Emerging roles of inositol 1,4,5-trisphosphate signaling in cardiac myocytes.
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Increased InsP3Rs in the junctional sarcoplasmic reticulum augment Ca2+ transients and arrhythmias associated with cardiac hypertrophyPostural changes in blood pressure associated with interactions between candidate genes for chronic respiratory diseases and exposure to particulate matterCalcium and IP3 dynamics in cardiac myocytes: experimental and computational perspectives and approachesGene knock-outs of inositol 1,4,5-trisphosphate receptors types 1 and 2 result in perturbation of cardiogenesisEpac enhances excitation-transcription coupling in cardiac myocytesLocation matters: clarifying the concept of nuclear and cytosolic CaMKII subtypesCalmodulin binding proteins provide domains of local Ca2+ signaling in cardiac myocytesCalcium-sensing receptors regulate cardiomyocyte Ca2+ signaling via the sarcoplasmic reticulum-mitochondrion interface during hypoxia/reoxygenation.CaMKII regulation of cardiac ryanodine receptors and inositol triphosphate receptors.Early remodeling of perinuclear Ca2+ stores and nucleoplasmic Ca2+ signaling during the development of hypertrophy and heart failure.Isoform- and tissue-specific regulation of the Ca(2+)-sensitive transcription factor NFAT in cardiac myocytes and heart failure.Developmental aspects of cardiac Ca(2+) signaling: interplay between RyR- and IP(3)R-gated Ca(2+) storesInositol trisphosphate receptor Ca2+ release channels in neurological diseases.A follow-up study for left ventricular mass on chromosome 12p11 identifies potential candidate genesInhibition of miR-25 improves cardiac contractility in the failing heart.Post-conditioning protecting rat cardiomyocytes from apoptosis via attenuating calcium-sensing receptor-induced endo(sarco)plasmic reticulum stress.The IP3 receptor regulates cardiac hypertrophy in response to select stimuli.Activation of NFATc1 is directly mediated by IP3 in adult cardiac myocytesRole of inositol 1,4,5-trisphosphate in the regulation of ventricular Ca(2+) signaling in intact mouse heart.In situ calibration of nucleoplasmic versus cytoplasmic Ca²+ concentration in adult cardiomyocytesDietary ω3 fatty acids modulate the substrate for post-operative atrial fibrillation in a canine cardiac surgery model.Endoplasmic reticulum resident protein 44 (ERp44) deficiency in mice and zebrafish leads to cardiac developmental and functional defectsIntercellular Ca(2+) waves: mechanisms and function.Absence of the Regulator of G-protein Signaling, RGS4, Predisposes to Atrial Fibrillation and Is Associated with Abnormal Calcium HandlingBDNF modulates heart contraction force and long-term homeostasis through truncated TrkB.T1 receptor activationCytosolic and nuclear calcium signaling in atrial myocytes: IP3-mediated calcium release and the role of mitochondria.Modulation of inositol 1,4,5-trisphosphate receptor type 2 channel activity by Ca2+/calmodulin-dependent protein kinase II (CaMKII)-mediated phosphorylationThe role of the paracrine/autocrine mediator endothelin-1 in regulation of cardiac contractility and growth.Differential activation of cultured neonatal cardiomyocytes by plasmalemmal versus intracellular G protein-coupled receptor 55.Akt mediated mitochondrial protection in the heart: metabolic and survival pathways to the rescue.Inositol 1, 4, 5-trisphosphate receptors and human left ventricular myocytesInhibition of thromboxane A2-induced arrhythmias and intracellular calcium changes in cardiac myocytes by blockade of the inositol trisphosphate pathway.Role of the excessive amounts of circulating catecholamines and glucocorticoids in stress-induced heart disease.Calcineurin-NFATc regulates type 2 inositol 1,4,5-trisphosphate receptor (InsP3R2) expression during cardiac remodeling.Trimeric intracellular cation channels and sarcoplasmic/endoplasmic reticulum calcium homeostasisApoptosis and autophagy: decoding calcium signals that mediate life or death.Visualization of Ca²+ signaling during embryonic skeletal muscle formation in vertebrates.Calcium signaling in cardiac myocytes.Redox control of cardiac excitability.The other side of cardiac Ca(2+) signaling: transcriptional control.
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Emerging roles of inositol 1,4,5-trisphosphate signaling in cardiac myocytes.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 15 June 2008
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Emerging roles of inositol 1,4,5-trisphosphate signaling in cardiac myocytes.
@en
Emerging roles of inositol 1,4,5-trisphosphate signaling in cardiac myocytes.
@nl
type
label
Emerging roles of inositol 1,4,5-trisphosphate signaling in cardiac myocytes.
@en
Emerging roles of inositol 1,4,5-trisphosphate signaling in cardiac myocytes.
@nl
prefLabel
Emerging roles of inositol 1,4,5-trisphosphate signaling in cardiac myocytes.
@en
Emerging roles of inositol 1,4,5-trisphosphate signaling in cardiac myocytes.
@nl
P2093
P2860
P50
P1476
Emerging roles of inositol 1,4,5-trisphosphate signaling in cardiac myocytes.
@en
P2093
Aleksey V Zima
Jens Kockskämper
Lothar A Blatter
P2860
P304
P356
10.1016/J.YJMCC.2008.05.014
P577
2008-06-15T00:00:00Z