Stim1 and Orai1 mediate CRAC currents and store-operated calcium entry important for endothelial cell proliferation.
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The phospholipid-binding protein SESTD1 is a novel regulator of the transient receptor potential channels TRPC4 and TRPC5Store-independent activation of Orai1 by SPCA2 in mammary tumorsCell-cell contact formation governs Ca2+ signaling by TRPC4 in the vascular endothelium: evidence for a regulatory TRPC4-beta-catenin interactionSOCE and cancer: Recent progress and new perspectivesOrai1 calcium channels in the vasculaturePhospholipase C signaling and calcium influxEmerging role of TRP channels in cell migration: from tumor vascularization to metastasisStore-operated Ca2+ entry (SOCE) regulates melanoma proliferation and cell migrationThe STIM1-Orai1 pathway of store-operated Ca2+ entry controls the checkpoint in cell cycle G1/S transition.Pathophysiological Significance of Store-Operated Calcium Entry in Megakaryocyte Function: Opening New Paths for Understanding the Role of Calcium in ThrombopoiesisStore-operated calcium entry and diabetic complicationsStore-operated Ca2+ entry is remodelled and controls in vitro angiogenesis in endothelial progenitor cells isolated from tumoral patientsMitochondria control store-operated Ca2+ entry through Na+ and redox signals.S1P activates store-operated calcium entry via receptor- and non-receptor-mediated pathways in vascular smooth muscle cells.Mechanisms of STIM1 activation of store-independent leukotriene C4-regulated Ca2+ channels.ORAI1 calcium channel orchestrates skin homeostasis.Sodium entry through endothelial store-operated calcium entry channels: regulation by Orai1.STIM1 controls endothelial barrier function independently of Orai1 and Ca2+ entry.ORAI1 deficiency and lack of store-operated Ca2+ entry cause immunodeficiency, myopathy, and ectodermal dysplasia.Essential role for STIM1/Orai1-mediated calcium influx in PDGF-induced smooth muscle migrationLocal Ca²+ entry via Orai1 regulates plasma membrane recruitment of TRPC1 and controls cytosolic Ca²+ signals required for specific cell functionsA novel native store-operated calcium channel encoded by Orai3: selective requirement of Orai3 versus Orai1 in estrogen receptor-positive versus estrogen receptor-negative breast cancer cells.Glutathione adducts on sarcoplasmic/endoplasmic reticulum Ca2+ ATPase Cys-674 regulate endothelial cell calcium stores and angiogenic function as well as promote ischemic blood flow recovery.Association of ORAI1 haplotypes with the risk of HLA-B27 positive ankylosing spondylitis.Calcium/Calmodulin-dependent protein kinase II delta 6 (CaMKIIdelta6) and RhoA involvement in thrombin-induced endothelial barrier dysfunction.Actin cytoskeleton regulates stretch-activated Ca2+ influx in human pulmonary microvascular endothelial cells.CRAC channelopathies.Cooperative signaling via transcription factors NF-κB and AP1/c-Fos mediates endothelial cell STIM1 expression and hyperpermeability in response to endotoxin.ORAI1 genetic polymorphisms associated with the susceptibility of atopic dermatitis in Japanese and Taiwanese populationsPharmacology of store-operated calcium channelsStromal interaction molecule 1 (STIM1) and Orai1 mediate histamine-evoked calcium entry and nuclear factor of activated T-cells (NFAT) signaling in human umbilical vein endothelial cells.The effect of calcium channel blockers on the outcome of acute myeloid leukemia.VEGF-induced neoangiogenesis is mediated by NAADP and two-pore channel-2-dependent Ca2+ signaling.Pyk2 phosphorylation of VE-PTP downstream of STIM1-induced Ca2+ entry regulates disassembly of adherens junctions.TRPC5 channel sensitivities to antioxidants and hydroxylated stilbenes.Angiogenic functions of voltage-gated Na+ Channels in human endothelial cells: modulation of vascular endothelial growth factor (VEGF) signaling.The physiological function of store-operated calcium entrySTIM2 Contributes to Enhanced Store-operated Ca Entry in Pulmonary Artery Smooth Muscle Cells from Patients with Idiopathic Pulmonary Arterial Hypertension.Leukotriene-C4 synthase, a critical enzyme in the activation of store-independent Orai1/Orai3 channels, is required for neointimal hyperplasia.Enhanced expression of Stim, Orai, and TRPC transcripts and proteins in endothelial progenitor cells isolated from patients with primary myelofibrosis.
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Stim1 and Orai1 mediate CRAC currents and store-operated calcium entry important for endothelial cell proliferation.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 09 October 2008
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Stim1 and Orai1 mediate CRAC c ...... ndothelial cell proliferation.
@en
Stim1 and Orai1 mediate CRAC c ...... ndothelial cell proliferation.
@nl
type
label
Stim1 and Orai1 mediate CRAC c ...... ndothelial cell proliferation.
@en
Stim1 and Orai1 mediate CRAC c ...... ndothelial cell proliferation.
@nl
prefLabel
Stim1 and Orai1 mediate CRAC c ...... ndothelial cell proliferation.
@en
Stim1 and Orai1 mediate CRAC c ...... ndothelial cell proliferation.
@nl
P2093
P2860
P1433
P1476
Stim1 and Orai1 mediate CRAC c ...... ndothelial cell proliferation.
@en
P2093
Iskandar F Abdullaev
Jonathan M Bisaillon
Jose C Gonzalez
Marie Potier
Mohamed Trebak
Rajender K Motiani
P2860
P304
P356
10.1161/01.RES.0000338496.95579.56
P577
2008-10-09T00:00:00Z