Identifying advanced glycation end products as a major source of oxidants in aging: implications for the management and/or prevention of reduced renal function in elderly persons - Test2 - elhamkhani - TriplyDB

Identifying advanced glycation end products as a major source of oxidants in aging: implications for the management and/or prevention of reduced renal function in elderly persons

Identifying advanced glycation end products as a major source of oxidants in aging: implications for the management and/or prevention of reduced renal function in elderly persons

http://www.wikidata.org/entity/Q37214116

about

Glycation-altered proteolysis as a pathobiologic mechanism that links dietary glycemic index, aging, and age-related disease (in nondiabetics)Sugar and chromosome stability: clastogenic effects of sugars in vitamin B6-deficient cells Diabetic patients could do as well as non-diabetic patients without inflammation on peritoneal dialysis Direct Observation of Enhanced Nitric Oxide in a Murine Model of Diabetic Nephropathy.Oxidant stress and mitochondrial signaling regulate reversible changes of ERα expression and apoptosis in aging mouse glomeruli and mesangial cells.Advanced glycation end products: Key players in skin aging?Managing chronic inflammation in the aging diabetic patient with CKD by diet or sevelamer carbonate: a modern paradigm shift.AGE restriction in diabetes mellitus: a paradigm shift.Reactive metabolites and AGE/RAGE-mediated cellular dysfunction affect the aging process: a mini-review.Kidney aging--inevitable or preventable?Contrast-induced nephropathy: protective role of fenoldopam.Advanced Glycation End Products, Inflammation, and Chronic Metabolic Diseases: Links in a Chain?Renal Aging: Causes and Consequences.Advanced Glycation End Product (AGE) Accumulation in the Skin is Associated with Depression: The Maastricht Study.Age dependent accumulation patterns of advanced glycation end product receptor (RAGE) ligands and binding intensities between RAGE and its ligands differ in the liver, kidney, and skeletal muscle.Consequences of Advanced Glycation End Products Accumulation in Chronic Kidney Disease and Clinical Usefulness of Their Assessment Using a Non-invasive Technique - Skin Autofluorescence.Why do our kidneys get old?Role of advanced glycation end products in mobility and considerations in possible dietary and nutritional intervention strategies

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Identifying advanced glycation end products as a major source of oxidants in aging: implications for the management and/or prevention of reduced renal function in elderly persons

http://www.wikidata.org/entity/Q37214116

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Identifying advanced glycation ...... al function in elderly persons

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Identifying advanced glycation ...... al function in elderly persons

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J.SEMNEPHROL.2009.07.013

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Seminars in Nephrology

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Identifying advanced glycation ...... al function in elderly persons

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Feng Zheng

http://www.w3.org/2001/XMLSchema#string

Gary E Striker

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Helen Vlassara

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Jaime Uribarri

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James B Post

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Luigi Ferrucci

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Massimo Torreggiani

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Weijing Cai

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P2860

Advanced glycation endproduct (AGE) receptor 1 is a negative regulator of the inflammatory response to AGE in mesangial cells

Circulating glycotoxins and dietary advanced glycation endproducts: two links to inflammatory response, oxidative stress, and aging

Orally absorbed reactive glycation products (glycotoxins): an environmental risk factor in diabetic nephropathy

Oxidants, oxidative stress and the biology of ageing

Presence of diabetic complications in type 1 diabetic patients correlates with low expression of mononuclear cell AGE-receptor-1 and elevated serum AGE

Glycoxidation and lipoxidation in atherogenesis.

The biology of the receptor for advanced glycation end products and its ligands.

Manifestations of oxidant stress in uremia.

The multiligand receptor RAGE as a progression factor amplifying immune and inflammatory responses.

Inflammatory mediators are induced by dietary glycotoxins, a major risk factor for diabetic angiopathy

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