Regulation of cell-cell contact molecules and the metastatic phenotype of medullary thyroid carcinoma by the Raf-1/MEK/ERK pathway.
about
Medullary thyroid carcinoma: targeted therapies and future directionsFocal adhesion kinase, a downstream mediator of Raf-1 signaling, suppresses cellular adhesion, migration, and neuroendocrine markers in BON carcinoid cellsA novel RET inhibitor with potent efficacy against medullary thyroid cancer in vivo.Signaling pathways as specific pharmacologic targets for neuroendocrine tumor therapy: RET, PI3K, MEK, growth factors, and Notch.Nodular Thyroid Disease and Thyroid Cancer in the Era of Precision Medicine.Activation of VEGF/Flk-1-ERK Pathway Induced Blood-Brain Barrier Injury After Microwave Exposure.
P2860
Regulation of cell-cell contact molecules and the metastatic phenotype of medullary thyroid carcinoma by the Raf-1/MEK/ERK pathway.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on December 2008
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Regulation of cell-cell contac ...... by the Raf-1/MEK/ERK pathway.
@en
Regulation of cell-cell contac ...... by the Raf-1/MEK/ERK pathway.
@nl
type
label
Regulation of cell-cell contac ...... by the Raf-1/MEK/ERK pathway.
@en
Regulation of cell-cell contac ...... by the Raf-1/MEK/ERK pathway.
@nl
prefLabel
Regulation of cell-cell contac ...... by the Raf-1/MEK/ERK pathway.
@en
Regulation of cell-cell contac ...... by the Raf-1/MEK/ERK pathway.
@nl
P2860
P1433
P1476
Regulation of cell-cell contac ...... a by the Raf-1/MEK/ERK pathway
@en
P2093
P2860
P304
920-4; discussion 924-5
P356
10.1016/J.SURG.2008.07.020
P407
P577
2008-12-01T00:00:00Z