Alterations in Gemin5 expression contribute to alternative mRNA splicing patterns and tumor cell motility. - Test2 - elhamkhani - TriplyDB

Alterations in Gemin5 expression contribute to alternative mRNA splicing patterns and tumor cell motility.

Alterations in Gemin5 expression contribute to alternative mRNA splicing patterns and tumor cell motility.

http://www.wikidata.org/entity/Q37182883

about

Aberrant RNA splicing and its functional consequences in cancer cells Metastasis Research Society-American Association For Cancer Research Joint Conference on Metastasis.Apoptosis resistance and PKC signaling: distinguishing features of high and low metastatic cells.A systems biology approach to defining metastatic biomarkers and signaling pathways Altered gene and protein expression by Nm23-H1 in metastasis suppression.Microarray-based cancer prediction using soft computing approach Insecticide resistance mediated by an exon skipping event.RRP1B is a metastasis modifier that regulates the expression of alternative mRNA isoforms through interactions with SRSF1.Invoking the power of thrombospondins: regulation of thrombospondins expression Meta-analysis and gene set analysis of archived microarrays suggest implication of the spliceosome in metastatic and hypoxic phenotypes.Gemin5: A Multitasking RNA-Binding Protein Involved in Translation Control.High resolution analysis of the human transcriptome: detection of extensive alternative splicing independent of transcriptional activity Identification of a gene expression signature associated with the metastasis suppressor function of NME1: prognostic value in human melanoma.

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P2860

Alterations in Gemin5 expression contribute to alternative mRNA splicing patterns and tumor cell motility.

http://www.wikidata.org/entity/Q37182883

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on February 2008

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Alterations in Gemin5 expressi ...... terns and tumor cell motility.

@en

Alterations in Gemin5 expressi ...... terns and tumor cell motility.

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type

label

Alterations in Gemin5 expressi ...... terns and tumor cell motility.

@en

Alterations in Gemin5 expressi ...... terns and tumor cell motility.

@nl

prefLabel

Alterations in Gemin5 expressi ...... terns and tumor cell motility.

@en

Alterations in Gemin5 expressi ...... terns and tumor cell motility.

@nl

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0008-5472.CAN-07-2632

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Cancer Research

P1476

Alterations in Gemin5 expressi ...... terns and tumor cell motility.

@en

P2093

Chand Khanna

http://www.w3.org/2001/XMLSchema#string

Christine E Horak

http://www.w3.org/2001/XMLSchema#string

Jong Heun Lee

http://www.w3.org/2001/XMLSchema#string

Li Rong Yu

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Patricia S Steeg

http://www.w3.org/2001/XMLSchema#string

Timothy D Veenstra

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Zhaojing Meng

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P2860

The Gemin5 protein of the SMN complex identifies snRNAs

Association of Lbc Rho guanine nucleotide exchange factor with alpha-catenin-related protein, alpha-catulin/CTNNAL1, supports serum response factor activation

Thrombospondin-4, an extracellular matrix protein expressed in the developing and adult nervous system promotes neurite outgrowth

Valproic acid increases the SMN2 protein level: a well-known drug as a potential therapy for spinal muscular atrophy

The DOCK180/Elmo complex couples ARNO-mediated Arf6 activation to the downstream activation of Rac1.

IQGAP1 regulates cell motility by linking growth factor signaling to actin assembly.

Cell motility is controlled by SF2/ASF through alternative splicing of the Ron protooncogene.

Essential role for the SMN complex in the specificity of snRNP assembly.

Regulation of alternative RNA splicing by exon definition and exon sequences in viral and mammalian gene expression

Unbalanced alternative splicing and its significance in cancer.

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639-644

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scholarly article

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10.1158/0008-5472.CAN-07-2632

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2008-02-01T00:00:00Z

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18245461

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