Glucose and glycolysis are required for the successful infection of macrophages and mice by Salmonella enterica serovar typhimurium. - Test2 - elhamkhani - TriplyDB

Glucose and glycolysis are required for the successful infection of macrophages and mice by Salmonella enterica serovar typhimurium.

Glucose and glycolysis are required for the successful infection of macrophages and mice by Salmonella enterica serovar typhimurium.

http://www.wikidata.org/entity/Q37256563

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Characterization of phosphofructokinase activity in Mycobacterium tuberculosis reveals that a functional glycolytic carbon flow is necessary to limit the accumulation of toxic metabolic intermediates under hypoxia Preferential use of central metabolism in vivo reveals a nutritional basis for polymicrobial infection PoxA, YjeK, and Elongation Factor P Coordinately Modulate Virulence and Drug Resistance in Salmonella enterica Host Delivery of Favorite Meals for Intracellular Pathogens Carbon metabolism of enterobacterial human pathogens growing in epithelial colorectal adenocarcinoma (Caco-2) cells RamA, a member of the AraC/XylS family, influences both virulence and efflux in Salmonella enterica serovar Typhimurium.The bacterial phosphoenolpyruvate:carbohydrate phosphotransferase system: regulation by protein phosphorylation and phosphorylation-dependent protein-protein interactions.An incomplete TCA cycle increases survival of Salmonella Typhimurium during infection of resting and activated murine macrophages.Identification of metabolic pathways essential for fitness of Salmonella Typhimurium in vivo.Role of intracellular carbon metabolism pathways in Shigella flexneri virulence.Transcriptomic responses of Salmonella enterica serovars Enteritidis and Typhimurium to chlorine-based oxidative stress CsrA and Cra influence Shigella flexneri pathogenesis.Parallel exploitation of diverse host nutrients enhances Salmonella virulence.Technologies and approaches to elucidate and model the virulence program of salmonella.Nutritional and metabolic requirements for the infection of HeLa cells by Salmonella enterica serovar Typhimurium Commensal and Pathogenic Escherichia coli Metabolism in the Gut.Vibrio vulnificus Secretes an Insulin-degrading Enzyme That Promotes Bacterial Proliferation in Vivo.A Comparison of the ATP Generating Pathways Used by S. Typhimurium to Fuel Replication within Human and Murine Macrophage and Epithelial Cell Lines.Inherent Variability of Growth Media Impacts the Ability of Salmonella Typhimurium to Interact with Host Cells.Disruption of glycolytic flux is a signal for inflammasome signaling and pyroptotic cell death Bacterial Metabolism Shapes the Host-Pathogen Interface RaoN, a small RNA encoded within Salmonella pathogenicity island-11, confers resistance to macrophage-induced stress.Salmonella require the fatty acid regulator PPARδ for the establishment of a metabolic environment essential for long-term persistence.A link between gut community metabolism and pathogenesis: molecular hydrogen-stimulated glucarate catabolism aids Salmonella virulence Small RNA functions in carbon metabolism and virulence of enteric pathogens.Salmonella-host interactions - modulation of the host innate immune system.Intracellular Salmonella metabolism.Differences in carbon source utilization of Salmonella Oranienburg and Saintpaul isolated from river water.The future of biology in driving the field of hyperthermia.Salmonella Co-opts Host Cell Chaperone-mediated Autophagy for Intracellular Growth.Bacterial peptide transporters: Messengers of nutrition to virulence.Deletions in the pyruvate pathway of Salmonella Typhimurium alter SPI1-mediated gene expression and infectivity Protein Acetylation Is Involved in Salmonella enterica Serovar Typhimurium Virulence.To Eat and to Be Eaten: Mutual Metabolic Adaptations of Immune Cells and Intracellular Bacterial Pathogens upon Infection.Glucocorticoid receptor promotes the function of myeloid-derived suppressor cells by suppressing HIF1α-dependent glycolysis.The infectious intracellular lifestyle of Salmonella enterica relies on the adaptation to nutritional conditions within the Salmonella-containing vacuole.Gluconeogenesis, an essential metabolic pathway for pathogenic Francisella.Salmonella enterica: a surprisingly well-adapted intracellular lifestyle.Understanding the sequential activation of Type III and Type VI Secretion Systems in Salmonella typhimurium using Boolean modeling.Elongation factor P: Function and effects on bacterial fitness.

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Glucose and glycolysis are required for the successful infection of macrophages and mice by Salmonella enterica serovar typhimurium.

http://www.wikidata.org/entity/Q37256563

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 20 April 2009

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vedecký článok

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vetenskaplig artikel

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vědecký článek

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name

Glucose and glycolysis are req ...... enterica serovar typhimurium.

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Glucose and glycolysis are req ...... enterica serovar typhimurium.

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Glucose and glycolysis are req ...... enterica serovar typhimurium.

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Glucose and glycolysis are req ...... enterica serovar typhimurium.

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Glucose and glycolysis are req ...... enterica serovar typhimurium.

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Glucose and glycolysis are req ...... enterica serovar typhimurium.

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Infection and Immunity

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Glucose and glycolysis are req ...... a enterica serovar typhimurium

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Arthur Thompson

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Gary Rowley

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Steven D Bowden

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P2860

Phosphoenolpyruvate:carbohydrate phosphotransferase systems of bacteria

One-step inactivation of chromosomal genes in Escherichia coli K-12 using PCR products

Gene disruption in Escherichia coli: TcR and KmR cassettes with the option of Flp-catalyzed excision of the antibiotic-resistance determinant

Construction of versatile low-copy-number vectors for cloning, sequencing and gene expression in Escherichia coli

The global burden of typhoid fever

Modelling within-host spatiotemporal dynamics of invasive bacterial disease

Transport of galactose, glucose and their molecular analogues by Escherichia coli K12

Modulation of Escherichia coli adenylyl cyclase activity by catalytic-site mutants of protein IIA(Glc) of the phosphoenolpyruvate:sugar phosphotransferase system.

Of mice, calves, and men. Comparison of the mouse typhoid model with other Salmonella infections.

The alternative sigma factor, sigmaE, is critically important for the virulence of Salmonella typhimurium

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3117-3126

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scholarly article

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10.1128/IAI.00093-09

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7

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77

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Jay C. D. Hinton

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2009-04-20T00:00:00Z

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24311327

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19380470

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Salmonella enterica

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2708584

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