Chromatin modifications associated with DNA double-strand breaks repair as potential targets for neurological diseases. - Test2 - elhamkhani - TriplyDB

Chromatin modifications associated with DNA double-strand breaks repair as potential targets for neurological diseases.

Chromatin modifications associated with DNA double-strand breaks repair as potential targets for neurological diseases.

http://www.wikidata.org/entity/Q37249162

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Prion-induced neurotoxicity: Possible role for cell cycle activity and DNA damage response Extracellular vesicles as trans-genomic agents: Emerging roles in disease and evolution.Oxidative stress and its significant roles in neurodegenerative diseases and cancer.Poly(ADP-ribose) polymerase 1 is a novel target to promote axonal regeneration.Epigenetic drug discovery for Alzheimer's disease.NRAGE is involved in homologous recombination repair to resist the DNA-damaging chemotherapy and composes a ternary complex with RNF8-BARD1 to promote cell survival in squamous esophageal tumorigenesis.Looking above but not beyond the genome for therapeutics in neurology and psychiatry: epigenetic proteins and RNAs find a new focus.

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Chromatin modifications associated with DNA double-strand breaks repair as potential targets for neurological diseases.

http://www.wikidata.org/entity/Q37249162

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on October 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Chromatin modifications associ ...... ets for neurological diseases.

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Chromatin modifications associ ...... ets for neurological diseases.

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type

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Chromatin modifications associ ...... ets for neurological diseases.

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Chromatin modifications associ ...... ets for neurological diseases.

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Chromatin modifications associ ...... ets for neurological diseases.

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Chromatin modifications associ ...... ets for neurological diseases.

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S13311-013-0210-9

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Neurotherapeutics

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Chromatin modifications associ ...... ets for neurological diseases.

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Camille Brochier

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Double strand breaks can initiate gene silencing and SIRT1-dependent onset of DNA methylation in an exogenous promoter CpG island

Translating the Histone Code

SIRT1 regulates UV-induced DNA repair through deacetylating XPA

SIRT1 regulates autoacetylation and histone acetyltransferase activity of TIP60

Tyrosine dephosphorylation of H2AX modulates apoptosis and survival decisions

SIRT1 deacetylates APE1 and regulates cellular base excision repair

Poly(ADP-ribose)-dependent regulation of DNA repair by the chromatin remodeling enzyme ALC1

Mutations in the JARID1C gene, which is involved in transcriptional regulation and chromatin remodeling, cause X-linked mental retardation.

An epigenetic blockade of cognitive functions in the neurodegenerating brain

Histone methylation regulates memory formation.

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10.1007/S13311-013-0210-9

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