Inability to activate Rac1-dependent forgetting contributes to behavioral inflexibility in mutants of multiple autism-risk genes.
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The Biology of Forgetting-A Perspective.Basic reversal-learning capacity in flies suggests rudiments of complex cognition.The neuronal protein Neurexin directly interacts with the Scribble-Pix complex to stimulate F-actin assembly for synaptic vesicle clusteringNeuroligin 4 regulates synaptic growth via the bone morphogenetic protein (BMP) signaling pathway at the Drosophila neuromuscular junction.Pharmacological Rescue of Hippocampal Fear Learning Deficits in Fragile X Syndrome.The Role of Actin Cytoskeleton in Dendritic Spines in the Maintenance of Long-Term Memory.Rho GTPases in Intellectual Disability: From Genetics to Therapeutic Opportunities.A retrieval-specific mechanism of adaptive forgetting in the mammalian brain
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Inability to activate Rac1-dependent forgetting contributes to behavioral inflexibility in mutants of multiple autism-risk genes.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 22 June 2016
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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Inability to activate Rac1-dep ...... of multiple autism-risk genes.
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Inability to activate Rac1-dep ...... of multiple autism-risk genes.
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type
label
Inability to activate Rac1-dep ...... of multiple autism-risk genes.
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Inability to activate Rac1-dep ...... of multiple autism-risk genes.
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prefLabel
Inability to activate Rac1-dep ...... of multiple autism-risk genes.
@en
Inability to activate Rac1-dep ...... of multiple autism-risk genes.
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Inability to activate Rac1-dep ...... of multiple autism-risk genes.
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Lianzhang Wang
Shiqing Wang
Yuqi Cheng
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10.1073/PNAS.1602152113
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2016-06-22T00:00:00Z