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Understanding the Impact of Infection, Inflammation, and Their Persistence in the Pathogenesis of Bronchopulmonary DysplasiaPostnatal inflammation in the pathogenesis of bronchopulmonary dysplasiaIncreased lung inflammation with oxygen supplementation in tracheotomized spontaneously breathing rabbits: an experimental prospective randomized study.Omeprazole Attenuates Pulmonary Aryl Hydrocarbon Receptor Activation and Potentiates Hyperoxia-Induced Developmental Lung Injury in Newborn MiceCD11b(+) Mononuclear Cells Mitigate Hyperoxia-Induced Lung Injury in Neonatal Mice.Standardisation of oxygen exposure in the development of mouse models for bronchopulmonary dysplasia.Recent advances in the mechanisms of lung alveolarization and the pathogenesis of bronchopulmonary dysplasia.Looking ahead: where to next for animal models of bronchopulmonary dysplasia?Perinatal Endotoxemia Induces Sustained Hepatic COX-2 Expression through an NFκB-Dependent Mechanism.
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P2860
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 25 April 2013
@en
vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Cyclooxygenase-2 in newborn hyperoxic lung injury
@en
Cyclooxygenase-2 in newborn hyperoxic lung injury.
@nl
type
label
Cyclooxygenase-2 in newborn hyperoxic lung injury
@en
Cyclooxygenase-2 in newborn hyperoxic lung injury.
@nl
prefLabel
Cyclooxygenase-2 in newborn hyperoxic lung injury
@en
Cyclooxygenase-2 in newborn hyperoxic lung injury.
@nl
P2093
P2860
P1476
Cyclooxygenase-2 in newborn hyperoxic lung injury
@en
P2093
Leif D Nelin
Lynette K Rogers
Markus Velten
Rodney D Britt
Trent E Tipple
P2860
P304
P356
10.1016/J.FREERADBIOMED.2013.04.012
P577
2013-04-25T00:00:00Z