Differential expression of Gs in a murine model of homocysteinemic heart failure
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Attenuation of beta2-adrenergic receptors and homocysteine metabolic enzymes cause diabetic cardiomyopathy.Exercise mitigates homocysteine - β2-adrenergic receptor interactions to ameliorate contractile dysfunction in diabetesHydrogen sulfide mitigates homocysteine-mediated pathological remodeling by inducing miR-133a in cardiomyocytesFunctional and Molecular Insights of Hydrogen Sulfide Signaling and Protein Sulfhydration.
P2860
Differential expression of Gs in a murine model of homocysteinemic heart failure
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 08 April 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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Differential expression of Gs in a murine model of homocysteinemic heart failure
@en
Differential expression of Gs in a murine model of homocysteinemic heart failure.
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Differential expression of Gs in a murine model of homocysteinemic heart failure
@en
Differential expression of Gs in a murine model of homocysteinemic heart failure.
@nl
prefLabel
Differential expression of Gs in a murine model of homocysteinemic heart failure
@en
Differential expression of Gs in a murine model of homocysteinemic heart failure.
@nl
P2093
P2860
P1476
Differential expression of Gs in a murine model of homocysteinemic heart failure
@en
P2093
John C Passmore
Jonathan C Vacek
Munish Kumar
Neetu Tyagi
Suresh C Tyagi
Thomas P Vacek
William M Hughes
P2860
P407
P577
2009-04-08T00:00:00Z