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Reactive oxygen species contribute to sleep apnea-induced hypertension in rats

Reactive oxygen species contribute to sleep apnea-induced hypertension in rats

http://www.wikidata.org/entity/Q37220930

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Chemistry and antihypertensive effects of tempol and other nitroxides Hypoxia-inducible factor 1 mediates increased expression of NADPH oxidase-2 in response to intermittent hypoxia The efficacy of antihypertensive drugs in chronic intermittent hypoxia conditions.Pathophysiology of sleep apnea Effect of AT1 receptor blockade on intermittent hypoxia-induced endothelial dysfunction.Hydroethidine- and MitoSOX-derived red fluorescence is not a reliable indicator of intracellular superoxide formation: another inconvenient truth Chronic intermittent hypoxia augments chemoreflex control of sympathetic activity: role of the angiotensin II type 1 receptor.Time-dependent adaptation in the hemodynamic response to hypoxia.Oxidative stress mediates cardiac infarction aggravation induced by intermittent hypoxia.Intermittent hypoxia-induced rat pancreatic β-cell apoptosis and protective effects of antioxidant intervention.NADPH oxidase 2 mediates intermittent hypoxia-induced mitochondrial complex I inhibition: relevance to blood pressure changes in rats Enhanced carotid body chemosensory activity and the cardiovascular alterations induced by intermittent hypoxia Eucapnic intermittent hypoxia augments endothelin-1 vasoconstriction in rats: role of PKCdelta.Intermittent hypoxia: cause of or therapy for systemic hypertension?Therapeutic potential of intermittent hypoxia: a matter of dose.HIF-1α activation by intermittent hypoxia requires NADPH oxidase stimulation by xanthine oxidase Intermittent hypoxia augments pulmonary vascular smooth muscle reactivity to NO: regulation by reactive oxygen species Xanthine oxidase inhibition attenuates endothelial dysfunction caused by chronic intermittent hypoxia in rats.Intermittent hypoxia in rats increases myogenic tone through loss of hydrogen sulfide activation of large-conductance Ca(2+)-activated potassium channels.Cardiac reactive oxygen species after traumatic brain injury.Transcriptional responses to intermittent hypoxia Inflammation induced by increased frequency of intermittent hypoxia is attenuated by tempol administration Obstructive sleep apnea syndrome (OSAS) and hypertension: pathogenic mechanisms and possible therapeutic approaches Intermittent hypoxia in rats reduces activation of Ca2+ sparks in mesenteric arteries.Intermittent Hypoxia-Induced Carotid Body Chemosensory Potentiation and Hypertension Are Critically Dependent on Peroxynitrite Formation Phosphoinositide-dependent kinase-1 and protein kinase Cδ contribute to endothelin-1 constriction and elevated blood pressure in intermittent hypoxia Molecular mechanisms of chronic intermittent hypoxia and hypertension Endothelin type A receptor antagonist normalizes blood pressure in rats exposed to eucapnic intermittent hypoxia NFAT is required for spontaneous pulmonary hypertension in superoxide dismutase 1 knockout mice.Effect of Continuous Positive Airway Pressure on Cardiovascular Biomarkers: The Sleep Apnea Stress Randomized Controlled Trial.Endothelin-1 mediates attenuated carotid baroreceptor activity by intermittent hypoxia.Obstructive sleep apnea: the new cardiovascular disease. Part I: Obstructive sleep apnea and the pathogenesis of vascular disease.Adaptive and maladaptive cardiorespiratory responses to continuous and intermittent hypoxia mediated by hypoxia-inducible factors 1 and 2.Hypertension in obstructive sleep apnea: risk and therapy.Vascular effects of intermittent hypoxia Intermittent hypoxia-induced increases in reactive oxygen species activate NFATc3 increasing endothelin-1 vasoconstrictor reactivity.Mechanisms of intermittent hypoxia induced hypertension.Cardiovascular complications of sleep apnea: role of oxidative stress Mechanisms of cardiac dysfunction in obstructive sleep apnea.Obstructive sleep apnea, oxidative stress and cardiovascular disease: lessons from animal studies

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Reactive oxygen species contribute to sleep apnea-induced hypertension in rats

http://www.wikidata.org/entity/Q37220930

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 31 August 2007

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Reactive oxygen species contribute to sleep apnea-induced hypertension in rats

@en

Reactive oxygen species contribute to sleep apnea-induced hypertension in rats.

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type

label

Reactive oxygen species contribute to sleep apnea-induced hypertension in rats

@en

Reactive oxygen species contribute to sleep apnea-induced hypertension in rats.

@nl

prefLabel

Reactive oxygen species contribute to sleep apnea-induced hypertension in rats

@en

Reactive oxygen species contribute to sleep apnea-induced hypertension in rats.

@nl

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AJPHEART.00219.2007

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American Journal of Physiology - Heart and Circulatory Physiology

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Reactive oxygen species contribute to sleep apnea-induced hypertension in rats

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Ana Q da Silva

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Carmen M Troncoso Brindeiro

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Kyan J Allahdadi

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Nancy L Kanagy

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Victoria Youngblood

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P2860

Prospective study of the association between sleep-disordered breathing and hypertension

Detection and characterization of the product of hydroethidine and intracellular superoxide by HPLC and limitations of fluorescence

Increased adhesion molecules expression and production of reactive oxygen species in leukocytes of sleep apnea patients.

Incidence of sleep-disordered breathing in an urban adult population: the relative importance of risk factors in the development of sleep-disordered breathing.

Association of sleep-disordered breathing, sleep apnea, and hypertension in a large community-based study. Sleep Heart Health Study.

Intermittent hypoxia is associated with oxidative stress and spatial learning deficits in the rat.

Prevalence of symptoms and risk of sleep apnea in the US population: Results from the national sleep foundation sleep in America 2005 poll.

The prevalence, cost implications, and management of sleep disorders: an overview.

Cardiovascular disorders and obstructive sleep apnea syndrome.

Relation of sleep-disordered breathing to cardiovascular disease risk factors: the Sleep Heart Health Study.

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10.1152/AJPHEART.00219.2007

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arterial hypertension

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