Virulence of Staphylococcus aureus in a mouse mastitis model: studies of alpha hemolysin, coagulase, and protein A as possible virulence determinants with protoplast fusion and gene cloning.
about
A Staphylococcus aureus small RNA is required for bacterial virulence and regulates the expression of an immune-evasion moleculeVirulence of protein A-deficient and alpha-toxin-deficient mutants of Staphylococcus aureus isolated by allele replacementStaphylococcus aureus α-toxin: nearly a century of intrigueStaphylococcus aureus secretes coagulase and von Willebrand factor binding protein to modify the coagulation cascade and establish host infectionsStachylysin may be a cause of hemorrhaging in humans exposed to Stachybotrys chartarumInitial characterization of the hemolysin stachylysin from Stachybotrys chartarum.Inactivation of staphylococcal virulence factors using a light-activated antimicrobial agentSurface proteins of gram-positive bacteria and mechanisms of their targeting to the cell wall envelope.EsxA and EsxB are secreted by an ESAT-6-like system that is required for the pathogenesis of Staphylococcus aureus infections.Identification of single nucleotide polymorphisms associated with hyperproduction of alpha-toxin in Staphylococcus aureus.Virulence strategies of the dominant USA300 lineage of community-associated methicillin-resistant Staphylococcus aureus (CA-MRSA)The multifunctional Staphylococcus aureus autolysin aaa mediates adherence to immobilized fibrinogen and fibronectin.The psmα locus regulates production of Staphylococcus aureus alpha-toxin during infectionMolecular basis of virulence in Staphylococcus aureus mastitis.Expression of the biofilm-associated protein interferes with host protein receptors of Staphylococcus aureus and alters the infective process.Low levels of β-lactam antibiotics induce extracellular DNA release and biofilm formation in Staphylococcus aureusAbility of Staphylococcus aureus coagulase genotypes to resist neutrophil bactericidal activity and phagocytosis.Host defenses against Staphylococcus aureus infection require recognition of bacterial lipoproteins.Nucleotide sequence of the plasminogen activator gene of Yersinia pestis: relationship to ompT of Escherichia coli and gene E of Salmonella typhimurium.Roles of alpha-toxin and beta-toxin in virulence of Staphylococcus aureus for the mouse mammary gland.Mouse models of mastitis - how physiological are they?The use of BMP-2 coupled - Nanosilver-PLGA composite grafts to induce bone repair in grossly infected segmental defectsSpecific roles of alpha-toxin and beta-toxin during Staphylococcus aureus corneal infection.Corneal virulence of Staphylococcus aureus: roles of alpha-toxin and protein A in pathogenesisStaphylococcal alpha toxin promotes blood coagulation via attack on human platelets.Bovine and rabbit models for the study of a Staphylococcus aureus avirulent mutant strain, RC122Recurrent infections and immune evasion strategies of Staphylococcus aureus.Exploring Staphylococcus aureus pathways to disease for vaccine development.Role of pore-forming toxins in neonatal sepsisStaphylococcus aureus sortase mutants defective in the display of surface proteins and in the pathogenesis of animal infectionsReaction of staphylococcal alpha-toxin with peptide-induced antibodies.Staphylococcus aureus exopolysaccharide in vivo demonstrated by immunomagnetic separation and electron microscopy.The Staphylococcus aureus rsbW (orf159) gene encodes an anti-sigma factor of SigB.Role of coagulase in a murine model of hematogenous pulmonary infection induced by intravenous injection of Staphylococcus aureus enmeshed in agar beads.Virulence factors among gram-positive bacteria in experimental endocarditis.Murine model of cutaneous infection with gram-positive cocci.In vivo activation of neutrophil function in hamsters by recombinant human granulocyte colony-stimulating factor.Quantitation of monomeric and oligomeric forms of membrane-bound staphylococcal alpha-toxin by enzyme-linked immunosorbent assay with a neutralizing monoclonal antibody.Characterization of hemolysin in extracellular products of Pseudomonas cepacia.Dabigatran inhibits Staphylococcus aureus coagulase activity.
P2860
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P2860
Virulence of Staphylococcus aureus in a mouse mastitis model: studies of alpha hemolysin, coagulase, and protein A as possible virulence determinants with protoplast fusion and gene cloning.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on September 1985
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Virulence of Staphylococcus au ...... plast fusion and gene cloning.
@en
Virulence of Staphylococcus au ...... plast fusion and gene cloning.
@nl
type
label
Virulence of Staphylococcus au ...... plast fusion and gene cloning.
@en
Virulence of Staphylococcus au ...... plast fusion and gene cloning.
@nl
prefLabel
Virulence of Staphylococcus au ...... plast fusion and gene cloning.
@en
Virulence of Staphylococcus au ...... plast fusion and gene cloning.
@nl
P2093
P2860
P1476
Virulence of Staphylococcus au ...... plast fusion and gene cloning.
@en
P2093
Haraldsson I
Lindberg M
Wadström T
P2860
P304
P407
P577
1985-09-01T00:00:00Z