Cannabinoids inhibit HIV-1 Gp120-mediated insults in brain microvascular endothelial cells. - Test2 - elhamkhani - TriplyDB

Cannabinoids inhibit HIV-1 Gp120-mediated insults in brain microvascular endothelial cells.

Cannabinoids inhibit HIV-1 Gp120-mediated insults in brain microvascular endothelial cells.

http://www.wikidata.org/entity/Q37074777

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Targeting brain microvascular endothelial cells: a therapeutic approach to neuroprotection against stroke Blood-brain barrier and retroviral infections Neurogenic inflammation after traumatic brain injury and its potentiation of classical inflammation Excess cerebral TNF causing glutamate excitotoxicity rationalizes treatment of neurodegenerative diseases and neurogenic pain by anti-TNF agents The endocannabinoid/endovanilloid N-arachidonoyl dopamine (NADA) and synthetic cannabinoid WIN55,212-2 abate the inflammatory activation of human endothelial cells Interleukin-25 expressed by brain capillary endothelial cells maintains blood-brain barrier function in a protein kinase Cepsilon-dependent manner.Cannabinoid inhibition of macrophage migration to the trans-activating (Tat) protein of HIV-1 is linked to the CB(2) cannabinoid receptor.CB2 receptor activation inhibits melanoma cell transmigration through the blood-brain barrier.Inhibition of telomerase activity alters tight junction protein expression and induces transendothelial migration of HIV-1-infected cells.HIV-1 gp120 and drugs of abuse: interactions in the central nervous system Cannabinoids and Viral Infections.HIV-1 infection and alcohol abuse: neurocognitive impairment, mechanisms of neurodegeneration and therapeutic interventions.Anandamide inhibits Theiler's virus induced VCAM-1 in brain endothelial cells and reduces leukocyte transmigration in a model of blood brain barrier by activation of CB(1) receptors Cannabinoid neuroimmune modulation of SIV disease.Activation of cannabinoid receptor 2 attenuates leukocyte-endothelial cell interactions and blood-brain barrier dysfunction under inflammatory conditions Impaired neurogenesis by HIV-1-Gp120 is rescued by genetic deletion of fatty acid amide hydrolase enzyme.Inflammation in acute CNS injury: a focus on the role of substance P CD44-mediated monocyte transmigration across Cryptococcus neoformans-infected brain microvascular endothelial cells is enhanced by HIV-1 gp41-I90 ectodomain Attenuation of HIV-1 replication in macrophages by cannabinoid receptor 2 agonists.Endocannabinoids in cerebrovascular regulation.Blocking neurogenic inflammation for the treatment of acute disorders of the central nervous system.X chromosomal variation is associated with slow progression to AIDS in HIV-1-infected women Molecular mechanisms of neuroinvasion by monocytes-macrophages in HIV-1 infection.Immunomodulatory properties of kappa opioids and synthetic cannabinoids in HIV-1 neuropathogenesis.Analysis of absorption enhancers in epithelial cell models.Cannabinoid receptor 2: potential role in immunomodulation and neuroinflammation.Functions of the CB1 and CB 2 receptors in neuroprotection at the level of the blood-brain barrier.Effects of cannabinoids and their receptors on viral infections.Oxidative Stress Induced by Cigarette Smoke Extracts in Human Brain Cells (T98G) and Human Brain Microvascular Endothelial Cells (HBMEC) in Mono- and Co-Culture.Neuropeptide substance P and the immune response.Δ9-Tetrahydrocannabinol Suppresses Secretion of IFNα by Plasmacytoid Dendritic Cells From Healthy and HIV-Infected Individuals.The effect of tobacco smoke exposure on the generation of reactive oxygen species and cellular membrane damage using co-culture model of blood brain barrier with astrocytes.Curcumin Increases HSP70 Expression in Primary Rat Cortical Neuronal Apoptosis Induced by gp120 V3 Loop Peptide.The Role of Neurogenic Inflammation in Blood-Brain Barrier Disruption and Development of Cerebral Oedema Following Acute Central Nervous System (CNS) Injury.Cannabinoids mediate opposing effects on inflammation-induced intestinal permeability.Cannabinoid receptor-2 and HIV-associated neurocognitive disorders.Walker 256 tumour cells increase substance P immunoreactivity locally and modify the properties of the blood-brain barrier during extravasation and brain invasion.Maintaining unperturbed cerebral blood flow is key in the study of brain metastasis and its interactions with stress and inflammatory responses.Relationship between G proteins coupled receptors and tight junctions.Angiopoietin-2 mediates blood-brain barrier impairment and colonization of triple-negative breast cancer cells in brain.

P2860

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P2860

Cannabinoids inhibit HIV-1 Gp120-mediated insults in brain microvascular endothelial cells.

http://www.wikidata.org/entity/Q37074777

description

2008 nî lūn-bûn

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2008年の論文

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年學術文章

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2008年學術文章

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2008年學術文章

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name

Cannabinoids inhibit HIV-1 Gp1 ...... crovascular endothelial cells.

@en

Cannabinoids inhibit HIV-1 Gp1 ...... crovascular endothelial cells.

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type

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Cannabinoids inhibit HIV-1 Gp1 ...... crovascular endothelial cells.

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Cannabinoids inhibit HIV-1 Gp1 ...... crovascular endothelial cells.

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Cannabinoids inhibit HIV-1 Gp1 ...... crovascular endothelial cells.

@en

Cannabinoids inhibit HIV-1 Gp1 ...... crovascular endothelial cells.

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JIMMUNOL.181.9.6406

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Journal of Immunology

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Cannabinoids inhibit HIV-1 Gp1 ...... crovascular endothelial cells.

@en

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Hava Karsenty Avraham

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Henry Koziel

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Seyha Seng

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Shalom Avraham

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Souvenir D Tachado

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Tzong-Shi Lu

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P2860

Tight junctions of the blood-brain barrier: development, composition and regulation

The endocannabinoid system as an emerging target of pharmacotherapy

Endothelial claudin: claudin-5/TMVCF constitutes tight junction strands in endothelial cells

Substance P and neurokinin-1 receptor modulation of HIV

The blood-brain barrier/neurovascular unit in health and disease

Substance P antagonist blocks leakage and reduces activation of cytokine-stimulated rat brain endothelium.

Enhanced disruption of the blood brain barrier by intracarotid mannitol injection during transient cerebral hypoperfusion in rabbits.

Heat shock treatment protects osmotic stress-induced dysfunction of the blood-brain barrier through preservation of tight junction proteins

Therapeutic potential of cannabinoids in CNS disease.

Hypertonicity, but not hypothermia, elicits substance P release from rat C-fiber neurons in primary culture

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Alexandros Makriyannis

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