Multiple heparan sulfate binding site engagements are required for the infectious entry of human papillomavirus type 16.
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Large scale RNAi reveals the requirement of nuclear envelope breakdown for nuclear import of human papillomavirusesThe C-Terminal Arm of the Human Papillomavirus Major Capsid Protein Is Immunogenic and Involved in Virus-Host InteractionDirect binding of retromer to human papillomavirus type 16 minor capsid protein L2 mediates endosome exit during viral infectionLoss of Glycosaminoglycan Receptor Binding after Mosquito Cell Passage Reduces Chikungunya Virus InfectivityTranslocation of the papillomavirus L2/vDNA complex across the limiting membrane requires the onset of mitosisMeasurement of neutralizing serum antibodies of patients vaccinated with human papillomavirus L1 or L2-based immunogens using furin-cleaved HPV Pseudovirions.The nuclear retention signal of HPV16 L2 protein is essential for incoming viral genome to transverse the trans-Golgi network.Human papillomavirus species-specific interaction with the basement membrane-resident non-heparan sulfate receptor.Intermediate heparan sulfate binding during HPV-16 infection in HaCaTs.Identification of Broad-Genotype HPV L2 Neutralization Site for Pan-HPV Vaccine Development by a Cross-Neutralizing Antibody.Diversity of heparan sulfate and HSV entry: basic understanding and treatment strategies.Interaction of human tumor viruses with host cell surface receptors and cell entryHeparan sulfate-dependent enhancement of henipavirus infection.Naturally Occurring Capsid Protein Variants of Human Papillomavirus Genotype 31 Represent a Single L1 Serotype.Topography of the Human Papillomavirus Minor Capsid Protein L2 during Vesicular Trafficking of Infectious Entry.The U4 Antibody Epitope on Human Papillomavirus 16 Identified by Cryo-electron MicroscopyA human monoclonal antibody against HPV16 recognizes an immunodominant and neutralizing epitope partially overlapping with that of H16.V5Incoming human papillomavirus type 16 genome resides in a vesicular compartment throughout mitosis.Furin Cleavage of L2 during Papillomavirus Infection: Minimal Dependence on CyclophilinsThe Cytoskeletal Adaptor Obscurin-Like 1 Interacts with the Human Papillomavirus 16 (HPV16) Capsid Protein L2 and Is Required for HPV16 Endocytosis.Variants in human papillomavirus receptor and associated genes are associated with type-specific HPV infection and lesion progression of the cervix.The DE and FG loops of the HPV major capsid protein contribute to the epitopes of vaccine-induced cross-neutralising antibodies.Preparation and properties of a papillomavirus infectious intermediate and its utility for neutralization studies.The tetraspanin CD151 in papillomavirus infection.Functional assessment and structural basis of antibody binding to human papillomavirus capsid.Vimentin Modulates Infectious Internalization of Human Papillomavirus 16 Pseudovirions.Incoming human papillomavirus 16 genome is lost in PML protein-deficient HaCaT keratinocytes.Human papillomavirus major capsid protein L1 remains associated with the incoming viral genome throughout the entry process.Cruising the cellular highways: How human papillomavirus travels from the surface to the nucleus.HPV entry into cells.Structural features of glycan recognition among viral pathogens.The CD63-Syntenin-1 Complex Controls Post-Endocytic Trafficking of Oncogenic Human Papillomaviruses.A clinicopathological study of episomal papillomavirus infection of the human placenta and pregnancy complications.Crystal Structures of Two Immune Complexes Identify Determinants for Viral Infectivity and Type-Specific Neutralization of Human PapillomavirusThe cellular endosomal protein stannin inhibits intracellular trafficking of human papillomavirus during virus entry.Superinfection Exclusion between Two High-Risk Human Papillomavirus (HPV) Types During a Co-Infection.Extracellular conformational changes in the capsid of human papillomaviruses contribute to asynchronous uptake into host cells.A new cell culture model to genetically dissect the complete human papillomavirus life cycle.Papillomaviruses and Endocytic TraffickingHeterotetrameric annexin A2/S100A10 (A2t) is essential for oncogenic human papillomavirus trafficking and capsid disassembly, and protects virions from lysosomal degradation
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Multiple heparan sulfate binding site engagements are required for the infectious entry of human papillomavirus type 16.
description
article científic
@ca
article scientifique
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articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 21 August 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Multiple heparan sulfate bindi ...... human papillomavirus type 16.
@en
Multiple heparan sulfate bindi ...... human papillomavirus type 16.
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type
label
Multiple heparan sulfate bindi ...... human papillomavirus type 16.
@en
Multiple heparan sulfate bindi ...... human papillomavirus type 16.
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prefLabel
Multiple heparan sulfate bindi ...... human papillomavirus type 16.
@en
Multiple heparan sulfate bindi ...... human papillomavirus type 16.
@nl
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P356
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Multiple heparan sulfate bindi ...... human papillomavirus type 16.
@en
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Jhimli Dasgupta
Kathleen F Richards
Malgorzata Bienkowska-Haba
Martin Sapp
Xiaojiang S Chen
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P304
11426-11437
P356
10.1128/JVI.01721-13
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P577
2013-08-21T00:00:00Z