Interstitial myofibroblasts: predictors of progression in membranous nephropathy. - Test2 - elhamkhani - TriplyDB

Interstitial myofibroblasts: predictors of progression in membranous nephropathy.

Interstitial myofibroblasts: predictors of progression in membranous nephropathy.

http://www.wikidata.org/entity/Q37202587

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Role of Bone Marrow-Derived Fibroblasts in Renal Fibrosis Smad3 signaling activates bone marrow-derived fibroblasts in renal fibrosis The molecular mechanism of leptin secretion and expression induced by aristolochic acid in kidney fibroblast Transforming growth factor-{beta}1 induces Smad3-dependent {beta}1 integrin gene expression in epithelial-to-mesenchymal transition during chronic tubulointerstitial fibrosis.Myofibroblast: a prognostic marker and target cell in progressive renal disease.Effect of interleukin 6 deficiency on renal interstitial fibrosis.Pathophysiology of renal disease associated with liver disorders: implications for liver transplantation. Part I.Etiopathology of chronic tubular, glomerular and renovascular nephropathies: clinical implications Dissection of key events in tubular epithelial to myofibroblast transition and its implications in renal interstitial fibrosis.Hepatocyte growth factor suppresses renal interstitial myofibroblast activation and intercepts Smad signal transduction.Anti-fibrotic effect of natural toxin bee venom on animal model of unilateral ureteral obstruction.Tissue-type plasminogen activator promotes murine myofibroblast activation through LDL receptor-related protein 1-mediated integrin signaling The expression of cytoskeletal proteins in kidney specimens of children with primary focal segmental glomerulosclerosis.Tumor Necrosis Factor-stimulated Gene 6 (TSG-6)-mediated Interactions with the Inter-α-inhibitor Heavy Chain 5 Facilitate Tumor Growth Factor β1 (TGFβ1)-dependent Fibroblast to Myofibroblast Differentiation Novel actions of tissue-type plasminogen activator in chronic kidney disease.C3a mediates epithelial-to-mesenchymal transition in proteinuric nephropathy Combined administration of a mutant TGF-beta1/Fc and rapamycin promotes induction of regulatory T cells and islet allograft tolerance.Astragalus mongholicus ameliorates renal fibrosis by modulating HGF and TGF-beta in rats with unilateral ureteral obstruction.Adiponectin promotes monocyte-to-fibroblast transition in renal fibrosis.Renal fibrosis is attenuated by targeted disruption of KCa3.1 potassium channels.The role of pericyte detachment in vascular rarefaction.The inhibitory effect of chimeric decoy oligodeoxynucleotide against NF-κB and Sp1 in renal interstitial fibrosis.Disruption of tissue-type plasminogen activator gene in mice reduces renal interstitial fibrosis in obstructive nephropathy.The Protective Effect of Melittin on Renal Fibrosis in an Animal Model of Unilateral Ureteral Obstruction.Role of P311 in interleukin-1α-induced epithelial to myofibroblast transition in kidney tubular epithelial cells.AEG-1 participates in TGF-beta1-induced EMT through p38 MAPK activation.Rho kinase pathway is likely responsible for the profibrotic actions of aldosterone in renal epithelial cells via inducing epithelial-mesenchymal transition and extracellular matrix excretion.Gelatinase A (MMP-2) is necessary and sufficient for renal tubular cell epithelial-mesenchymal transformation.Role of myofibroblasts and collagen type IV in patients of IgA nephropathy as markers of renal dysfunction The evolving contribution of renal pathology to understanding interstitial nephritis.Expression of calponin in periglomerular myofibroblasts of rat kidney with experimental chronic injuries Glomerular epithelial-myofibroblast transdifferentiation in the evolution of glomerular crescent formation.Do changes in transglutaminase activity alter latent transforming growth factor beta activation in experimental diabetic nephropathy?Mast cells: the forgotten cells of renal fibrosis.The role of myofibroblasts and interstitial fibrosis in the progression of membranous nephropathy.Systemic administration of naked plasmid encoding hepatocyte growth factor ameliorates chronic renal fibrosis in mice.Kidney injury molecule-1 expression in murine polycystic kidney disease.

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P2860

Interstitial myofibroblasts: predictors of progression in membranous nephropathy.

http://www.wikidata.org/entity/Q37202587

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on February 1997

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Interstitial myofibroblasts: predictors of progression in membranous nephropathy.

@en

Interstitial myofibroblasts: predictors of progression in membranous nephropathy.

@nl

type

label

Interstitial myofibroblasts: predictors of progression in membranous nephropathy.

@en

Interstitial myofibroblasts: predictors of progression in membranous nephropathy.

@nl

prefLabel

Interstitial myofibroblasts: predictors of progression in membranous nephropathy.

@en

Interstitial myofibroblasts: predictors of progression in membranous nephropathy.

@nl

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Journal of Clinical Pathology

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Interstitial myofibroblasts: predictors of progression in membranous nephropathy.

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Burrows C

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McWilliam LJ

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Roberts IS

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Shanks JH

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Venning M

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P2860

The natural history of renal function in untreated idiopathic membranous glomerulonephritis in adults.

Interstitial nephritis induced by protein-overload proteinuria.

The biology of the myofibroblast.

Interstitial myofibroblasts in IgA glomerulonephritis.

Predicting renal outcomes in severe lupus nephritis: contributions of clinical and histologic data.

Differentiation repertoire of fibroblastic cells: expression of cytoskeletal proteins as marker of phenotypic modulations.

Significance of tubulointerstitial changes in the renal cortex for the excretory function and concentration ability of the kidney: a morphometric contribution.

The role of lymphocytes in the progression of interstitial disease.

Loss of glomerular function and tubulointerstitial fibrosis: cause or effect?

Tubular-derived growth factors and cytokines in the pathogenesis of tubulointerstitial fibrosis: implications for human renal disease progression.

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123-127

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10.1136/JCP.50.2.123

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499736

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