells Platelet Activating Factor (PAF) Receptor Deletion or Antagonism Attenuates Severe HSV-1 Meningoencephalitis.Toll-like receptor prestimulation increases phagocytosis of Escherichia coli DH5alpha and Escherichia coli K1 strains by murine microglial cells.

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Toll-like receptors in defense and damage of the central nervous system.

http://www.wikidata.org/entity/Q37015536

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 03 April 2007

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Toll-like receptors in defense and damage of the central nervous system.

@en

Toll-like receptors in defense and damage of the central nervous system.

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type

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Toll-like receptors in defense and damage of the central nervous system.

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Toll-like receptors in defense and damage of the central nervous system.

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Toll-like receptors in defense and damage of the central nervous system.

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Toll-like receptors in defense and damage of the central nervous system.

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S11481-007-9071-5

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Journal of Neuroimmune Pharmacology

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Toll-like receptors in defense and damage of the central nervous system.

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P2093

James R Lokensgard

http://www.w3.org/2001/XMLSchema#string

Phillip K Peterson

http://www.w3.org/2001/XMLSchema#string

Rajagopal N Aravalli

http://www.w3.org/2001/XMLSchema#string

P2860

Role of MD-2 in TLR2- and TLR4-mediated recognition of Gram-negative and Gram-positive bacteria and activation of chemokine genes

Microglia initiate central nervous system innate and adaptive immune responses through multiple TLRs

MD-2 enables Toll-like receptor 2 (TLR2)-mediated responses to lipopolysaccharide and enhances TLR2-mediated responses to Gram-positive and Gram-negative bacteria and their cell wall components

Cutting edge: heat shock protein 60 is a putative endogenous ligand of the toll-like receptor-4 complex

Human TLR10 is a functional receptor, expressed by B cells and plasmacytoid dendritic cells, which activates gene transcription through MyD88

Hyaluronan fragments act as an endogenous danger signal by engaging TLR2.

Inflammatory mechanisms after ischemia and stroke.

Mechanisms of bacterial lipopolysaccharide-induced endothelial apoptosis.

Central role for MyD88 in the responses of microglia to pathogen-associated molecular patterns

Chlamydial heat shock protein 60 activates macrophages and endothelial cells through Toll-like receptor 4 and MD2 in a MyD88-dependent pathway.

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s11481-007-9071-5

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297-312

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scholarly article

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10.1007/S11481-007-9071-5

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4

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2

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2007-04-03T00:00:00Z

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18040848

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Toll-like receptor