Gastrointestinal growth factors and hormones have divergent effects on Akt activation. - Test2 - elhamkhani - TriplyDB

Gastrointestinal growth factors and hormones have divergent effects on Akt activation.

Gastrointestinal growth factors and hormones have divergent effects on Akt activation.

http://www.wikidata.org/entity/Q37180159

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Therapeutic potential for novel drugs targeting the type 1 cholecystokinin receptor Insights into bombesin receptors and ligands: Highlighting recent advances Long-term mTOR inhibitors administration evokes altered calcium homeostasis and platelet dysfunction in kidney transplant patients.Gastrointestinal hormones/neurotransmitters and growth factors can activate P21 activated kinase 2 in pancreatic acinar cells by novel mechanisms Crosstalk between VEGFR2 and muscarinic receptors regulates the mTOR pathway in serum starved SK-N-SH human neuroblastoma cells Elucidation of the roles of the Src kinases in pancreatic acinar cell signaling PKD1 mediates negative feedback of PI3K/Akt activation in response to G protein-coupled receptors.PKCθ activation in pancreatic acinar cells by gastrointestinal hormones/neurotransmitters and growth factors is needed for stimulation of numerous important cellular signaling cascades.Insulin protects pancreatic acinar cells from cytosolic calcium overload and inhibition of plasma membrane calcium pump.Hormetic Effect of Berberine Attenuates the Anticancer Activity of Chemotherapeutic Agents.The Src kinase Yes is activated in pancreatic acinar cells by gastrointestinal hormones/neurotransmitters, but not pancreatic growth factors, which stimulate its association with numerous other signaling molecules The p21-activated kinase, PAK2, is important in the activation of numerous pancreatic acinar cell signaling cascades and in the onset of early pancreatitis events Comparative pharmacology of bombesin receptor subtype-3, nonpeptide agonist MK-5046, a universal peptide agonist, and peptide antagonist Bantag-1 for human bombesin receptors Regulation of acinar cell function in the pancreas.CCK1 and CCK2 receptors are expressed on pancreatic stellate cells and induce collagen production.Insulin protects pancreatic acinar cells from palmitoleic acid-induced cellular injury.

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Gastrointestinal growth factors and hormones have divergent effects on Akt activation.

http://www.wikidata.org/entity/Q37180159

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

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scientific article published on 07 January 2009

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Gastrointestinal growth factors and hormones have divergent effects on Akt activation.

@en

Gastrointestinal growth factors and hormones have divergent effects on Akt activation.

@nl

type

label

Gastrointestinal growth factors and hormones have divergent effects on Akt activation.

@en

Gastrointestinal growth factors and hormones have divergent effects on Akt activation.

@nl

prefLabel

Gastrointestinal growth factors and hormones have divergent effects on Akt activation.

@en

Gastrointestinal growth factors and hormones have divergent effects on Akt activation.

@nl

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J.CELLSIG.2009.01.003

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Cellular Signalling

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Gastrointestinal growth factors and hormones have divergent effects on Akt activation

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Andrea Pace

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K Martin Hoffmann

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Marc J Berna

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Michelle Thill

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Robert T Jensen

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Veronica Sancho

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P2860

Src-family tyrosine kinases in activation of ERK-1 and p85/p110-phosphatidylinositol 3-kinase by G/CCKB receptors

Phosphorylation and regulation of Akt/PKB by the rictor-mTOR complex

Translocation of PDK-1 to the plasma membrane is important in allowing PDK-1 to activate protein kinase B

Regulation of Akt signaling by D2 and D3 dopamine receptors in vivo

Cholecystokinin induces caspase activation and mitochondrial dysfunction in pancreatic acinar cells. Roles in cell injury processes of pancreatitis

Gastrointestinal hormones cause rapid c-Met receptor down-regulation by a novel mechanism involving clathrin-mediated endocytosis and a lysosome-dependent mechanism

Cholecystokinin stimulates extracellular signal-regulated kinase through activation of the epidermal growth factor receptor, Yes, and protein kinase C. Signal amplification at the level of Raf by activation of protein kinase Cepsilon

Ontogeny of hepatocyte growth factor and c-met/hgf receptor in rat pancreas

The Src family kinase, Lyn, is activated in pancreatic acinar cells by gastrointestinal hormones/neurotransmitters and growth factors which stimulate its association with numerous other signaling molecules

Role of translocation in the activation and function of protein kinase B

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622-638

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scholarly article

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10.1016/J.CELLSIG.2009.01.003

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4

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21

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L González-Fernández

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2009-01-07T00:00:00Z

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